February 12, 2025 • 52 mins
In this episode, Dr. Carol McMakin and Kim Pittis discuss Frequency Specific Microcurrent (FSM) protocols for treating nerve pain, including post-herpetic neuralgia and ataxia. They explain the mechanisms behind nerve inflammation, polarization, and scarring, and shares case studies demonstrating effective FSM treatments. The conversation covers topics such as treating the psoas muscle, addressing ticklishness, and managing complex neurological cases. Dr. McMakin also provides insights on using FSM for patients with cancer and emphasizes the importance of comprehensive assessments in identifying the root causes of pain.
01:13 Understanding Voltage-Gated Ion Channels
02:42 FSM and Nerve Pain Treatment
05:59 Case Study: Cavernous Hemangioma
07:42 Nerve Pain Treatment Insights
12:03 Treating the psoas Muscle
17:04 Scarring and Tissue Treatment
18:14 Case Study: Knee Pain Post-Surgery
23:14 Holistic Approach to Pain Treatment
28:12 Addressing Ticklishness in Treatment
30:23 Nerve Inflammation and Pain Management
34:18 Case Study: Ataxic Gait and Neurological Concerns
42:02 Chronic Conditions and Treatment Approaches
47:08 Treating Cancer Patients: Guidelines and Considerations
50:04 Upcoming Courses and Final Thoughts
As medical practitioners, it's crucial to constantly refine our approaches to treating conditions like nerve pain, ataxia, and other related dysfunctions. In a recent episode of the Frequency Specific Microcurrent podcast, various experts, including Dr. Carol and Kim Pittis, shared valuable insights that can benefit practitioners seeking to enhance their understanding of nerve-related treatments.
Understanding Nerve Pain and Treatment Modalities
Dr. Carol emphasizes the complexities of nerve pain treatment, explaining how conditions like postherpetic neuralgia and traction injuries can influence nerve function. Practitioners are encouraged to consider the underlying causes of nerve numbness or hypersensitivity. Dr. Carol suggests the combination of polarizing frequencies and specific microcurrent protocols to address these issues effectively.
One primary focus is on using combinations such as 40 Hz and 396 Hz to alleviate inflammation and enhance nerve conductivity. Complementing this approach, Dr. Carol underscores the importance of addressing the surrounding scar tissue, often by applying frequencies such as 13 Hz to reduce adhesions and further alleviate nerve discomfort.
Techniques for Addressing Complex Cases
Throughout the podcast, both Dr. Carol and Kim Pittis share personal case studies to illustrate treatment strategies. For instance, Dr. Carol describes a particular case wherein a patient developed knee pain post-tummy tuck procedure. Here, the importance of a holistic approach is evident. Realigning the patella and addressing underlying scar tissues from the surgery was pivotal. Additionally, careful inquiry into the patient's history—identifying past surgical or traumatic events—can help in piecing together effective treatment plans.
Addressing Ataxia and the Importance of Neurological Assessments
In discussing ataxia—an often misunderstood and misdiagnosed condition—Dr. Carol stresses the potential need for thorough assessments beyond standard examinations. She shares anecdotal experiences highlighting that an ataxic gait may stem from spinal, rather than purely cerebral, origins. This revelation points medical practitioners toward the need for detailed diagnostics, including MRI assessments of both the brain and spinal regions.
Promoting Safe and Effective Treatment Environments
Kim Pittis touches on the critical aspect of patient safety and comfort. Ensuring the nervous system perceives treatment interventions as safe is paramount for achieving lasting results. Furthermore, practitioners are encouraged to adopt a patient-centered approach, conside
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(00:00):
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Last week, I ran all over yourlist, so I figured this week that I
would let you and your list start.
(00:24):
Okay.
I almost wish I, I do think weactually have to go back to last
week for a little bit because itprompted, I don't know about you,
but I got a bunch of emails sent.
There's one, oh.
Before we go any further.
Okay.
I have to clarify somethingthat you said on January 22nd.
Somebody wrote something in, soI didn't know what it meant and
(00:48):
I didn't go back and listen toit, so I'll try to paraphrase it.
Okay.
It was regarding postherpetic neuralgia.
So the quote said something like,polarize the heck out of it.
The curve makes the voltage gated channelsin the nerve membrane flip over, forcing
them and then dot just knocks into them.
(01:08):
They would like a little more explanation.
Oh, first you have to understandthat the first you have to Google
voltage gated ion channels in nerves.
I can't describe that there.
Nerves, nerve membranes, fire.
You get a, action potentialbecause there's sodium on one
(01:35):
side and potassium on the other.
And I'd love to pretend thatI remember which ones, which I
think sodium's on the outside.
And anyway, so when the actionpotential comes along, the way it is
propagated is this channel opens andsodium runs in, and potassium runs
out, and then it flips over again.
(01:58):
And it does that.
That's how the actionpotential goes along.
So if you've ever had anerve conduction study.
There's this little spike that goes well,that is a sodium potassium voltage gated.
So voltage gated means that there's a,
(02:20):
that channel that lets the sodiumin and the potassium out and
lets the actual potential go is.
Operated by or influenced by voltage.
All I know is that Terry Phillipscalls 'em voltage gated ion channels
and he knows a lot more than I do.
So I, that's what I call him.
(02:42):
And then, so I think, this is myhypothesis, that one of the ways that FSM.
