January 29, 2025 • 53 mins
Kim Pittis, LCSP, (PHYS), MT - fsmsports365.com
Carolyn McMakin, MA, DC - frequencyspecific.com/getstarted
00:14 A Surprising Medical Case
03:08 Exploring Cranial Nerve Frequencies
04:31 Understanding Muscle Spasticity
06:40 Neurological Insights and Patient Education
09:19 FSM Practitioners and Complex Cases
14:10 Muscle Mechanics and Treatment Strategies
25:39 TMJ and Retrodiscal Tissue Treatment
29:13 Exploring TMJ and Facial Nerve Innervation
30:42 Understanding Trigeminal and Facial Nerve Functions
31:55 Upcoming Events and Notable Speakers
33:18 Heart Health and FSM Applications
43:38 Addressing Stenosis and FSM Treatment
52:19 Concluding Remarks and Next Steps
Understanding Complex Neurological Cases
The podcast opens with a fascinating case study involving a Ph.D. research biochemist who developed a condition known as Cavernous Hemangioma. This case illustrates the intricacies of treating fragile neurological conditions with FSM. The discussion delves into the importance of understanding cranial nerves and their frequencies, highlighting the necessity of increased secretions in nerves and innovative approaches to manage cranial nerve involvement. Practitioners are encouraged to familiarize themselves with the specifics of cranial nerve treatment, particularly how conditions such as thalamic pain can be influenced by surgery-related changes.
Handling Muscle Spasticity and Weakness
The session further delves into muscle management, emphasizing the concept of "tight weakness" and "stretch weakness" in muscle physiology. For medical practitioners, understanding the cellular mechanics, including the role of actin and myosin, can be crucial in diagnosing and managing spastic or chronically shortened muscles. The podcast underscores the benefit of FSM in increasing ATP for muscle relaxation and strengthening—both critical for optimal muscle function.
Stenosis: Misconceptions and Treatment Strategies
Stenosis, a common concern, is addressed with clarity, dismissing the myth that FSM is contraindicated for such conditions. Practitioners learn that understanding the mechanics of spinal fluid flow and managing inflammation are key to effective treatment. Special attention is given to cervical and lumbar stenosis, advising on how to approach these differently based on anatomical and physiological variances.
Innovative Techniques for Treating Shoulder and TMJ Issues
The podcast emphasizes the versatility of FSM in treating complex conditions like shoulder injuries and TMJ disorders. It introduces the idea of using specific frequencies for labrum and annular ligament issues, which can be potent in resolving chronic pain and dysfunction in shoulder and TMJ patients.
Genetic and Mitochondrial Considerations in Treatment
The session discusses a genetic condition, SPG7, shedding light on the importance of understanding genetic and mitochondrial contributions to disease progression. For practitioners, using FSM for abnormal protein accumulation and mitochondrial health can stave off advancement in otherwise degenerative conditions.
Final Thoughts and Peer Involvement
The concluding parts stress the importance of continuous learning and collaboration in the FSM community, urging practitioners to remain curious and collaborative, utilizing tools like Netter's Anatomy for inspiration. Sharin
Mark as Played
Transcript
Episode Transcript
Available transcripts are automatically generated. Complete accuracy is not guaranteed.
(00:00):
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I have to tell you somethingbecause I did something this week
that I didn't think was possible.
(00:22):
Yeah.
There's this, she's aPhD research biochemist.
Very smart.
Had a ca, it's ca, it's calledCavernous Hemangioma or a cavernoma,
so it's a tangle of blood vessels.
It was.
At the top part of her cerebellumand in between her cerebellum and
(00:46):
her pons and she had a stroke.
They, the problem with thosevessels is they're fragile
and they just break and bleed.
So she got off of the stroke and.
In about six months, all of her functionback, and then some nice neurosurgeon
(01:08):
said, those cavernomas are still thereand they can break again anytime.
So I should go in And take them out.
Okay.
That was a year and a half ago.
And when you look, if you look inNetter, I think it's on a page 117,
those of you that have your books there.
It's the base, it's the bottom ofthe brain that shows where All the
(01:31):
nerves, cranial nerves come out andwhat they do I have a frequency for
the medulla, I have a frequency of thepons, but at some point in the last
10 years of his life, I, George gotannoyed with me because I was less than
(01:53):
enthusiastic about thiswhole page of frequencies he
created for the mitochondria.
And so he stopped telling me aboutfrequencies that he scanned for.
So I'm treating this lady thefirst day, I get only so far.
And then during the surgery, soshe's got long track symptoms.
(02:16):
That don't make, on theright side, it makes sense.
She's got thalamic pain fromthe bleed that was, that
happened during the surgery.
Because when you take out a wholebunch of blood vessels, this is
the webinar everybody has from along time ago that nobody watches.
It's called take it apart.
What happened in what order and whatever.
(02:38):
So when he took out the hemangioma,the cavernous hemangioma he had
to cut off the blood supply.
Oops.
And so the right sided body pain startedthree to four months after the surgery.
(03:00):
Right leg.
That's the thalamus.
So I knew I got rid of that with four.
For sure.
I knew I could do that.
But on the second day, it's Ineed, I know George gave somebody
frequencies for the cranial nerves.
So I texted JJ.
And j. was awake and answeringtexts and j. said here's the list.
(03:24):
She couldn't move theright side of her face.
Couldn't close her eyelid.
We haven't got to that nerve yet.
We did.
She couldn't do this.
She, the facial nerve wasaffected and that's motor.
The trigeminal nerve wasaffected that was sensory.
I wrapped around her neck, contactdown the middle of her face, and I ran
(03:51):
briefly, increased trauma, necrosis.
Torn or broken, but then just 81and these cranial nerves, but 81
and the, there are four frequencies.
I don't know how there are fourfrequencies for the facial nerve, but
(04:13):
it actually makes sense because there's.
A branch that goes here, a branchthat goes here, a branch that goes
here, and here, eyebrow, right?
And her, she couldn't chew.
And her, when muscles are spastic, it'sbecause they lose, innervation, right?
(04:39):
Before they get flaccid, they get spastic.
Sometimes they just stay spastic.
It's like having a sensory cortex stroke.
You just get spastic.
So her jaw muscles were spastic andthen weak, so they wouldn't contract
because they were already spastic.
(05:02):
So I'm running 81 and thesefour frequencies and it's
Did those muscles just suck?
It didn't.
They did.
They just, they softened, right?
She went, I don't know.
And then pretty soon after theyrelaxed, they started for circulating.
Like they would just twitch Oh,the nerves are talking to me now.
(05:26):
I didn't think it was possible.
There are two frequenciesfor the accessory nerve.
She said, Oh, I have thispain here in my shoulder.
And it's that's the upper trapezius.
That's the accessory nerve.
Here on JJ's list, George's list.
There's the accessory nerve, so Iunwrapped it, so it just was laid
(05:47):
across the back of her neck and thebase of her skull, and two frequencies
for the accessory nerve, and shesaid, Oh, it feels funny down below.
I said, Yeah, that's the lowerpart of the upper trapezius.
It worked.
Spasticity went away.
And then, she said, it hurtsback here, base of the skull,
(06:09):
and right here, sensory pain.
This hurts.
Okay, take it apart.
This is not a cranial nerve.
When they did the surgery, he wentin between branches of the occipital
nerve, which is a C2 nerve root.
(06:30):
And I just That was easy.
C2, we know it can fixbecause that's just 396.
So we're on 40 and 396 while Iwent and looked up something.
And at the end of, at the endof an hour, we spent two hours
educating her and me about what thesecranial nerves did and how it is.
(06:58):
She had a disc bulge that was justtouching, barely, the spinal cord.
After this, that was anMRI before the surgery.
After the surgery.
Her spinal cord is peanut shapedbecause think of the position they
have to put you in to get a fiber opticscope into the base of your skull.
(07:22):
She had to be in hardcervical flexion, right?
So then there's this
pressure inflection on this disc.
And so after the surgery, the disc
pushes into the cord.
That created 40 and 10 and 81 and 10 downher left side and her right side, right?
(07:52):
So there's part of her symptoms.
That's the take it apart part.
Kevin just posted thetake it apart webinar.
That's the take it apart part.
What about your long tracksymptoms is from your spinal
cord and what's from your brain?
The first day I had 11 machines on her.
Concussion and vagus because you have toincrease the circulation of the brain.
(08:15):
Right and then what it was a thing.
Thank you, George and I'm sorry.
And thank you.
J. and I know we're going to seethese frequencies somewhere at
the advanced and I have, I willchange the advanced laminate.
Kevin just asked me 2 days ago.
(08:36):
Are your advanced slides finished?
I went.
Oh, yeah, sure.
No, just kidding.
Not just kidding.
But that means I have to talk evenfaster at the advanced and the advanced
laminate is going to get longer.
Actually, there's a whole sheetof investigational frequencies.
An end of one.
Doesn't mean that they're real, right?
(08:59):
It's I'm sorry, everybody.
It takes me at least5 before I believe it.
And that really used to annoy George.
I'm sorry.
And then by 10, you're pretty suretakes 20 before you're certain.
It's, but it gives us someplace to go.
(09:19):
And it also means that FSMpractitioners have to know.
More about neurology than mostneurologists tell your patients.
So this is a PhD in biochemistry,native IQ, just talking to her, probably
about 150 and we spent two hours.
(09:41):
Talking about what these cranialnerves do, where they come from,
why she has, why is she dizzy?
It's because her seventh nerve is,eighth nerve is, goes to here and
here, and this disagrees with that.
She went, why didn't theneurologist tell me this?
Either they don't know how toexplain it, or they don't think
(10:01):
you're going to understand it,or they simply don't have time.
Could be any of the above.
Maybe all three.
Yeah.
So that was two hours, but mostly it wasme educating me to tell you the truth.
Okay.
Remind myself, made my brain hurt.
Right.
And then, but then I had,we have living visual.
(10:24):
I didn't need a video.
I had this person next to me with the, Ican't even remember the name of the nerve.
The one that makes your eye close.
Her eye can't close.
Right.
Her eye muscles don't work right.
So she just has a patchover her right eye.
It's okay, so we, so I had living proof ofwhat each one of these cranial nerves did.
(10:50):
And then we had the book in front ofus telling us where they came from.
Okay, that makes sense, that make, okay,now I, I understand why those nose don't
work because the bleed, And the hemangiomawent to the pons, and the pons is where
all these cranial nerves come out of.
And so I educated me, I educated her,and now I'm going to educate you guys.
(11:13):
And I'm really sorry ahead of time,but it's going to be fun, really.
And so maybe I'll find some videos aboutwhat facial nerve does, and maybe I can
take patient pictures of this patient.
I don't know.
Anyway, so that's, that was, that,that was what I did the last two days.
That was pretty fun.
So you led with, you did something thatyou were surprised that it works when
(11:38):
you say these things and then you explainthe course of action that you took, I'm
sitting here going that makes sense.
That makes sense.
That makes sense.
Like you don't do anythingthat doesn't make sense.
It's not like you'rerunning frequency for the.
I don't know, 10.
Like everything probably makes sense.
I guess if I tried to bring myself there,but it makes sense what you're saying.
(12:01):
So I like that you took the leapof faith with the new frequencies
as opposed to just running nerve.
They're either going towork or they're not work.
It's okay, I, George was wrong.
And that's what I'm always waitingfor, but he's hardly ever wrong.
And it was 1st, you had to figureout how they're supposed to work
(12:22):
and what they're supposed to doand understand why her masseter.
So if you feel up and next year, likefrom this bone down to that bone,
there's a little muscle there, right?
You've got an Audi instead of it.
She's got a hole.
That muscle is just gone, right?
(12:43):
Her jaw muscle, hard as rock,and she can't close her mouth.
She has to chew soft things.
Okay, what happens to amuscle when it's denervated?
It spasms.
Rock hard.
So how do you un denervate it?
(13:03):
You increase secretions in thenerve, and you just drive the mother.
Just, I'm sorry, I know you've beenroad hard and put away wet, but come
with me now, it's gonna be fine.
You got, you have to do this now.
Okay!
Give me some ATP, will you?
Yeah, I got that.
(13:23):
So the current does the, how do I do this?
Because the nerve is asking you.
Okay, I'm willing.
You're telling me what to dowith this 81 and 292 thing.
But how do I do that?
I don't have, oh, 500 percent ATP.
I guess I can try.
And then the nerve goes, okay,so the first branch, all of a
(13:48):
sudden this started to fill in.
And I'm going.
No, and the second branch was the funone because the master was just like
a brick, and it started to soften.
So it's the, that's actuallywhat we're going to end up
doing in the neuro section.
It's pretty fun.
That is fun.
(14:08):
So I want to talk about this.
Concept really quickly because it'ssomething that we talk about in the sports
and the sports advanced is when you havesomething that's called a tight weakness.
A muscle that is chronically shortened,we say has a, so you can have a tight
weakness and a stretch weakness.
So the tight weakness is like what you'retalking about when a muscle is spastic or
(14:28):
chronically shortened, it's weak, it can'tcontract and relax and it comes from two
sort of philosophies of why that happened.
You explained the first neurophysiologicalphenomenon about innervation and the
other is the mechanical property.
So when the actin and myosin, so peoplewho are just listening, you're going
to have to, I'll verbally walk youthrough what my fingers are doing.
(14:50):
My hands are, my fingersare interlaced and.
I'm having optimal space between them.
So that would be likeoptimal actin and myosin.
So the myosin and actin can pulland that's our sliding filament
mechanism and the muscle shorten.
But if a muscle is too tight andthere's too much overlap, the actin
and myosin can't optimally shorten.
(15:10):
So you get this Chronically shortenedstate or a short or a a short weakness
or a tight weakness is what we call it.
So I would even follow it upplaying with the sarcomere.
Yeah.
It takes, the sarcomere is, you'redoing this in a lacing your fingers.
I do this with the creepy crawlies.
Yeah.
Because they have these little heads that,grab each other and walk along each other.
(15:35):
Then hold
onto the mycin.
Yes.
And so they're like, golf takes.
3 ATP to make him go like this.
It takes 2 ATP to relax him.
That's where the ATP part comes.
And in order
to go ahead.
No, I said, that's why it makes sense.
What when we use frequencies like49 and 81, that this works is
(15:57):
because there's no more juice left.
You being those vial fibrils, the energythat it needs to optimally contract
and shorten this just makes sense.
Yeah, and then the, in order to bestrong, so strong and normal, a muscle
has to be able to contract and relax.
(16:20):
It has to be able to do bothcontract, relax, contract, relax.
You can't get strong if all,if you're just doing contract.
And if it's constantly elongated,because, and I'm insistent on this.
Which makes me sort of a pain,but it's constantly elongated
(16:42):
because the antagonist muscleis chronically short, right?
So if your internal rotators in yourhip, pectineus, the brevis, the adductor
is really tight because of adhesionsin the femoral plexus, or because
(17:05):
of degeneration in the hip joint.
You're chronically short.
internally rotated.
That means your piriformis and yourglute med are chronically elongated
which makes them weak becausethey don't ever get, they're just
elongated to elongated, they don'tever get to shorten and then elongate.
(17:29):
You have to be able todo both to be strong.
So it's.
And that's why we see thesecomplexes all the time.
So we talk about the psoas beingchronically shortened and the glutes
being chronically elongated, right?
So we are sitting.
So we are in perpetual hip flexion.
(17:49):
too much.
We should not be sitting as much as we do.
So our psoas, iliacus, ourhip flexors get shortened.
And then like you just said, theantagonist is chronically elongated.
There has to be A symbiotic relationshipbetween agonists and antagonists or
flexors, extensors, internal, external.
You name it.
There's going to be an oppositemuscle that we really have to pay a
(18:12):
lot of attention to, especially whenwe work with FSM, biopic treatment.
Yeah.
It's just fascinating.
I've had two impossiblepatients this week.
One has a condition called SPG 7,and it's, I had to look it up again
after It's a genetic abnormality whereyou're missing the enzyme that puts
(18:38):
together peptide that the ribosomesneed to make a neurotransmitter.
Somehow it involves the mitochondria.
And
there's this frequency forabnormal protein accumulation.
(18:59):
And another place where actually thiswas the first place where I got in
trouble with George, because he wentthrough and came up with all these
frequencies for the inner and theouter and then this and then all these
different parts of the mitochondria.
It's whatever.
How are you going to tell if they work?
(19:19):
So I. Use the frequency for abnormalprotein accumulation, which is what
happens in SPG7, and it is incurable.
And we, she, it, I've been treatingher for three years and she hasn't
(19:42):
progressed, and she should be in awheelchair by now, and she's not.
She's still progressing, butat about three or four percent
of what she should be doing.
And she's one of the things,one of the recommendations,
we finally put her in a brace.
(20:03):
She was resisting that for fiveyears and it's look, if you fall
and break something, it's game over.
So get a brace and use a cane.
And then I had to teach her to puther cane in her left hand because
it's a right leg that's really worse.
And then it's the part where youtake one Baclofen three times a
(20:23):
day, For the spasticity, you mightconsider taking two in the morning.
There's, I, we're not gettingit with the spasticity.
We soften it and it'sbetter, but just try it.
Okay.
(20:44):
And so it's a collaboration.
Between FSM, making it betterthan it should be, and initially
we treated her spinal cord andthat took care of the spasticity.
Then after about a year, we hadto start treating her brain.
(21:08):
Yesterday we were back to her spinal cord.
And it's because the SPG7 has movedup to her arm and her shoulder.
I thought, oh, it's justyour neck and your arm.
It's the SPG7.
It's finally hit the upper extremity.
It was just in the lowerextremity for three years.
And now it's okay, so I use Google a lot.
(21:31):
Yeah, nobody on earth would know allthe things that walk into your clinic
or have to think about treating themall the way an FSM practitioner might
have to think about these things.
And then it's really funwhen somebody walks in.
My last patient last night justhas a vestibular injury and
low back pain, arthritic knees.
Yes.
(21:54):
And then you second guess it.
Is it that easy?
This is really all that's.
He came in and one of his primarycomplaint besides his knee pain and
his low back pain was, and he justmentioned in passing his balance.
One or two questions.
(22:15):
I handed him the BIVSS,his score was 48 over 18.
I said, yeah, don't even come backuntil after you've seen Rescue.
Get your prescription.
And that's when he, so now hecame back and he said can I
get my lenses made at Costco?
I said, absolutely not.
Why not?
(22:35):
It's so explaining that, butit's just a nice, simple shoulder
would be nice, and then so
many shoulders the last little while.
It's.
You're right.
I think you said that they alljump on a bus and then I have to
think, okay what am I here to learn?
I am here to learn somethingabout all these shoulders.
(22:58):
Peck minor.
Can you get out of my brain for a second?
I was just going to say the amountof times that I just start now
at Peck minor and subclavius.
Subscap always.
Yeah that's like a, but nowI feel like I have to treat
Subclavius Impact minor first.
Back
to get back.
(23:19):
So then I can go in.
Oh, okay.
I hadn't thought about that.
And then the labrum that's beenthe last part, especially where
they talk about how it happened.
My dog pulled on the leash.
Is there any way to jam the humoralhead forward in the fossa without
(23:42):
trashing the front part of the.
No,
no I think that's an important partto talk about is people will say,
Oh, I had imaging in my labrum spine.
I understand that you don'thave a surgical repair labrum.
However, there is micro trauma everywhere.
And so people who have had.
(24:03):
Dislocations, traction injuries,even though the labrum may appear
fine and intact on imaging, stillgo to torn and broken in the labrum.
2 14 is labrum or meniscus
and
I didn't like that frequency in thebeginning and now I am addicted to it.
(24:25):
I can't not never use it.
It's like the holy grailof shoulder injuries.
Shoulders, hips, knees, and it'sthank God elbows don't hurt.
Wait, do elbows.
They don't
know that I have that very peskyannular ligament that can get trashed.
Yeah, annular ligament is really
icky.