Treats nerve pain so easily is,there's if you run 40 and 3 96,
(03:04):
if a nerve is inflamed, it stopsconducting the inflammation and
the nerve stops conductivity.
That is pretty good forrelieving nerve pain and it will.
Also improve
(03:25):
nerve atrophy in a muscle.
But to get a nerve from numb
to hypersensitive to normal, firstyou have to relieve the inflammation.
'cause that's what made itnumb in the first place.
(03:45):
But then.
That will help the muscles likeshow up, but to get the muscles
to contract, whoa, that's weird.
That's good.
Get the muscles to contract and to getthe nerve from hypersensitive to normal.
(04:06):
Sometimes you have to run increasedsecretions in the nerve and
the nerve has to be polarized.
It's nerves are polarized.
We're positive, centrallynegative distally.
That means there's more electronsout here than there is here, right?
'cause electrons are negative.
And I think of polarized positiveincreased secretions, polarize
(04:33):
positive, reduce inflammation as.
Forcing the nerve membrane to work.
And it's just, when I say drive it,I'm, that's exactly what I mean.
It's yeah, you don't get to vote.
You are going to work.
(04:54):
Yeah.
This is how this goes.
And as long as the nerve isstill there, it usually works.
Yeah.
If somebody's cut the nerve.
Or if the virus has completelydestroyed the nerve.
So if you have postherpetic neuralgia andthe tissue is numb and painful, there are
(05:17):
times when the nerve has been destroyedby the virus, like it's just not there.
That's when you have basicallyphantom limb pain in your rib, right?
You're on 40 and 89 and hope for the best.
Post-traumatic neurology is not easy.
(05:38):
And it's like the more youknow about neurology, the
more you know you don't know.
Yeah, it's I, one of the ways I wentdown the rabbit hole and screwed up the
advanced list today was trying to addthe frequencies for the cranial nerves.
(05:59):
Because I finally, after 30 years,had a patient that had cavernous
hemangioma that burst, and cavernoushemangioma is like a tangle of blood
vessels that are pretty fragile.
First, she had a bleed that wasa hemorrhagic stroke, which had
(06:19):
two little hemangiomas, one in hercerebellum and one in between the pons.
And the fourth ventricle, and thenthe surgeon said, so six months
later, she recovered from the stroke.
Then the surgeon said, those,he angios are still there.
They could go any time, sowe should take them out.
(06:42):
Spend a year and a half since he tookthem out and she has deficits in.
Cranial nerves 3, 4, 5, 6, 7, 8, andnine that have just not recovered.
And that's, I found out thatGeorgia's experimental frequency
(07:03):
for the facial nerve works.
Yay.
The accessory nerve kind of works someof the frequencies on his list for.
Two of the cranial nerves are ab pairs,and I'm not sure what to do with those.
So it's I'm gonna put it in, but it, thequestion is, how often do people see these
(07:30):
kind of really esoteric neurologic things?
Nerve pain, everybody treats,at least I hope they do.
Yeah.
So
that's.
That will, that phrase will alwaysbring me back to my first core.
When you say nerve pain is gonna beone of the easiest things that you
treat, and I'm always a nerd and I'malways sitting either in the front row
(07:52):
or I'm sitting in the very back row,but this course I was sitting in the
very front row and I remember lookingaround being like, did anybody else
just hear her say, nerve pain is theeasiest thing that we're going to treat.
It's, it is, it's the easiest thing we
treat.
Can you, while we're on the nerve,and I'm gonna derail my own list right
now, but since we're talking aboutit, I thought I would just jump on it.
(08:15):
When you have somebody, can youexplain to, maybe there isn't an
explanation how numbness goes to hyperaesthetic and then it goes to normal.
Oh it's like the reverse process,when a nerve first becomes inflamed,
it becomes hyper aesthetic, right?
(08:36):
Hypersensitive.
So with a pinwheel, you havesharp or prickly, you have
icky, and then you have numb.
That's the progressionof nerve dysfunction.
If a nerve is numb, if you'relucky, the nerve is still there.
(08:58):
It's just so messed up thatit can't conduct anything.
Right.
Sensory is easier for us than motor.
Yeah.
But we can in, becausemost nerves are mixed.
Sensory motor, the ones that we dealwith in the arms and legs and back.
(09:19):
So when you go from normalto hypersensitive the next.
Stage of dysfunction is numb whena nerve is, when an area is numb.
So I was treating a, an osteopath ata medical meeting who had open heart
(09:41):
surgery and he had a brachial plexustraction injury and his left arm where
his arm fell off the board as theywere doing the open heart surgery.
And he said his hand was so numb.
That he would blister his fingerstaking a cup out of the microwave
'cause he couldn't feel it.
So I went, that's not thathard, is it okay if I treat you?
(10:08):
And he went, okay.
And he is looking around the booth andI just wrapped towel around his neck
and wrapped a towel around his arm.
And we talked about his kids and hisgrandkids and his open heart surgery and
my open heart surgery and blah, blah blah.
And after about 30 minutes.
His hand and his c.
(10:31):
Where I used the pinwheelon him and he went, ow.
I said, oh, that's great.
He said, what are you talking about?
I said it was numb aminute, 30 minutes ago.
The next step back towardsnormal is hypersensitive.
What this mean is the nerve meansis the nerve is still there.