Yeah,
(24:47):
and now we have frequency for it.
And for the 34 people who are with uson the 38 people who are with us in
person on this adventure, all of youare now figuring out that you come to
the podcast with netter that and maybea glass of wine, but netter next to you.
So it's, yeah.
(25:08):
So it's necessary.
And it's not to say that youdon't know your anatomy and yes,
there's all this great softwarethat people have on their iPads and
their phones and their computers.
I'm sorry.
Nothing to me beats anatomy.
Netter.
It's like my laminate.
Like sometimes I'll and look veryquickly, but sometimes just having
the page open for some inspiration.
(25:29):
Can be very helpful.
Look at this, would 214
help JAWS?
Okay, so I have somethingto say about that.
So it's I've fooled around withit a little bit because I've been
doing a lot of work with TMJ, talkwith Mary Ellen Chalmers all the
time with our orthodontic patients.
So I've used it because I've hadan open mind about that that disc.
(25:54):
Okay.
But, and it's on one of my TMJ talks,that the disc that's in the jaw is not
the same as the disc that's in our back,so those disc frequencies didn't work.
The 710 works.
The annulus works, doesn't it?
A little bit, but for pain, sothe superior slip of the lateral
pterygoid is what attaches tothe disc and pulls the disc down.
(26:17):
Forward, but if the pain andthe bleeding and the scarring
is caused from the retrodiscaltissue, which is behind the disc
Right,
it's like this long band of Scarves thatare attached to scarves that are attached
to scarves You have to treat the pterygoidthat attaches to the disc attaches to
the retrodiscal tissue on a pathologylevel we're seeing that the tearing
the bleeding and the pain is more to dowith the retrodiscal tissue because The
(26:42):
disc in the jaw is this by concave YouMostly cartilaginous aneural structure.
So it's not innervated, so wedon't get pain from the disc.
And the other thing with thepterygoid is that the pterygoid
gets tight because it's protecting
(27:03):
loose ligaments between the toothand the jaw, the tooth and the jaw.
And once the pterygoid gets tight, itgets micro tears in the flat connective
tissue, because it's not a roundligament, it's a flat connective tissue
that connects to that disc tissue.
And when, so the pterygoid should relax.
(27:29):
When you close your jaw and let thedisc slide back, the pterygoid doesn't
relax because it's holding on fordear life because you've got a loose
ligament or you had braces or you gothit in the mouth in a bar fight or you
hit your face on the steering wheel.
And so when the pterygoid doesn't relax,doesn't let the disc go back, the bone
(27:51):
goes back, pulls the disc back, andit's that snap that causes the bleeding.
Is that how it works?
So it can, so you're talking aboutreduction versus a non reducing disc.
So when a disc doesn't reduce inthe fossa that pop and that snap,
so when it's being dragged, that'swhen that retro discal tissue leads.
(28:16):
Okay.
So it's on the forwardpop, not the closing pop.
It's the direct,
it's when you open.
When it reduces and it snaps back, becausethe retrodiscal tissue is pretty much
made out of adipose it just reduces backand it just slurps it back into space.
I'm sure the disc could get trashedin that fossil, so running torn
(28:37):
and broken would help that, butthe bleeding that happens, so
running bleeding in the adipose, Has
been
cracked, but like any type of TMJ work.
So yeah, we're good at treating thepterygoids, but running bleeding.
In the ADEPT, so 18 on A, 97 onB, has been a total game changer
(28:58):
because you don't think ofHow does the ADEPT post bleed?
The retrodiscal tissue bleedsbecause it's very vascularized.
So when there's bleeding Shouldn't there
be 18 and 162 capillaries?
Yes, I've run that, but when it's You saidit and I've never been able to unhear it.
It's not necessarily a structurethat bleeds, but if it was bled onto.
(29:19):
Okay.
So then 3 and 97 as well.
Correct.
Okay.
Okay, got it.
Yeah.
Interesting.
And 284 then right in 97 becausethere's like bruising back there.
Yes.
I will stop rolling my eyes on that slidein the core where you have all these
tissues that you treat mostly becauseI've got what a minute and a half to
(29:42):
cover that slide and it's just Ask him.
It's
and I love being asked, but when I, again,once you hear something and once you,
and it was during COVID that I took a TMJcourse and the videos that we saw of a
cadaver and they're on YouTube as well ofjust the disc and how that retro discal
tissue, which I hadn't really didn't knowmuch about, but to see how important that
(30:05):
is, how innovated it is is fascinating.
It has to be.
When you look at the innervationto the face, and that is, so you've
got motor to the facial muscles.
Pterygoid is innervatedby the facial nerve.
(30:28):
Triteminal is sensory.
Every other motor in the face.
The pterygoid has got to be facial nerve.
It's masseter pterygoid, temporalis,and whatever this little thing is.
Thank God
for Google.
Honestly, pterygoids, bothlateral and medial are inhibited
by the mandibular branch oftrigeminal nerve, cranial nerve 5.
(30:50):
What?
Trigeminal nerve is motor?
Wait.
Facial nerve is motor.
Trigeminal is not just sensory.
Trigeminal is motor.
That makes my brain hurt just when youthought you knew something you don't.
I hate that part.
Speaking of, we havesome questions to get to.
That's a lot easier thandealing with weight.
(31:12):
As I continue to google.
We're going to be disinterested by the
I'm going to
have a
hard time looking at this.
Because the mandibular branch of thetrigeminal, the mandibular maxillary
branch of the trigeminal nerve.
Lateral pterygoid innervated bymandibular nerve, which is third branch
of trigeminal nerve, cranial nerve 5.
Specifically, the informationcomes from the pancreas.
So
the mandibular branch goes upand innervates the pterygoid,
(31:35):
which is actually in theregion of the maxillary branch?
This middle part is themaxillary branch, right?
Yep.
So the trigeminal nerveis both motor and sensory?
Okay.
I'll be fine.
Branch
and vestibular nerve.
Wow.
Okay.
I want to get Mary Ellen on.
(31:56):
Okay.
Oh.
She's going to be in Phoenix.
Do you guys realize, do you realizewhat is happening in Phoenix?
Magic.
Just Reski, Mary Ellen, Jay Shaw, Eduardo.
The Julianna Mortenson Jennifer Ernst.
(32:17):
Now this, we had to shorten it becauseDiana Cross announced three weeks ago,
four weeks ago, that she's coming up.
I'm coming!
I have an hour.
And it's wait, it's all full.
Okay, I had this hour presentation andnow we're going to squash into 30 minutes.
So it's Diana Cross is comingwith more news on whatever
124 is supposed to be doing.
(32:39):
Jennifer Ernst, we have the datafrom the Feinstein Institute.
You guys, the Vegas.
The vagal tone protocol is patented.
It has enough data thatTIVIC has a patent on it.
I just want y'all to swimthat there for a second.
(33:01):
And understand why, if the onlyreason you came was for that
little 30 minute thing that we get.
Because I got 20 minutes onwhat the vagus does, and she's
got 10 minutes on the TIVIC.
Now, on the data, which is, and thenwe all have an hour with time across.
Okay, questions.
Mom had a stent placeda year and a half ago.
(33:22):
Sensorized mild electrical heart block.
That's like being a littlepregnant, but that's another.
And congestive heart failure.
Those two things go together.
I have run off a seminar.
Wait.
Congestive heart failure.
Time out.
How long has she been on statins?
I have run FSM and are greatresults for a number of things.
(33:43):
Never run heart health.
Oh, no, it's not beyondyour area of expertise.
Heart disease, all heart diseaseis inflammation, oxidative stress,
and autoimmune dysfunction.
That's problems with the heart.
Congestive heart failure.
So there's a little bit missing here in.
(34:06):
The history, which is heart block is whenthe electrical system on the left side
depends, you can have left bundle branchblock, right bundle branch block, you
can have both one or the other, or both.
Usually it's left bundle branchblock starts first and then you
get right bundle branch block.
(34:27):
It's there's this electrical wiringthat goes up the left side of your heart
that makes it go smush at contract.
And.
The wiring gets, the insulationgets thin and heart block is
just the wires don't work.
It's like your heart's been inflamedlong enough that it's dissolved the
myelin that's on these, they're my, Ithink they're myelinated nerves because
(34:51):
they're really fast with your heart.
And.
So heart block, I get that's, thatjust happens when you get old.
Congestive heart failure does notnecessarily go with heart block right?
Congestive heart failuregoes with statins.
I'm gonna guess I'm not, Ican't guess, I have to ask how
(35:15):
long Has she been on statins?
Because one of the main side effectsof statins is congestive heart failure.
It just, it kills the mitochondria in thebrain, the heart, and the skeletal muscle.
I have run FSM on her.
Heart health can't hurt anybody.
It's everybody.
(35:37):
Everybody should run heart healthon themselves once a month.
Men and women, but especiallywomen, should run breast health
on themselves once a month.
So you can have my once a month programthat combines heart health and breast
health, and you just have reducedinflammation in different tissues.
For the two sets and you put amagnetic converter on one side of
(36:00):
your chest and magnetic convert onthe other side of your chest, pick up
the breast and heart all in between.
Reducing inflammation on the heart isnot beyond the area of your expertise.
It's a no brainer.
It's there's nobody that's going tohurt as long as they don't have an
infection or infectious pericarditis.
And she doesn't have that.
(36:20):
She lives in the traditional as wellas an alternative medical desert.
Condolences.
No one else for her to see.
I'm with her right now.
She's not experiencing any symptoms.
Yay.
Was wondering if there arecontraindications or any wishes.
No, there's no reason notto run Howard Health on her.
It would be okay.
Recommendation Either one, wettowels, pulse DMF, depending
(36:41):
on how old your mom is.
Wet towels are just a pain.
Use wet washcloths oneither side of her chest.
I prefer not to run them backto front through the heart.
Either side is just fine.
So you create a field that goes throughthe heart warm, wet washcloths, and then
cover up with something that keeps warm.
(37:02):
If you have a magnetic converter,use that because the heart likes
pulsed EMF and it works just as well.
If she is not on 400, 400, yes, I said 400milligrams of CoQ10, if she's on statins,
And her cholesterol was 350 or 400.
(37:24):
Statins are fine.
I'm not going to fightwith anybody about it.
But she has to be on 400 milligramsof CoQ10 if she's still on statins.
And D ribose is what Mark Houstonuses for congestive heart failure.
The other thing with congestiveheart failure is The heart doesn't
(37:46):
use, like your skeletal muscles usesugar to make the mitochondria work.
The heart doesn't use sugar.
There's not enough energy in it.
It uses fatty acids.
There's this fat pad on your heart,like right in the middle of it,
and the heart just doesn't use it.
The body just replaces that,and your heart uses it.
(38:09):
As you get insulin resistant,everybody that's over the age
of 30, look down at your tummy.
And if your tummy is sticking out justa little bit, that's insulin resistance.
Embryologically, the fat that'sin your heart fat pad is the same
embryological origin as the fatthat's in your tummy that gets more
(38:32):
insulin resistant as we get older.
So type 2 diabetes is insulinresistance to start with.
So you want to run insulin resistanceor at least inflammation in the adipose.
It's part of heart health, andthat's why it's part of heart health,
because you want the fatty acids inthat fat tissue to be more available
(38:58):
for the heart to use for energy.
Congestive heart failure is
the muscle can't get enoughenergy to contract properly, so
it gets weak and it gets big.
It tries.
But that's why it gets big.
Sorry.
This is, that's no,
(39:18):
but she just said the person thatwrote that said that she bought her
on CoQ10 sometime after her stent.
So
that's
great.
Okay.
All right.
So it's make sure.
So look at her cholesterol.
That's the other thing.
If her cholesterol is 200when they put her on statins.
That's dumb.
It's 200 is normal.
If her cholesterol is 400, thenyou can make a case for it.
(39:40):
That's it's a wholedifferent conversation.
Sylvie patient who had a resection.
Oh, this is okay.
Resection of an acoustic neuroma.
Loss of hearing and dizziness.
No fooling.
What is the frequency ofthe eighth cranial nerve?
I'll let you I don'thave it in my phone yet.
JJ is going to show mehow to put it on my buddy.
(40:01):
Oh wait.
I have a thing.
I, somebody, he sent, JJsent me a text with that.
There it is.
Here it is.
Eighth nerve.
Vestibular cochlear nerve.
114, which is also the frequencyfor the thymus, which is why I
(40:27):
had this difficulty with Georgia.
It worked anyway.
And 137 is the vestibular cochlear nerve.
Yeah, and acoustic neuromas are,
an acoustic neuroma is a benign tumorthat gets onto the eighth nerve inside
(40:50):
the ear, or maybe a little behind theear, but you have the facial nerve and the
eighth nerve does the vestibular system.
You have the vestibular edge ofit and the hearing end of it.
That goes to the cochlea.
That's the eighth nerve.
But the seventh nerve that doessensation to the face, right?
(41:12):
Isn't that something?
Oh, no, go away.
Oh, Piper.
I'll tell you later.
Yeah.
Acoustic neuromas when they go inand take the neuroma out, the damage
the nerve and it's if she has loss ofhearing, she's dizzy because there's no
nerve going to the vestibular system.
Sylvia, I don't think you can treat it.
(41:33):
They take the tumor out,but they cut the nerve.
And it's, this is normal.
It, the dizziness usually goes away.
The hearing never comes back.
They're always deaf in that ear.
And if you're lucky you can try 40 and 44.
You can try If there's no nerve,I don't know how you can increase
(41:57):
secretions in our nerve that's not there.
And then the part of the brain that's,that does vestibular, somebody tell me,
I think it's the temporal lobe, right?
It's just, it's like just inboardfrom your ears and that's the only
thing there is the temporal lobe.
(42:18):
So it should be the temporal lobe.
So we have a frequency for that.
You can try that.
They, yeah, that's a tough 1.
You got the frequency see if it works.
Okay.
There's a leaf.
You covered George's cranial nerve.
Any update on the motor center numbersand exactly which centers they represent?
(42:38):
Absolutely.
Totally.
100%. No, I've neverused the motor centers.
I use 92.
I don't even know what the motorcenters are or where they are.
Or why they're even there on thelist that we have motor centers.
We have sensory centers.
Are they in the brain?
Are they appear on thesensory motor cortex?
Are they sections of the sensory motorcortex as you step down the homunculus?
(43:04):
Don't know.
Sorry, Leif.
I'll see in a couple of weeks.
No.
Okay.
Is there any, anybody have aquestion that I can answer?
Those were good.
Did we finish all the ones that
Oh, good.
Yay.
Yeah, and the chat, we'renot too chatty today.
So that's good.
Kevin did the chat part.
(43:27):
Okay, that was, we went.
Nowhere near my listtoday, but that is fine.
You got 10 minutes to start on your list.
I love your list.
It keeps us grounded.
The list was piggybacking offof after we got off last week,
we did a lot of spine disc.
One thing that always the pathology thatalways seems to come up every so often,
(43:52):
whether it's on the podcast or at thecourse, or I get an email is stenosis.
And, it's that face and a lot of peoplehave stenosis and for some reason people
seem to think that FSM is contraindicatedor it's going to make it worse.
It's not.
So let's just say that.
And FSM can, it can still be helpfuleven when there is stenosis present.
(44:16):
I think the only time we had thelike cautionary tale was with 40.
With anything you want to do withthe spinal cord, so this lady with
this disc bulge that goes into themiddle of her cord, that is stenosis.
Everything that I run from her neckto her feet has to be alternating.
I can't polarize anything.
(44:37):
As far as I can tell, when you polarizethe current, it appears to, just
clinically, it's like it increasesthe, the spinal fluid flows up the
dura, around the brain, down thecenter, then back up the front, and
then around, and whatever the flow is.
(44:59):
Okay, so when you polarize.
The current positive.
The first thing that happens is the pain.
If the patient has stenosis,they will say, Oh, hurts in
between my shoulder blades.
And then the next thing theysay is, Whoa, I got a headache.
(45:22):
All right.
Now, if you look at what does thatmean, if you take that apart, if the
spinal fluid is trying to come downand the stenosis blocks it, right?
What is it?
What is the spinal fluid pressing on?
It's the disc annulus.
So there's pressure on the annulus.
(45:43):
They get pain in betweentheir shoulder blades first.
And then as the pressure builds up.
So you got a hair in the drain here,can't get down, pushes on the annulus,
you get pain on the shoulder blades,and then it fills up and fills in the
ventricles and increases intercranialpressure and they get a headache.
(46:07):
Stop the current, make it alternating.
That's the first thing you can doif you catch it soon enough and
you can tell I'm speaking from
Experience So you make it alternatingfirst if that doesn't work and
they still have a headache.
You just have them set up I hadone that just was really bad.
She had stenosis, I didn't suspectand So she sat up and I ran
(46:32):
just inflammation in the spinalfluid And got her headache down.
Now, if you know from imaging that theyhave stenosis, you treat the spinal cord.
So you already know that the spinal cordis at least touched by the disc annulus
(46:52):
that and the disc annulus may be leaking.
So you have to run 40 and 10to get their body pain down.
And if there's enough compression, youmight have to run 81 and 10 to get the
tone down in their legs and their trunk.
And you have to do thosealternating neck to feet, but then
(47:15):
sometimes stenosis comes from facetsin the back, arthritis pushing on the
backside of the cord, and the disc,which is calcified and fibrous and
nasty, it's pushing on the front.
So what would you do?
You'd run.
What where is the pushing on the backcoming from 217 hard calcium or 91 in
(47:44):
the periosteum in the joint capsule.
On the back side and on thedisc annulus on the front.
So you set up from front to backat the level of stenosis, front to
back at the neck to treat the discannulus and the facets in the back.
(48:07):
If the stenosis is in thelumbar spine, it's not a deal.
The lumbar spine stenosis isalmost always at L3, 4 and as long
as they can feel their crotch.
Urinate defecate.
As long as they can do thosethings, the stenosis in their lumbar
spine is just about nerve pain.
(48:27):
So you treat nerve pain and it, there'sno spinal fluid problems there at all.
'cause there's no spinal cord at all.
Three spinal cord stops.
It's at T 10.
I just, it's just, it's not there.
So L three stenosis is easy.
You just keep the nerves and makesure they can feel the crotch.
If their crotch gets numb,you call the surgeon.
(48:50):
That's just, I'm alittle hyper today, huh?
No, it's good.
It's good.
It's good energy.
I'm just like, like everybody else.
I'm just like taking it all in becauseyou always hear something different, but
yeah, I just wanted to, I said, stenosisis always something that comes up and
it's not contraindicated, but thereare just things to be aware of and the
(49:11):
way you explained it, it makes just.
total sense of why we are aware of it.
It's like an acute injury whenthere's a lot of inflammation.
We don't want to ram a sharp,positive current through an
already hot bed of activity.
I just think about that.
Cynosis, just like an acuteinflammatory response, there's so
much activity, throwing a lot ofanything more at it is just chaotic.
(49:37):
You just have to think of cervicalstenosis like hair in the drain.
It just it interferes with spinalfluid flow and it backs it up.
I have to figure out a way to talk aboutit so that it doesn't scare people.
Apparently, I have too many slidesabout it in the core because
people, you don't see it very often.
In the first place.
(49:58):
No, I see a fair bit.
Do you?
The last couple years I definitely have.
Remember you asked formore difficult patients.
I did.
I remember that plane ride.
I remember.
I remember warning you about that.
It was answered and I'm gratefulbecause I'm learning so much and maybe
(50:18):
that's why I'm seeing that learningall about shoulders all over again.
I don't know.
And at four o'clock in twotwo and a half minutes.
Yes.
I get to do intro to FSM webinar.
And it's, we've got likefour, 300 and some odd.
400 people signed up for itofficially, but anybody can join in
(50:41):
and it made me go over introduction.
Why would you do FSM?
And there's this 1 slide thatI just had so much fun with.
It was really cool
for
14, 414, 414 people.
Really?
That's going to be on.
Yeah, it's and it'll end up onYouTube, but the intro is really fun.