It's not dead, it's not broken.
(10:52):
It just has to be repolarized.
It.
If it goes from numb to hypersensitive,it will eventually go back to normal.
May or may not stay there.
Traction injuries are the easiest becausethere's no perpetuating factor, right?
It just got stretched.
And the stretching, if you thinkof it, you take those voltage
(11:14):
gated ion channels and you justrip 'em apart so they don't work.
That's a nerve traction entry.
That's how I think of them anyway.
And then nerve traction Injury.
Do you focus on 1 24 first?
Nope.
Just 40.
Just 40.
And then 81.
I guess I could do 1 24.
I just never thought of it.
'cause I never needed it.
(11:35):
So it might help and I couldjust be, a creature of habit.
And maybe all it needs is current.
Yeah.
But I, it's, it has always been40 and 81 with nerves for me.
So yeah.
Yeah, those are the very fundamental,basic factors I think when
(12:00):
we're dealing with nerve pain.
Okay.
So that's that part.
While we're on pain though, we dida lot of visceral talk yesterday.
Or last week and yesterday.
What's that?
I said it seems like yesterday.
It, it does.
And I was thinking because I'mgetting treatment on my hip right now,
(12:22):
and people are against me bringingmy machine to my own treatment.
So I'm treating myself first at homeor in the car before I get Yeah.
Treatment done.
They won't let you bring your own machine.
Okay.
Okay.
That's, nevermind.
(12:42):
It's okay.
So when people are trying toget into treat your SOAs, you
know where I'm going with this.
How do you treat theSOAs without 13 and 60?
It's just insane.
Why?
Why would you let them do that to you?
What am wait.
(13:03):
Go The time.
This is my, I'm changing hats now.
Okay.
To my patient advocate hat?
Yes.
My patient advocate hatis, they work for you.
Correct.
And so the conversation is, I wantyou, we both agree that my SOAs needs
working on, I'm going to run a frequencywhile you're working on my SOAs.
(13:27):
That should make workingon my SOAs easier.
They don't need to knowthat it's 13 and 60.
And if they say no, youfind a different therapist,
which is why I've been on the run.
Okay.
Literally, sorry.
It just,
no.
No, I, and I give peoplethe benefit of the doubt.
The problem is not really the problem.
(13:48):
It's the eyeopening experience ofknowing a, how deep the SOAs is.
Let's just think about this fora second, and iliac is, okay.
So SOAs attaches onto the lumbar vertebra
from the
front.
On the front.
And people are taking an anteriorapproach to go through everything that
(14:12):
is on top, and nobody is taking theirtime and they're just using force.
And all I can see is the microtraumathat is happening on top of everything.
And I, like I said I've been on the run
the thing that was fun with this guythat we got on videotape last week,
(14:36):
that he's got just quite a tummy on him.
But even with that, if you roll off ofthe rectus abdominis and the omentum
So you start in the middle andI'm on his right hand side.
Okay.
And.
You come to a ditch, you come toa soft spot, and that if you hit
(15:02):
it in the right place, you'reon the lateral edge of the SOAs.
Because it goes, thething is this wide and.
So you go on the lateral edge ofthe SOAs, you are on 13 and 60,
and underneath all the visceralorgans, you don't go through them.
(15:24):
You go lateral to medial and you hit theSOAs and you could, or the ureter, and
you can tell because the muscle splintsup and the patient makes scrunchie faces,
right,
and you just back off andwait for it to soften.
Then you just roll it medial and whenit gets stiff again and they make s
(15:45):
crunchy faces, you stop and you wait,and then it softens about a millimeter,
and then you roll it another three,four millimeters, and you wait.
It's, so there's no you, the,
yeah.
There's, in a perfect world, that wouldbe the approach that everybody would take
(16:08):
to treat the SOAs or the iass, however.
I'm here to say that is not theapproach that some therapists take.
And why would
you let them do that to you?
I don't let them.
I have left.
I I have said I don't think thisis the right approach for me.
Can we move on to something else andI'll get somebody else to work on my
(16:28):
So as I promise and that someone is me.
I'll see you in March.
Yes.
Yes, that's that, that'll work.
Yeah.
But it got me thinking because Idon't know about you, but when I get
worked on, I start over analyzingwhat are they on, what are you doing?
(16:50):
Why are you doing what you're doing?
And I'm thinking, why didthe tissue get like this?
Because I am a good FSM clinician,so when I'm treating others, I'm
thinking, how did we get here?
So when it comes to scarring,like scarring is one of my
obviously favorite frequencies.
(17:11):
We talked about this,I think, podcast one.
If you were on a deserted islandand you could bring five, eight
channels with you, I think 13would easily make that list for me.
Am I freezing or are you freezing?
I don't know.
I'm not freezing.
You're freezing on andoff, but not for very long.
Okay.
(17:33):
Maybe my seats.
It's electronic.
It's electricity today.
But now that I'm thinking more andmore, how did the scarring get here?
Why did the scarring get here?
13 isn't the holy grail that it once was.
It's necessary and it's useful becauseit will help with the adhesions and
(17:56):
it will help with the scar tissue,but I have to think more now.
How did we get here?
Was something inflamed first?
Did something, tear first was somethingtraumatized first was something.
I've got one for.
Patient's presenting complaintis swelling and pain.
(18:19):
She's a runner.
Presenting a complaint isswelling and pain in her knee.