(51:02):
And I think we're going to put it onthe resources page for practitioners
so you can send it to people.
How does FSM work?
Why would I use it?
And I only used with two exceptions.
I only used published papers.
Everything we have is digital.
Data.
Oh, can you give the link?
And there's Kevin.
(51:23):
I was just going to interruptto say, Kevin, can you, but
so we get off this one.
Yeah, I know.
Are there Dan those patients moresusceptible to stenosis in some ways?
Yes, because they'reconnective tissue is looser.
And they're more likely to getdysbulgias, so it's possible, right?
(51:44):
And then there's the wholetethered cord thing that happens
with Ehlers Danlos patients.
There's a higher propensityfor tethered cord.
Go looking for tethered cords.
On an MRI the tethered cord sofar has been really easy to treat.
It's, the spinal cord shouldend up, should end at about T10.
(52:05):
When you have quote unquote tetheredcord it's down at about, L1.
It's just, it just stretches thecord a bit, and so you treat the
discs, and you treat the cord for 40.
And it's
four o'clock, and I'll let you get offhere, and get on the new one, and so glad
you're doing it, and thanks everybody.
(52:27):
Thank you.
Some of you guys will probably needto log off of this one, and get on the
other one if you're staying the extrahour, and we'll see everybody next week.
See you next
week.
Bye.
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(52:49):
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com as well as more information aboutfrequency specific microcurrent.
I have to tell you somethingbecause I did something this week
that I didn't think was possible.
(00:22):
Yeah.
There's this, she's aPhD research biochemist.
Very smart.
Had a ca, it's ca, it's calledCavernous Hemangioma or a cavernoma,
so it's a tangle of blood vessels.
It was.
At the top part of her cerebellumand in between her cerebellum and
(00:46):
her pons and she had a stroke.
They, the problem with thosevessels is they're fragile
and they just break and bleed.
So she got off of the stroke and.
In about six months, all of her functionback, and then some nice neurosurgeon
(01:08):
said, those cavernomas are still thereand they can break again anytime.
So I should go in And take them out.
Okay.
That was a year and a half ago.
And when you look, if you look inNetter, I think it's on a page 117,
those of you that have your books there.
It's the base, it's the bottom ofthe brain that shows where All the
(01:31):
nerves, cranial nerves come out andwhat they do I have a frequency for
the medulla, I have a frequency of thepons, but at some point in the last
10 years of his life, I, George gotannoyed with me because I was less than
(01:53):
enthusiastic about thiswhole page of frequencies he
created for the mitochondria.
And so he stopped telling me aboutfrequencies that he scanned for.
So I'm treating this lady thefirst day, I get only so far.
And then during the surgery, soshe's got long track symptoms.
(02:16):
That don't make, on theright side, it makes sense.
She's got thalamic pain fromthe bleed that was, that
happened during the surgery.
Because when you take out a wholebunch of blood vessels, this is
the webinar everybody has from along time ago that nobody watches.
It's called take it apart.
What happened in what order and whatever.
(02:38):
So when he took out the hemangioma,the cavernous hemangioma he had
to cut off the blood supply.
Oops.
And so the right sided body pain startedthree to four months after the surgery.
(03:00):
Right leg.
That's the thalamus.
So I knew I got rid of that with four.
For sure.
I knew I could do that.
But on the second day, it's Ineed, I know George gave somebody
frequencies for the cranial nerves.
So I texted JJ.
And j. was awake and answeringtexts and j. said here's the list.
(03:24):
She couldn't move theright side of her face.
Couldn't close her eyelid.
We haven't got to that nerve yet.
We did.
She couldn't do this.
She, the facial nerve wasaffected and that's motor.
The trigeminal nerve wasaffected that was sensory.
I wrapped around her neck, contactdown the middle of her face, and I ran
(03:51):
briefly, increased trauma, necrosis.
Torn or broken, but then just 81and these cranial nerves, but 81
and the, there are four frequencies.
I don't know how there are fourfrequencies for the facial nerve, but
(04:13):
it actually makes sense because there's.
A branch that goes here, a branchthat goes here, a branch that goes
here, and here, eyebrow, right?
And her, she couldn't chew.
And her, when muscles are spastic, it'sbecause they lose, innervation, right?
(04:39):
Before they get flaccid, they get spastic.
Sometimes they just stay spastic.
It's like having a sensory cortex stroke.
You just get spastic.
So her jaw muscles were spastic andthen weak, so they wouldn't contract
because they were already spastic.
(05:02):
So I'm running 81 and thesefour frequencies and it's
Did those muscles just suck?
It didn't.
They did.
They just, they softened, right?
She went, I don't know.
And then pretty soon after theyrelaxed, they started for circulating.
Like they would just twitch Oh,the nerves are talking to me now.
(05:26):
I didn't think it was possible.
There are two frequenciesfor the accessory nerve.
She said, Oh, I have thispain here in my shoulder.
And it's that's the upper trapezius.
That's the accessory nerve.
Here on JJ's list, George's list.
There's the accessory nerve, so Iunwrapped it, so it just was laid
(05:47):
across the back of her neck and thebase of her skull, and two frequencies
for the accessory nerve, and shesaid, Oh, it feels funny down below.
I said, Yeah, that's the lowerpart of the upper trapezius.
It worked.
Spasticity went away.
And then, she said, it hurtsback here, base of the skull,
(06:09):
and right here, sensory pain.
This hurts.
Okay, take it apart.
This is not a cranial nerve.
When they did the surgery, he wentin between branches of the occipital
nerve, which is a C2 nerve root.
(06:30):
And I just That was easy.
C2, we know it can fixbecause that's just 396.
So we're on 40 and 396 while Iwent and looked up something.
And at the end of, at the endof an hour, we spent two hours
educating her and me about what thesecranial nerves did and how it is.
(06:58):
She had a disc bulge that was justtouching, barely, the spinal cord.
After this, that was anMRI before the surgery.
After the surgery.
Her spinal cord is peanut shapedbecause think of the position they
have to put you in to get a fiber opticscope into the base of your skull.
(07:22):
She had to be in hardcervical flexion, right?
So then there's this
pressure inflection on this disc.
And so after the surgery, the disc
pushes into the cord.
That created 40 and 10 and 81 and 10 downher left side and her right side, right?
(07:52):
So there's part of her symptoms.
That's the take it apart part.
Kevin just posted thetake it apart webinar.
That's the take it apart part.
What about your long tracksymptoms is from your spinal
cord and what's from your brain?
The first day I had 11 machines on her.
Concussion and vagus because you have toincrease the circulation of the brain.
(08:15):
Right and then what it was a thing.
Thank you, George and I'm sorry.
And thank you.
J. and I know we're going to seethese frequencies somewhere at
the advanced and I have, I willchange the advanced laminate.
Kevin just asked me 2 days ago.
(08:36):
Are your advanced slides finished?
I went.
Oh, yeah, sure.
No, just kidding.
Not just kidding.
But that means I have to talk evenfaster at the advanced and the advanced
laminate is going to get longer.
Actually, there's a whole sheetof investigational frequencies.
An end of one.
Doesn't mean that they're real, right?
(08:59):
It's I'm sorry, everybody.
It takes me at least5 before I believe it.
And that really used to annoy George.
I'm sorry.
And then by 10, you're pretty suretakes 20 before you're certain.
It's, but it gives us someplace to go.
(09:19):
And it also means that FSMpractitioners have to know.
More about neurology than mostneurologists tell your patients.
So this is a PhD in biochemistry,native IQ, just talking to her, probably
about 150 and we spent two hours.
(09:41):
Talking about what these cranialnerves do, where they come from,
why she has, why is she dizzy?
It's because her seventh nerve is,eighth nerve is, goes to here and
here, and this disagrees with that.
She went, why didn't theneurologist tell me this?
Either they don't know how toexplain it, or they don't think
(10:01):
you're going to understand it,or they simply don't have time.
Could be any of the above.
Maybe all three.
Yeah.
So that was two hours, but mostly it wasme educating me to tell you the truth.
Okay.
Remind myself, made my brain hurt.
Right.
And then, but then I had,we have living visual.
(10:24):
I didn't need a video.
I had this person next to me with the, Ican't even remember the name of the nerve.
The one that makes your eye close.
Her eye can't close.
Right.
Her eye muscles don't work right.
So she just has a patchover her right eye.
It's okay, so we, so I had living proof ofwhat each one of these cranial nerves did.
(10:50):
And then we had the book in front ofus telling us where they came from.
Okay, that makes sense, that make, okay,now I, I understand why those nose don't
work because the bleed, And the hemangiomawent to the pons, and the pons is where
all these cranial nerves come out of.
And so I educated me, I educated her,and now I'm going to educate you guys.
(11:13):
And I'm really sorry ahead of time,but it's going to be fun, really.
And so maybe I'll find some videos aboutwhat facial nerve does, and maybe I can
take patient pictures of this patient.
I don't know.
Anyway, so that's, that was, that,that was what I did the last two days.
That was pretty fun.
So you led with, you did something thatyou were surprised that it works when
(11:38):
you say these things and then you explainthe course of action that you took, I'm
sitting here going that makes sense.
That makes sense.
That makes sense.
Like you don't do anythingthat doesn't make sense.
It's not like you'rerunning frequency for the.
I don't know, 10.
Like everything probably makes sense.
I guess if I tried to bring myself there,but it makes sense what you're saying.
(12:01):
So I like that you took the leapof faith with the new frequencies
as opposed to just running nerve.
They're either going towork or they're not work.
It's okay, I, George was wrong.
And that's what I'm always waitingfor, but he's hardly ever wrong.
And it was 1st, you had to figureout how they're supposed to work
(12:22):
and what they're supposed to doand understand why her masseter.
So if you feel up and next year, likefrom this bone down to that bone,
there's a little muscle there, right?
You've got an Audi instead of it.
She's got a hole.
That muscle is just gone, right?
(12:43):
Her jaw muscle, hard as rock,and she can't close her mouth.
She has to chew soft things.
Okay, what happens to amuscle when it's denervated?
It spasms.