And the MRI shows the MRI reportsays an intact meniscus, but she
has pain on the medial side ofher knee, so I don't believe it.
And conia patella.
(18:39):
When you look at her knee, it's acentimeter larger than her right
knee, and the patella is pulledover to the lateral side, so the VAs
later lysis too tight pulling it out.
The quadriceps is too tight.
(19:02):
She's got hyperesthesia in allof the cervical dermatomes and.
Is very tick Oliver.
It's this is interesting.
So I'm working on her leg andfind out that her hamstrings
are like guitar strings.
They're really tight.
So the first visit I ran40 and 10 and 91 and 10.
(19:25):
Why are you doing that?
Because your hamstrings and yourquads shouldn't be that tight
and the knee pain didn't start.
Until after she had a tummy tuckand liposuction, and the nice man
did a liposuction on her back,and L one was numb from where
(19:49):
the incision was in her abdomen.
But L two and three and fourwere all way hypersensitive.
She said, I want you to fix my knee.
And I went unfortunatelyyour knee is not the problem.
It's the symptom, but before thesurgery you didn't have any knee pain.
(20:15):
And after the surgery, your patellais tracking to the left, and I'm
thinking to myself, you really needthe Livermore to go to Livermore and
see Kim, but she's got two kids and shethree kids and she doesn't have time.
So I had to channel you and I thought,wait a minute, what did he do?
(20:40):
He.
Did a liposuction around the lumbar spine.
And L two was, I think thetechnical medical term you use
is pissed off, was irritable.
That would make the vastlateralis cranky as well.
(21:01):
So the day we made the most progress.
It was the next day when I ran 124 and the meniscus for an hour.
I ran so torn and broken repair and healedthe meniscus 'cause she has joint, she has
(21:24):
pain at the joint line on the medial side.
The only thing that does thatis the meniscus and then the.
Patella is just gettingground down in the groove.
So I did inflammation and I thinknecrosis in the patella, but
inflammation in the patella for an hour.
(21:46):
Inflammation in the periosteumfor an hour, and in inflammation
in the nerve for an hour.
So she had four machineson her left side and on.
I went over on her right side.
And started taking apart the scar tissuefrom the tummy tuck and liposuction.
(22:08):
Right.
And at the end of it, for thefirst time, I think it was the
third session, she got up and she'swalking around and she's so excited.
She said, it doesn't hurt.
It's still half a centimeter bigger.
They're still diffuseswelling, but it didn't hurt.
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They did an MRI of her knee.
Nobody's, nobody, none of the physicaltherapists, none of the surgeons,
none of her gps, nobody that's seenher has te tested her sensation.
Nobody has done her reflexes.
(22:48):
So that's, that is why, that's the otherreason FSM practitioners are special.
It's like you can learn anawful lot with a pinwheel.
Yeah it's been a huge adjunctto my assessment findings.
I'm biomechanic geek.
I'm also adding fascial linesand that kind of movement
(23:09):
pattern in, and, everythingcompliments the other thing, right?
The thing with FSM is itforces you, allows you.
To think outside the box andcross reference, and you said it
before, it's pattern recognition.
So once you see something, onceit's impossible to unsee it, and
(23:31):
then you start seeing it everywhere.
So I do think it it always hasbeen beneficial when somebody
comes in with pain in their kneeto not be so myopic and just look
at the knee to do your assessment.
And think, could it becoming from the hip?
Could it be coming from the back?
Could it be coming from?
(23:52):
And the other question you ask is,what happened just before this?
I had a tummy tuck in Novemberand I got knee pain in January.
Those that, that, those are not unrelated.
It's not, what is it?
(24:13):
Be before, therefore, becausenot necessarily, it's not
always this happened before.
Therefore, what happened afterward?
It has been caused bywhat happened before.
But in medicine at least,they're usually related,
right?
Yeah.
Side note, I've been binge watchingthis old show on Netflix called The
(24:36):
Resident, and I love medical dramas.
It's called the what?
The Resident.
Oh, good.
Yeah.
Okay.
I've never seen that.
He's an internal medicine doctor and heputs together he does pattern recognition.
I just keep thinking, he wouldbe so good at FSM and I realize
he's not a real doctor, but Icredit that type of thinking.
(24:59):
I value that type of thinking somuch more because, and you've said
it time and time again, you don'tthink about these areas because before
you never had a tool to treat it.
So who cared about the Durham really?
Yeah.
Who cared about the periosteum, whocared about the fascia, who cared
about the mentum, we knew it was there.
(25:20):
But the respect that you have ismore profound, I guess with FSM
you have a way of affecting,you have a way of affecting it.
So I just I love my dearfriends who are fashionistas.
They think it's the fascia.
They think it's the muscle.
(25:41):
But, and this is why I say, with allhonesty, I'm a terrible chiropractor.
'cause I don't think bones do anything.
They either move or they don't move.
And if they don't move, there'sa reason they don't move.
Muscles move bones.
Nerves move muscles.
The brain moves the nerves, right?
(26:01):
And it's like some.
You just have to find outwhere along that chain Yes.
Things went wrong.
Yeah.
And I don't think that is far off.
I think a lot of therapists do, whenwe do our assessment we're trying to
figure out why isn't this working?
Why is this not firingthe way that it should?
(26:22):
But now you just have so muchmore support in your treatment
plan after figuring out the cause.
Yeah.
We have, I've seen biomechanical feedback.
People do some amazing things here,push on this and then do that, and
then do this, and then do that, andwhatever the problem was goes away.
(26:45):
The question is how long will that stay?
Did you fix it?
Strain, counter strength.
Great.
Terrific.
Yeah.
Will it stay fixed?
I don't know.
It's, yeah,
The staying fixed ismy, it's my love affair.
That's where I I takethat part very seriously.
(27:07):
I guess once you start getting goodat what you can do with your hands in
the clinic the real challenge is howcan we keep these results as permanent
or as long lasting as possible?
And I do believe with everything in myheart that it comes down to feeling safe.
Body has to feel safe tocreate the movement and the
contraction, otherwise nothing.
(27:29):
So the real work, I think, beginswith so I think, yes, what's
happening, how did we get there?
And then after that is.
How can I create a safe environment?
How can I get this nervous systemto believe these changes are safe
and the movement is gonna be safe?
And this lady with the knee said, I'mjust, I've just really ticklish while
(27:58):
ticklish is, goes up the pain pathways.
So when I did the sensory exam, she washyper aesthetic from C two to S two.
Like she had two nerves thatwere not hypersensitive.
Wow.
So I ran 40 and 10 and 40 and 92, andthat wasn't till the second day that
(28:25):
I needed to run 81 and 10 toloosen the hamstrings, but.
Safe is ticklish is not safe.
So she said, how are yougoing to make me not ticklish?
And I said we just talk to thesensory cortex and tell it's safe.
(28:47):
We just dial it down a little bit.
And so you run 40 and 92, quiet, thesensor motor cortex, and I've never.
Not work.
It's like I couldn't touch her abdomento work on the liposuction scar
(29:08):
'cause I couldn't touch her abdomen.
She's so tickly.
You can't treat somebody that'slevitating, so I ran 40 and 92 for,
I don't know, it took 20 minutes,which is a really long time.
Usually it's two or three.
That's what made me have 40 and 10.
It's interesting.
Yeah.
So it's like the body had to befeel safe and there's something
(29:33):
about nerve hyper irritability orhyper sensory awareness, whatever
tickler is about, and that is unsafe.
Yeah.
And there used to be a trick thatwe would do, especially again, it's
like the SOAs going into the abdomen.
(29:55):
If they were ticklish goinginto, rec femme area, iliac is
to put their hand down and thenyou put your hand on top of it
that works.
Yeah.
But.
It's just nicer if you don't haveto involve more things on top
of where you're trying to go.
Sometimes.
There's a lot of questions.
(30:15):
One are like a couple case studies.
So I just, I wanna jump to summersreally quick because we're on the
nerve sort of train right now.
She says on nerve inflammation,you said 43 96 polarized.
And are you also seeing81 3 96 polarized too?
Just want to be sure I understand that.
I have a client withserious leg nerve pain major
(30:36):
fasciculations from back surgery.
So I imagine scar tissue involved too.
So 13 3 96 2,
Run 40 and 3 96 first so that youcan touch the nerve and then take the
scarring out, and then you can try.
(30:57):
Fasciculations is.
When the nerve is, whenthe muscle is denervated.
So then you do 81 and 3 96,or even sometimes 81 and 10 to
get rid of the fasciculations.
That's a, that shouldn't be athing after low back surgery.
It's, that's curious.
(31:18):
Yep, you're on the right track.
Keep in mind, you mighthave to run 81 and 10.
I know it sounds like I know whatI'm doing, but you guys understand
I make this up as I go along, right?
When it comes to nerve pain,we just talked about two,
two a channels, 40 and 13.
How do you discern which one to use?
(31:40):
Oh, you always use for 41st becauseany nerve that's scarred is inflamed.
Yeah.
So one of the.
Techniques and research for creating nervepain or simulating nerve pain is they go
in and they just tie a knot around it.
They put a piece of thread around.
(32:00):
It basically creates scar tissueon the nerve, so you have to
run 40 so that you can touch
nerve.
And then on the secondit was 13 and or 81.
But, and then you follow the nervealong its path and release it because
(32:21):
it can be scarred just about any place.
What'd you do
with your hand?
Oh I burn myself.
You have a bandaid on.
I burnt myself.
Oh, okay.
As long as, yeah, I got actuallygot a cold, a flu, a sinus
infection, all those things.
(32:41):
It's the bug is that's going around.
Just in case it's viral.
I've been running long covid every night.
Yeah.
And
then just in case it's bacterialon top of viral, I started
on Augmentin yesterday it's.
We look,
we look, you look in shock.
More than one thing.
(33:02):
Can talk summer.
Asked about timing.
Say again?
Summer.
Just asked about timing.
I'm guessing about the nerve frequencies.
Summer asked about, I lost summer.
Oh, what about timing?
Yeah,
I erased it.
You run 40 and 3 96 until you can touchthe nerve without having the patient jump.
(33:22):
So you run 40 and 3 96 for maybe threeto five minutes while you're setting up
the second machine to run 13 and 3 96.
So if you're moving anerve that's scarred.
It's a pretty good chance you'regonna traction it, which will increase
(33:43):
the pain.
So if you have ones running 40 and3 96 constantly, then you can treat
the scar tissue easily and that wayif you tug on the nerve and create
a traction injury, it's not gonna
hurt anything.