Rock hard.
So how do you un denervate it?
(13:03):
You increase secretions in thenerve, and you just drive the mother.
Just, I'm sorry, I know you've beenroad hard and put away wet, but come
with me now, it's gonna be fine.
You got, you have to do this now.
Okay!
Give me some ATP, will you?
Yeah, I got that.
(13:23):
So the current does the, how do I do this?
Because the nerve is asking you.
Okay, I'm willing.
You're telling me what to dowith this 81 and 292 thing.
But how do I do that?
I don't have, oh, 500 percent ATP.
I guess I can try.
And then the nerve goes, okay,so the first branch, all of a
(13:48):
sudden this started to fill in.
And I'm going.
No, and the second branch was the funone because the master was just like
a brick, and it started to soften.
So it's the, that's actuallywhat we're going to end up
doing in the neuro section.
It's pretty fun.
That is fun.
(14:08):
So I want to talk about this.
Concept really quickly because it'ssomething that we talk about in the sports
and the sports advanced is when you havesomething that's called a tight weakness.
A muscle that is chronically shortened,we say has a, so you can have a tight
weakness and a stretch weakness.
So the tight weakness is like what you'retalking about when a muscle is spastic or
(14:28):
chronically shortened, it's weak, it can'tcontract and relax and it comes from two
sort of philosophies of why that happened.
You explained the first neurophysiologicalphenomenon about innervation and the
other is the mechanical property.
So when the actin and myosin, so peoplewho are just listening, you're going
to have to, I'll verbally walk youthrough what my fingers are doing.
(14:50):
My hands are, my fingersare interlaced and.
I'm having optimal space between them.
So that would be likeoptimal actin and myosin.
So the myosin and actin can pulland that's our sliding filament
mechanism and the muscle shorten.
But if a muscle is too tight andthere's too much overlap, the actin
and myosin can't optimally shorten.
(15:10):
So you get this Chronically shortenedstate or a short or a a short weakness
or a tight weakness is what we call it.
So I would even follow it upplaying with the sarcomere.
Yeah.
It takes, the sarcomere is, you'redoing this in a lacing your fingers.
I do this with the creepy crawlies.
Yeah.
Because they have these little heads that,grab each other and walk along each other.
(15:35):
Then hold
onto the mycin.
Yes.
And so they're like, golf takes.
3 ATP to make him go like this.
It takes 2 ATP to relax him.
That's where the ATP part comes.
And in order
to go ahead.
No, I said, that's why it makes sense.
What when we use frequencies like49 and 81, that this works is
(15:57):
because there's no more juice left.
You being those vial fibrils, the energythat it needs to optimally contract
and shorten this just makes sense.
Yeah, and then the, in order to bestrong, so strong and normal, a muscle
has to be able to contract and relax.
(16:20):
It has to be able to do bothcontract, relax, contract, relax.
You can't get strong if all,if you're just doing contract.
And if it's constantly elongated,because, and I'm insistent on this.
Which makes me sort of a pain,but it's constantly elongated
(16:42):
because the antagonist muscleis chronically short, right?
So if your internal rotators in yourhip, pectineus, the brevis, the adductor
is really tight because of adhesionsin the femoral plexus, or because
(17:05):
of degeneration in the hip joint.
You're chronically short.
internally rotated.
That means your piriformis and yourglute med are chronically elongated
which makes them weak becausethey don't ever get, they're just
elongated to elongated, they don'tever get to shorten and then elongate.
(17:29):
You have to be able todo both to be strong.
So it's.
And that's why we see thesecomplexes all the time.
So we talk about the psoas beingchronically shortened and the glutes
being chronically elongated, right?
So we are sitting.
So we are in perpetual hip flexion.
(17:49):
too much.
We should not be sitting as much as we do.
So our psoas, iliacus, ourhip flexors get shortened.
And then like you just said, theantagonist is chronically elongated.
There has to be A symbiotic relationshipbetween agonists and antagonists or
flexors, extensors, internal, external.
You name it.
There's going to be an oppositemuscle that we really have to pay a
(18:12):
lot of attention to, especially whenwe work with FSM, biopic treatment.
Yeah.
It's just fascinating.
I've had two impossiblepatients this week.
One has a condition called SPG 7,and it's, I had to look it up again
after It's a genetic abnormality whereyou're missing the enzyme that puts
(18:38):
together peptide that the ribosomesneed to make a neurotransmitter.
Somehow it involves the mitochondria.
And
there's this frequency forabnormal protein accumulation.
(18:59):
And another place where actually thiswas the first place where I got in
trouble with George, because he wentthrough and came up with all these
frequencies for the inner and theouter and then this and then all these
different parts of the mitochondria.
It's whatever.
How are you going to tell if they work?
(19:19):
So I. Use the frequency for abnormalprotein accumulation, which is what
happens in SPG7, and it is incurable.
And we, she, it, I've been treatingher for three years and she hasn't
(19:42):
progressed, and she should be in awheelchair by now, and she's not.
She's still progressing, butat about three or four percent
of what she should be doing.
And she's one of the things,one of the recommendations,
we finally put her in a brace.
(20:03):
She was resisting that for fiveyears and it's look, if you fall
and break something, it's game over.
So get a brace and use a cane.
And then I had to teach her to puther cane in her left hand because
it's a right leg that's really worse.
And then it's the part where youtake one Baclofen three times a
(20:23):
day, For the spasticity, you mightconsider taking two in the morning.
There's, I, we're not gettingit with the spasticity.
We soften it and it'sbetter, but just try it.
Okay.
(20:44):
And so it's a collaboration.
Between FSM, making it betterthan it should be, and initially
we treated her spinal cord andthat took care of the spasticity.
Then after about a year, we hadto start treating her brain.
(21:08):
Yesterday we were back to her spinal cord.
And it's because the SPG7 has movedup to her arm and her shoulder.
I thought, oh, it's justyour neck and your arm.
It's the SPG7.
It's finally hit the upper extremity.
It was just in the lowerextremity for three years.
And now it's okay, so I use Google a lot.
(21:31):
Yeah, nobody on earth would know allthe things that walk into your clinic
or have to think about treating themall the way an FSM practitioner might
have to think about these things.
And then it's really funwhen somebody walks in.
My last patient last night justhas a vestibular injury and
low back pain, arthritic knees.
Yes.
(21:54):
And then you second guess it.
Is it that easy?
This is really all that's.
He came in and one of his primarycomplaint besides his knee pain and
his low back pain was, and he justmentioned in passing his balance.
One or two questions.
(22:15):
I handed him the BIVSS,his score was 48 over 18.
I said, yeah, don't even come backuntil after you've seen Rescue.
Get your prescription.
And that's when he, so now hecame back and he said can I
get my lenses made at Costco?
I said, absolutely not.
Why not?
(22:35):
It's so explaining that, butit's just a nice, simple shoulder
would be nice, and then so
many shoulders the last little while.
It's.
You're right.
I think you said that they alljump on a bus and then I have to
think, okay what am I here to learn?
I am here to learn somethingabout all these shoulders.
(22:58):
Peck minor.
Can you get out of my brain for a second?
I was just going to say the amountof times that I just start now
at Peck minor and subclavius.
Subscap always.
Yeah that's like a, but nowI feel like I have to treat
Subclavius Impact minor first.
Back
to get back.
(23:19):
So then I can go in.
Oh, okay.
I hadn't thought about that.
And then the labrum that's beenthe last part, especially where
they talk about how it happened.
My dog pulled on the leash.
Is there any way to jam the humoralhead forward in the fossa without
(23:42):
trashing the front part of the.
No,
no I think that's an important partto talk about is people will say,
Oh, I had imaging in my labrum spine.
I understand that you don'thave a surgical repair labrum.
However, there is micro trauma everywhere.
And so people who have had.
(24:03):
Dislocations, traction injuries,even though the labrum may appear
fine and intact on imaging, stillgo to torn and broken in the labrum.
2 14 is labrum or meniscus
and
I didn't like that frequency in thebeginning and now I am addicted to it.
(24:25):
I can't not never use it.
It's like the holy grailof shoulder injuries.
Shoulders, hips, knees, and it'sthank God elbows don't hurt.
Wait, do elbows.
They don't
know that I have that very peskyannular ligament that can get trashed.
Yeah, annular ligament is really
icky.
Yeah,
(24:47):
and now we have frequency for it.
And for the 34 people who are with uson the 38 people who are with us in
person on this adventure, all of youare now figuring out that you come to
the podcast with netter that and maybea glass of wine, but netter next to you.
So it's, yeah.
(25:08):
So it's necessary.
And it's not to say that youdon't know your anatomy and yes,
there's all this great softwarethat people have on their iPads and
their phones and their computers.
I'm sorry.
Nothing to me beats anatomy.
Netter.
It's like my laminate.
Like sometimes I'll and look veryquickly, but sometimes just having
the page open for some inspiration.
(25:29):
Can be very helpful.
Look at this, would 214
help JAWS?
Okay, so I have somethingto say about that.
So it's I've fooled around withit a little bit because I've been
doing a lot of work with TMJ, talkwith Mary Ellen Chalmers all the
time with our orthodontic patients.
So I've used it because I've hadan open mind about that that disc.
(25:54):
Okay.
But, and it's on one of my TMJ talks,that the disc that's in the jaw is not
the same as the disc that's in our back,so those disc frequencies didn't work.
The 710 works.
The annulus works, doesn't it?
A little bit, but for pain, sothe superior slip of the lateral
pterygoid is what attaches tothe disc and pulls the disc down.
(26:17):
Forward, but if the pain andthe bleeding and the scarring
is caused from the retrodiscaltissue, which is behind the disc
Right,
it's like this long band of Scarves thatare attached to scarves that are attached
to scarves You have to treat the pterygoidthat attaches to the disc attaches to
the retrodiscal tissue on a pathologylevel we're seeing that the tearing
the bleeding and the pain is more to dowith the retrodiscal tissue because The
(26:42):
disc in the jaw is this by concave YouMostly cartilaginous aneural structure.
So it's not innervated, so wedon't get pain from the disc.