So it's where multiple machines comein handy, even if they're custom cares.
(34:05):
You have one that's 40th through 96.
The second one we're singlefrequency combinations.
Yep.
I'm gonna jump down to Jennifer'squestion 'cause it's a little bit
easier to read than the other ones.
Just quickly, she has a client who hasataxic gait works as a pool cleaner.
(34:26):
Undergone MRI showed noabnormalities in his brain.
According to pt, x-rays are good.
She does an assessmentand there are no reflexes.
Sensory, everything is sharp touch, supersensitive on paraspinals and stomach.
C3 is in an anterior position.
(34:47):
He has shallow breathing when attemptingto stand and stabilize his bounce.
He seen the PT for that.
Began treatment withconcussion sympathetics.
Vagus nerve emotions,inner ear frequencies.
However, even with light touchon the neck, he has discomfort
raising his chin, reporting asensation of suffocation muscles.
(35:12):
Do not feel particularly tense.
Did not observe the expected smush effect.
Did he?
They did an MRI of the brain,and it's normal what it says.
23 years old.
It just so everybody knows, ataxia, whenI had that herniated disc in my neck
(35:36):
that was pushing on the motor pathways
Yeah.
And I had a positive binky on the leftand spasticity or increased tone on
the left one of the neurosurgeons said.
Oh,
some people wait until they'reataxic before they come in.
So ataxia does not alwayscome from the brain.
(35:58):
I don't know that much about it.
All I know is people that comein with an ataxic gait, it's
coming from the spinal cord.
But if the spinal cord is compressed.
He'll have hyperactive reflexes.
This guy needs to see aneurologist that isn't an idiot.
(36:22):
But really, it doesn't sound somebodydid an MRI of his brain, but.
How about an MRI of his neck?
MRI of his neck would be good, buta neurologist would also be good.
It's the, you did all the thing.
The only thing I would trywould be 40 and 10 or 81 and 10.
(36:47):
Raising his chin, suffocationmeans his, that's the modela.
Muscles don't feel tense.
That's weird.
Also expected, you said
you ran 40 and 10, but only for 15 minutes
and it didn't do anything.
(37:09):
Yeah, it's in a 40 and 10 patient.
The patella reflexes are
hyperactive.
And you're saying that youdidn't get any reflexes anywhere,
and there's two possibilities.
You're a massage therapist, soone is you might not be doing 'em.
That's a possibility.
Even for me, I.
(37:30):
And
having no reflexes, is it doesn'tgo with what I know about ataxia.
Wait, Jennifer said what?
Oh.
It's not on the QA.
Oh, she wrote in the chat.
She agrees, but so did pt.
(37:51):
Yeah, that's weird.
I don't know.
Yeah.
It's getting reflexes is not always easy.
Took us, three 10 weeks.
Even now C seven is onethat escapes me sometimes.
Okay.
So they only did a neck,MRI, an neck x-ray.
(38:16):
They did a brain MRI
neck, MRI might be a good idea.
C3 is anterior, whichmeans it's translated.
Is, and the only way to tellif there's cord compression is
an MRI of the cervical spine.
Raising his chin.
(38:37):
Sounds like dura, doesn't it?
Yeah, I was say I was, I have thatimage right of trying to keep right.
Yeah.
C3 is anterior.
And the other thing is that a C3disc is like if you do the little
(38:58):
pinwheel thing, C two, C3, C four.
What are these nerves like?
Sensory, everything is sar touch.
Super sensitive on theparaspinals and any stomach area.
Wait.
The thoracic paraspinals arehypersensitive and the, so the stomach
(39:22):
area is the thoracic paraspinals?
Okay, so
take two.
Go back and find out whathappened before all this started.
The thoracic paraspinals super sensitive.
So Hyperesthesia on the thoracicparaspinals and on the stomach
(39:43):
area, those are thoracic.
And you can actually get athoracic disc herniation.
So the question is, didhe fall on his butt?
He's a pool cleaner.
So that's what I'm thinking of.
Slipped on the pool deck.
Yeah.
Oh, he wore a helmet when he wasa kid because he always fell.
(40:05):
So has there been like a balanceissue since the donna time?
I'm just thinking vestibular.
It's the thoracic paraspinalsthat makes me nervous.
The only patient I've ever.
Treated that turned intoan overnight quadri.
Paraplegic was a guy with a Tseven eight herniated disc that
(40:26):
was pressing on a spinal cord.
And the surgeon didn't use enough steroidswhen he took the vertebral body or the
spinous process and the lamina off.
He didn't use enough steroids.
The disc and the cord blew upand sheared all the nerves.
So sounds to me.
(40:49):
But see, that should make hispatella reflexes hyperactive.
That should make thepatella is hyperactive.
Right.
Somebody needs thoracic.
MRI.
I think something ispressing on his spinal cord.
And if it, if the ataxia isnot coming from the brain, then
(41:12):
it's coming from someplace.
And you said that he fell allthe time when he was a kid.
So this is something he could havelived with for a really long time.
You can.
Try 81 and 10.
(41:33):
Alternating, alternating.
Don't polarize it if he's bad enoughto be ataxic and if it is coming from
a disc, it there, it's something youprobably ought to run alternating
from next to feed.
Don't polarize it.
(41:53):
If there's cord compression enough tocause ataxia, there's cord compression.