And the other thing with thepterygoid is that the pterygoid
gets tight because it's protecting
(27:03):
loose ligaments between the toothand the jaw, the tooth and the jaw.
And once the pterygoid gets tight, itgets micro tears in the flat connective
tissue, because it's not a roundligament, it's a flat connective tissue
that connects to that disc tissue.
And when, so the pterygoid should relax.
(27:29):
When you close your jaw and let thedisc slide back, the pterygoid doesn't
relax because it's holding on fordear life because you've got a loose
ligament or you had braces or you gothit in the mouth in a bar fight or you
hit your face on the steering wheel.
And so when the pterygoid doesn't relax,doesn't let the disc go back, the bone
(27:51):
goes back, pulls the disc back, andit's that snap that causes the bleeding.
Is that how it works?
So it can, so you're talking aboutreduction versus a non reducing disc.
So when a disc doesn't reduce inthe fossa that pop and that snap,
so when it's being dragged, that'swhen that retro discal tissue leads.
(28:16):
Okay.
So it's on the forwardpop, not the closing pop.
It's the direct,
it's when you open.
When it reduces and it snaps back, becausethe retrodiscal tissue is pretty much
made out of adipose it just reduces backand it just slurps it back into space.
I'm sure the disc could get trashedin that fossil, so running torn
(28:37):
and broken would help that, butthe bleeding that happens, so
running bleeding in the adipose, Has
been
cracked, but like any type of TMJ work.
So yeah, we're good at treating thepterygoids, but running bleeding.
In the ADEPT, so 18 on A, 97 onB, has been a total game changer
(28:58):
because you don't think ofHow does the ADEPT post bleed?
The retrodiscal tissue bleedsbecause it's very vascularized.
So when there's bleeding Shouldn't there
be 18 and 162 capillaries?
Yes, I've run that, but when it's You saidit and I've never been able to unhear it.
It's not necessarily a structurethat bleeds, but if it was bled onto.
(29:19):
Okay.
So then 3 and 97 as well.
Correct.
Okay.
Okay, got it.
Yeah.
Interesting.
And 284 then right in 97 becausethere's like bruising back there.
Yes.
I will stop rolling my eyes on that slidein the core where you have all these
tissues that you treat mostly becauseI've got what a minute and a half to
(29:42):
cover that slide and it's just Ask him.
It's
and I love being asked, but when I, again,once you hear something and once you,
and it was during COVID that I took a TMJcourse and the videos that we saw of a
cadaver and they're on YouTube as well ofjust the disc and how that retro discal
tissue, which I hadn't really didn't knowmuch about, but to see how important that
(30:05):
is, how innovated it is is fascinating.
It has to be.
When you look at the innervationto the face, and that is, so you've
got motor to the facial muscles.
Pterygoid is innervatedby the facial nerve.
(30:28):
Triteminal is sensory.
Every other motor in the face.
The pterygoid has got to be facial nerve.
It's masseter pterygoid, temporalis,and whatever this little thing is.
Thank God
for Google.
Honestly, pterygoids, bothlateral and medial are inhibited
by the mandibular branch oftrigeminal nerve, cranial nerve 5.
(30:50):
What?
Trigeminal nerve is motor?
Wait.
Facial nerve is motor.
Trigeminal is not just sensory.
Trigeminal is motor.
That makes my brain hurt just when youthought you knew something you don't.
I hate that part.
Speaking of, we havesome questions to get to.
That's a lot easier thandealing with weight.
(31:12):
As I continue to google.
We're going to be disinterested by the
I'm going to
have a
hard time looking at this.
Because the mandibular branch of thetrigeminal, the mandibular maxillary
branch of the trigeminal nerve.
Lateral pterygoid innervated bymandibular nerve, which is third branch
of trigeminal nerve, cranial nerve 5.
Specifically, the informationcomes from the pancreas.
So
the mandibular branch goes upand innervates the pterygoid,
(31:35):
which is actually in theregion of the maxillary branch?
This middle part is themaxillary branch, right?
Yep.
So the trigeminal nerveis both motor and sensory?
Okay.
I'll be fine.
Branch
and vestibular nerve.
Wow.
Okay.
I want to get Mary Ellen on.
(31:56):
Okay.
Oh.
She's going to be in Phoenix.
Do you guys realize, do you realizewhat is happening in Phoenix?
Magic.
Just Reski, Mary Ellen, Jay Shaw, Eduardo.
The Julianna Mortenson Jennifer Ernst.
(32:17):
Now this, we had to shorten it becauseDiana Cross announced three weeks ago,
four weeks ago, that she's coming up.
I'm coming!
I have an hour.
And it's wait, it's all full.
Okay, I had this hour presentation andnow we're going to squash into 30 minutes.
So it's Diana Cross is comingwith more news on whatever
124 is supposed to be doing.
(32:39):
Jennifer Ernst, we have the datafrom the Feinstein Institute.
You guys, the Vegas.
The vagal tone protocol is patented.
It has enough data thatTIVIC has a patent on it.
I just want y'all to swimthat there for a second.
(33:01):
And understand why, if the onlyreason you came was for that
little 30 minute thing that we get.
Because I got 20 minutes onwhat the vagus does, and she's
got 10 minutes on the TIVIC.
Now, on the data, which is, and thenwe all have an hour with time across.
Okay, questions.
Mom had a stent placeda year and a half ago.
(33:22):
Sensorized mild electrical heart block.
That's like being a littlepregnant, but that's another.
And congestive heart failure.
Those two things go together.
I have run off a seminar.
Wait.
Congestive heart failure.
Time out.
How long has she been on statins?
I have run FSM and are greatresults for a number of things.
(33:43):
Never run heart health.
Oh, no, it's not beyondyour area of expertise.
Heart disease, all heart diseaseis inflammation, oxidative stress,
and autoimmune dysfunction.
That's problems with the heart.
Congestive heart failure.
So there's a little bit missing here in.
(34:06):
The history, which is heart block is whenthe electrical system on the left side
depends, you can have left bundle branchblock, right bundle branch block, you
can have both one or the other, or both.
Usually it's left bundle branchblock starts first and then you
get right bundle branch block.
(34:27):
It's there's this electrical wiringthat goes up the left side of your heart
that makes it go smush at contract.
And.
The wiring gets, the insulationgets thin and heart block is
just the wires don't work.
It's like your heart's been inflamedlong enough that it's dissolved the
myelin that's on these, they're my, Ithink they're myelinated nerves because
(34:51):
they're really fast with your heart.
And.
So heart block, I get that's, thatjust happens when you get old.
Congestive heart failure does notnecessarily go with heart block right?
Congestive heart failuregoes with statins.
I'm gonna guess I'm not, Ican't guess, I have to ask how
(35:15):
long Has she been on statins?
Because one of the main side effectsof statins is congestive heart failure.
It just, it kills the mitochondria in thebrain, the heart, and the skeletal muscle.
I have run FSM on her.
Heart health can't hurt anybody.
It's everybody.
(35:37):
Everybody should run heart healthon themselves once a month.
Men and women, but especiallywomen, should run breast health
on themselves once a month.
So you can have my once a month programthat combines heart health and breast
health, and you just have reducedinflammation in different tissues.
For the two sets and you put amagnetic converter on one side of
(36:00):
your chest and magnetic convert onthe other side of your chest, pick up
the breast and heart all in between.
Reducing inflammation on the heart isnot beyond the area of your expertise.
It's a no brainer.
It's there's nobody that's going tohurt as long as they don't have an
infection or infectious pericarditis.
And she doesn't have that.
(36:20):
She lives in the traditional as wellas an alternative medical desert.
Condolences.
No one else for her to see.
I'm with her right now.
She's not experiencing any symptoms.
Yay.
Was wondering if there arecontraindications or any wishes.
No, there's no reason notto run Howard Health on her.
It would be okay.
Recommendation Either one, wettowels, pulse DMF, depending
(36:41):
on how old your mom is.
Wet towels are just a pain.
Use wet washcloths oneither side of her chest.
I prefer not to run them backto front through the heart.
Either side is just fine.
So you create a field that goes throughthe heart warm, wet washcloths, and then
cover up with something that keeps warm.
(37:02):
If you have a magnetic converter,use that because the heart likes
pulsed EMF and it works just as well.
If she is not on 400, 400, yes, I said 400milligrams of CoQ10, if she's on statins,
And her cholesterol was 350 or 400.
(37:24):
Statins are fine.
I'm not going to fightwith anybody about it.
But she has to be on 400 milligramsof CoQ10 if she's still on statins.
And D ribose is what Mark Houstonuses for congestive heart failure.
The other thing with congestiveheart failure is The heart doesn't
(37:46):
use, like your skeletal muscles usesugar to make the mitochondria work.
The heart doesn't use sugar.
There's not enough energy in it.
It uses fatty acids.
There's this fat pad on your heart,like right in the middle of it,
and the heart just doesn't use it.
The body just replaces that,and your heart uses it.
(38:09):
As you get insulin resistant,everybody that's over the age
of 30, look down at your tummy.
And if your tummy is sticking out justa little bit, that's insulin resistance.
Embryologically, the fat that'sin your heart fat pad is the same
embryological origin as the fatthat's in your tummy that gets more
(38:32):
insulin resistant as we get older.
So type 2 diabetes is insulinresistance to start with.
So you want to run insulin resistanceor at least inflammation in the adipose.
It's part of heart health, andthat's why it's part of heart health,
because you want the fatty acids inthat fat tissue to be more available
(38:58):
for the heart to use for energy.
Congestive heart failure is
the muscle can't get enoughenergy to contract properly, so
it gets weak and it gets big.
It tries.
But that's why it gets big.
Sorry.
This is, that's no,
(39:18):
but she just said the person thatwrote that said that she bought her
on CoQ10 sometime after her stent.
So
that's
great.
Okay.
All right.
So it's make sure.
So look at her cholesterol.
That's the other thing.
If her cholesterol is 200when they put her on statins.
That's dumb.
It's 200 is normal.
If her cholesterol is 400, thenyou can make a case for it.
(39:40):
That's it's a wholedifferent conversation.
Sylvie patient who had a resection.
Oh, this is okay.
Resection of an acoustic neuroma.
Loss of hearing and dizziness.
No fooling.
What is the frequency ofthe eighth cranial nerve?
I'll let you I don'thave it in my phone yet.
JJ is going to show mehow to put it on my buddy.
(40:01):
Oh wait.
I have a thing.
I, somebody, he sent, JJsent me a text with that.
There it is.
Here it is.
Eighth nerve.
Vestibular cochlear nerve.
114, which is also the frequencyfor the thymus, which is why I
(40:27):
had this difficulty with Georgia.
It worked anyway.
And 137 is the vestibular cochlear nerve.
Yeah, and acoustic neuromas are,
an acoustic neuroma is a benign tumorthat gets onto the eighth nerve inside
(40:50):
the ear, or maybe a little behind theear, but you have the facial nerve and the
eighth nerve does the vestibular system.
You have the vestibular edge ofit and the hearing end of it.
That goes to the cochlea.
That's the eighth nerve.
But the seventh nerve that doessensation to the face, right?
(41:12):
Isn't that something?
Oh, no, go away.
Oh, Piper.
I'll tell you later.
Yeah.
Acoustic neuromas when they go inand take the neuroma out, the damage
the nerve and it's if she has loss ofhearing, she's dizzy because there's no
nerve going to the vestibular system.
Sylvia, I don't think you can treat it.
(41:33):
They take the tumor out,but they cut the nerve.
And it's, this is normal.
It, the dizziness usually goes away.
The hearing never comes back.
They're always deaf in that ear.
And if you're lucky you can try 40 and 44.
You can try If there's no nerve,I don't know how you can increase
(41:57):
secretions in our nerve that's not there.
And then the part of the brain that's,that does vestibular, somebody tell me,
I think it's the temporal lobe, right?
It's just, it's like just inboardfrom your ears and that's the only
thing there is the temporal lobe.
(42:18):
So it should be the temporal lobe.
So we have a frequency for that.
You can try that.
They, yeah, that's a tough 1.
You got the frequency see if it works.
Okay.
There's a leaf.
You covered George's cranial nerve.
Any update on the motor center numbersand exactly which centers they represent?
(42:38):
Absolutely.
Totally.
100%. No, I've neverused the motor centers.
I use 92.
I don't even know what the motorcenters are or where they are.
Or why they're even there on thelist that we have motor centers.
We have sensory centers.
Are they in the brain?
Are they appear on thesensory motor cortex?
Are they sections of the sensory motorcortex as you step down the homunculus?
(43:04):
Don't know.
Sorry, Leif.
I'll see in a couple of weeks.
No.
Okay.
Is there any, anybody have aquestion that I can answer?
Those were good.
Did we finish all the ones that
Oh, good.
Yay.
Yeah, and the chat, we'renot too chatty today.
So that's good.
Kevin did the chat part.
(43:27):
Okay, that was, we went.
Nowhere near my listtoday, but that is fine.
You got 10 minutes to start on your list.
I love your list.
It keeps us grounded.
The list was piggybacking offof after we got off last week,
we did a lot of spine disc.
One thing that always the pathology thatalways seems to come up every so often,
(43:52):
whether it's on the podcast or at thecourse, or I get an email is stenosis.
And, it's that face and a lot of peoplehave stenosis and for some reason people
seem to think that FSM is contraindicatedor it's going to make it worse.
It's not.
So let's just say that.
And FSM can, it can still be helpfuleven when there is stenosis present.
(44:16):
I think the only time we had thelike cautionary tale was with 40.
With anything you want to do withthe spinal cord, so this lady with
this disc bulge that goes into themiddle of her cord, that is stenosis.
Everything that I run from her neckto her feet has to be alternating.
I can't polarize anything.
(44:37):
As far as I can tell, when you polarizethe current, it appears to, just
clinically, it's like it increasesthe, the spinal fluid flows up the
dura, around the brain, down thecenter, then back up the front, and
then around, and whatever the flow is.
(44:59):
Okay, so when you polarize.
The current positive.
The first thing that happens is the pain.
If the patient has stenosis,they will say, Oh, hurts in
between my shoulder blades.
And then the next thing theysay is, Whoa, I got a headache.
(45:22):
All right.
Now, if you look at what does thatmean, if you take that apart, if the
spinal fluid is trying to come downand the stenosis blocks it, right?
What is it?
What is the spinal fluid pressing on?
It's the disc annulus.
So there's pressure on the annulus.
(45:43):
They get pain in betweentheir shoulder blades first.
And then as the pressure builds up.
So you got a hair in the drain here,can't get down, pushes on the annulus,
you get pain on the shoulder blades,and then it fills up and fills in the
ventricles and increases intercranialpressure and they get a headache.
(46:07):
Stop the current, make it alternating.
That's the first thing you can doif you catch it soon enough and
you can tell I'm speaking from
Experience So you make it alternatingfirst if that doesn't work and
they still have a headache.
You just have them set up I hadone that just was really bad.
She had stenosis, I didn't suspectand So she sat up and I ran
(46:32):
just inflammation in the spinalfluid And got her headache down.
Now, if you know from imaging that theyhave stenosis, you treat the spinal cord.
So you already know that the spinal cordis at least touched by the disc annulus
(46:52):
that and the disc annulus may be leaking.
So you have to run 40 and 10to get their body pain down.
And if there's enough compression, youmight have to run 81 and 10 to get the
tone down in their legs and their trunk.
And you have to do thosealternating neck to feet, but then
(47:15):
sometimes stenosis comes from facetsin the back, arthritis pushing on the
backside of the cord, and the disc,which is calcified and fibrous and
nasty, it's pushing on the front.
So what would you do?
You'd run.
What where is the pushing on the backcoming from 217 hard calcium or 91 in
(47:44):
the periosteum in the joint capsule.
On the back side and on thedisc annulus on the front.
So you set up from front to backat the level of stenosis, front to
back at the neck to treat the discannulus and the facets in the back.
(48:07):
If the stenosis is in thelumbar spine, it's not a deal.
The lumbar spine stenosis isalmost always at L3, 4 and as long
as they can feel their crotch.
Urinate defecate.
As long as they can do thosethings, the stenosis in their lumbar
spine is just about nerve pain.
(48:27):
So you treat nerve pain and it, there'sno spinal fluid problems there at all.
'cause there's no spinal cord at all.
Three spinal cord stops.
It's at T 10.
I just, it's just, it's not there.
So L three stenosis is easy.
You just keep the nerves and makesure they can feel the crotch.
If their crotch gets numb,you call the surgeon.
(48:50):
That's just, I'm alittle hyper today, huh?
No, it's good.
It's good.
It's good energy.
I'm just like, like everybody else.
I'm just like taking it all in becauseyou always hear something different, but
yeah, I just wanted to, I said, stenosisis always something that comes up and
it's not contraindicated, but thereare just things to be aware of and the
(49:11):
way you explained it, it makes just.
total sense of why we are aware of it.
It's like an acute injury whenthere's a lot of inflammation.
We don't want to ram a sharp,positive current through an
already hot bed of activity.
I just think about that.
Cynosis, just like an acuteinflammatory response, there's so
much activity, throwing a lot ofanything more at it is just chaotic.
(49:37):
You just have to think of cervicalstenosis like hair in the drain.
It just it interferes with spinalfluid flow and it backs it up.
I have to figure out a way to talk aboutit so that it doesn't scare people.
Apparently, I have too many slidesabout it in the core because
people, you don't see it very often.
In the first place.
(49:58):
No, I see a fair bit.
Do you?
The last couple years I definitely have.
Remember you asked formore difficult patients.
I did.
I remember that plane ride.
I remember.
I remember warning you about that.
It was answered and I'm gratefulbecause I'm learning so much and maybe
(50:18):
that's why I'm seeing that learningall about shoulders all over again.
I don't know.
And at four o'clock in twotwo and a half minutes.
Yes.
I get to do intro to FSM webinar.
And it's, we've got likefour, 300 and some odd.
400 people signed up for itofficially, but anybody can join in
(50:41):
and it made me go over introduction.
Why would you do FSM?
And there's this 1 slide thatI just had so much fun with.
It was really cool
for
14, 414, 414 people.
Really?
That's going to be on.
Yeah, it's and it'll end up onYouTube, but the intro is really fun.
(51:02):
And I think we're going to put it onthe resources page for practitioners
so you can send it to people.
How does FSM work?
Why would I use it?
And I only used with two exceptions.
I only used published papers.
Everything we have is digital.
Data.
Oh, can you give the link?
And there's Kevin.
(51:23):
I was just going to interruptto say, Kevin, can you, but
so we get off this one.
Yeah, I know.
Are there Dan those patients moresusceptible to stenosis in some ways?
Yes, because they'reconnective tissue is looser.
And they're more likely to getdysbulgias, so it's possible, right?
(51:44):
And then there's the wholetethered cord thing that happens
with Ehlers Danlos patients.
There's a higher propensityfor tethered cord.
Go looking for tethered cords.
On an MRI the tethered cord sofar has been really easy to treat.
It's, the spinal cord shouldend up, should end at about T10.
(52:05):
When you have quote unquote tetheredcord it's down at about, L1.
It's just, it just stretches thecord a bit, and so you treat the
discs, and you treat the cord for 40.
And it's
four o'clock, and I'll let you get offhere, and get on the new one, and so glad
you're doing it, and thanks everybody.
(52:27):
Thank you.
Some of you guys will probably needto log off of this one, and get on the
other one if you're staying the extrahour, and we'll see everybody next week.
See you next
week.
Bye.
The frequency specific microcurrentpodcast has been produced by
Frequency Specific Seminarsfor entertainment, educational,
and information purposes only.
The information and opinion provided inthe podcast are not medical advice, do
not create any type of doctor patientrelationship, and unless expressly stated.
(52:49):
Do not reflect the opinions of itsaffiliates, subsidiaries, or sponsors, or
the hosts, or any of the podcast guestsor affiliated professional organizations.
No person should act or refrainfrom acting on the basis of the
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(53:11):
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