Enough to cause problemswith polarization too.
Suzanne, chronic lymph, 66-year-oldmale with chronic lymphocytic leukemia.
What.
Okay.
Treating the lymphocytic leukemia,they're waiting for it to get worse.
(42:17):
We just have to move past that.
August fell with vasovagal syncope,
er intubated heart attack,and cancer before Id.
Infectious disease infectiousdisease, confirmed West Nile virus.
Two weeks in the hospital,one week in rehab.
(42:38):
Walker couldn't lift his arms
through miles, just assume.
Okay.
Okay.
You.
You can treat the nerves.
Everybody else seems to beignoring the chronic lymphocytic
leukemia, so I guess you can too.
Don't treat the immune systemand west Nile virus, I would.
(43:04):
The only thing I think wehave that's close, it's not
in the herpes family is it?
No idea.
Epstein Barr family, I don't remember,but I go through all the viruses.
We have 38, 41, 44, 56, 1 60, 180 9,and do it with the con spinal cord.
(43:26):
The nerves.
Yes.
There you go.
Facebook suggestion one60 for viruses in 10.
3 96 1 0 9. Okay.
But put a pulse oxometer on him.
Initially first treat the right arm.
No treat the left.
That has more deficit.
(43:47):
I'd go with that.
It's gonna be the easier to fix probably.
Or you could start in the better one.
Ask him.
Basically ask him if, is heleft-handed or right-handed?
Which one does he want fixed?
Which one bothers him more?
'cause we do work for them.
Oh,
(44:08):
okay.
We're gonna leave this client number two.
Alone and she is so faroutside your scope, you really
chronic pain, mono, herpes.
Yeah, it's that one's, I'd find a, do a PT
(44:32):
or an MD to take this one chronichip, back, neck, elbow leg and
okay, that sounds like 40 and 10.
She's had six auto accidents,so that sounds 40 and 10.
And Vegas concussion in Vegas.
(44:53):
You could do that.
Easily uncontrollable seizures.
That sounds a little scary.
Yeah.
I It's
knowing when you're outside your scope.
Oh, yeah.
Alf if they did an MRI ofthe brain, the radiologist.
(45:17):
Would have checked for Chiari, 'causebrain MRIs stop at C one or two.
So Alpha's talking aboutthe guy with ataxia with the
with the normal brain, MRI and nobody'sand hyperesthesia in the thoracic
spine and nobody's done a thoracic.
(45:40):
MRI,
but they did do a brain MRI, which thena Chiari and tumor in the modela will
cause ataxia and the cerebellum as well.
If they did an MRI of the brain,they would've checked for a qi
and.
(46:01):
Catherine, that's whatquestion am I answering?
That's a really good question.
It's, we just I justwent all over the place.
Sorry about that.
Did I lose everybody?
You're
muted.
I was, my dog started barking, so Ijust put myself, I just it's easy for us
because we see the questions popping up.
(46:22):
So I think the question we were, oh,
nobody else sees them.
Oh, wow.
I don't know if everybody can seethem and or if they just check on it.
Yeah,
so summer is correct.
Chronic lymphic lymphocytic leukemiais cancer and we don't treat that.
So as long as you.
Don't treat the immune systemor the bone marrow or the blood.
(46:46):
You're fine.
People on YouTube can't evensee the questions though.
Say again?
The people on YouTube,
people on YouTube can't see the questions.
Oh, the people on
YouTube can't see the questions.
It's
our little immediategroup that gets to see it.
So that's why we have to like,read them and talk about them.
So sorry, YouTubers.
But yeah, I think one of thecommon things while we're on it is.
(47:08):
Is Can, is treating people withcancer completely contraindicated?
Say again?
You froze.
I did freeze.
I say that.
You did freeze.
Yes.
Oh, we're almost done.
Sorry.
No.
One of the questions that we get alot is people with cancer is treating
them completely contraindicated.
(47:31):
No, you can't treat the cancer, right?
You can treat concussion.
Yeah.
You don't treat the vagus 'cause thevagus suppresses the immune system.
So you don't want to treat the vagus,but you can, according to Harry.
Treat concussion.
Support the adrenals, treat the emotionalfactors because they're scared to
death if they're refusing medical care.
(47:57):
You can treat bone mets and youcan treat the pain from the cancer.
If they're having traditionalcancer therapy, you can treat
toxicity from chemotherapy.
You can treat radiationburns during the radiation.
(48:20):
To prevent scarring.
You can treat preemptively, you can treatperipheral neuropathy from chemotherapy.
So
the cisplatin of chemo agents universally,I can't remember what the percentage
(48:41):
is, but it's it's, pretty high.
So we had one practitioner whotreated his blood vessels and
from knees, feet with chemotherapyperipheral neuropathy from toxicity.
And these days they are alsoduring chemotherapy using ice.
(49:07):
On the lower legs to reduce thecirculation in the lower legs and
reduce the amount of chemo agent thatgets to the blood vessels in the lower
legs so the nerves don't get damaged.
Carcinoma toxins, there is a numberfor that, but we don't use it to treat
cancer 'cause we don't treat cancer.
(49:27):
So I'm not even sure whatcarcinoma toxins are.
Leaf, I'm gonna see you next week,I think, or the week after, so I can
ask you what Harry thought about that.
But I, once a patient has beendiagnosed with cancer, or once
I suspect cancer I'm done.
It's, I'm not done.
I do concussion.
(49:49):
I do the adrenals, I doemotions, I do immune support.
I think those are the four thingsthat Harry did adjust the spine.
That's the other thing he did.
Yeah.
We have a minute left.
I'm going to, I'm going to bring thismeeting to a close and say if you have not
(50:10):
yet registered, if I'm frozen, I'm sorry.
For the advanced, the sportscourses are almost full.
I think there's two spots left andbecause we're in the one room we
can't pack very many people in.
Please sign up if you haven't yet,and please come because it's in person
and I can't wait to see everybody.
It's gonna be so much fun.
(50:31):
We have 28 people already for the core.
Wow.
We have 28 people for thecore, so we have room for.
How many more?
Two more.
10 more.
I'll take.
I'll take up to 40.
Kevin says he'll take up to 40.
I love the, whoever it was thatsaid, after six auto accidents,
(50:55):
perhaps slipping, TTH, and one ofthose sessions should be considered.
And.
Yeah, I'm excited for Phoenix too.
I'm working on my slides.
Yay.
Yay.
Alright everybody,
we made it.
It's four o'clock.
Sorry about my glitchy connection today.
(51:17):
Internet is internet.
Yes.
I'll see you next.
Bye.
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as well as more information aboutfrequency specific microcurrent.
Last week, I ran all over yourlist, so I figured this week that I
would let you and your list start.
(00:24):
Okay.
I almost wish I, I do think weactually have to go back to last
week for a little bit because itprompted, I don't know about you,
but I got a bunch of emails sent.
There's one, oh.
Before we go any further.
Okay.
I have to clarify somethingthat you said on January 22nd.
Somebody wrote something in, soI didn't know what it meant and
(00:48):
I didn't go back and listen toit, so I'll try to paraphrase it.
Okay.
It was regarding postherpetic neuralgia.
So the quote said something like,polarize the heck out of it.
The curve makes the voltage gated channelsin the nerve membrane flip over, forcing
them and then dot just knocks into them.
(01:08):
They would like a little more explanation.
Oh, first you have to understandthat the first you have to Google
voltage gated ion channels in nerves.
I can't describe that there.
Nerves, nerve membranes, fire.
You get a, action potentialbecause there's sodium on one
(01:35):
side and potassium on the other.
And I'd love to pretend thatI remember which ones, which I
think sodium's on the outside.
And anyway, so when the actionpotential comes along, the way it is
propagated is this channel opens andsodium runs in, and potassium runs
out, and then it flips over again.
(01:58):
And it does that.
That's how the actionpotential goes along.
So if you've ever had anerve conduction study.
There's this little spike that goes well,that is a sodium potassium voltage gated.
So voltage gated means that there's a,
(02:20):
that channel that lets the sodiumin and the potassium out and
lets the actual potential go is.
Operated by or influenced by voltage.
All I know is that Terry Phillipscalls 'em voltage gated ion channels
and he knows a lot more than I do.
So I, that's what I call him.
(02:42):
And then, so I think, this is myhypothesis, that one of the ways that FSM.
Treats nerve pain so easily is,there's if you run 40 and 3 96,
(03:04):
if a nerve is inflamed, it stopsconducting the inflammation and
the nerve stops conductivity.
That is pretty good forrelieving nerve pain and it will.
Also improve
(03:25):
nerve atrophy in a muscle.
But to get a nerve from numb
to hypersensitive to normal, firstyou have to relieve the inflammation.
'cause that's what made itnumb in the first place.
(03:45):
But then.
That will help the muscles likeshow up, but to get the muscles
to contract, whoa, that's weird.
That's good.
Get the muscles to contract and to getthe nerve from hypersensitive to normal.
(04:06):
Sometimes you have to run increasedsecretions in the nerve and
the nerve has to be polarized.
It's nerves are polarized.
We're positive, centrallynegative distally.
That means there's more electronsout here than there is here, right?
'cause electrons are negative.
And I think of polarized positiveincreased secretions, polarize
(04:33):
positive, reduce inflammation as.
Forcing the nerve membrane to work.
And it's just, when I say drive it,I'm, that's exactly what I mean.
It's yeah, you don't get to vote.
You are going to work.
(04:54):
Yeah.
This is how this goes.
And as long as the nerve isstill there, it usually works.
Yeah.
If somebody's cut the nerve.
Or if the virus has completelydestroyed the nerve.
So if you have postherpetic neuralgia andthe tissue is numb and painful, there are
(05:17):
times when the nerve has been destroyedby the virus, like it's just not there.
That's when you have basicallyphantom limb pain in your rib, right?
You're on 40 and 89 and hope for the best.
Post-traumatic neurology is not easy.
(05:38):
And it's like the more youknow about neurology, the
more you know you don't know.
Yeah, it's I, one of the ways I wentdown the rabbit hole and screwed up the
advanced list today was trying to addthe frequencies for the cranial nerves.
(05:59):
Because I finally, after 30 years,had a patient that had cavernous
hemangioma that burst, and cavernoushemangioma is like a tangle of blood
vessels that are pretty fragile.
First, she had a bleed that wasa hemorrhagic stroke, which had
(06:19):
two little hemangiomas, one in hercerebellum and one in between the pons.
And the fourth ventricle, and thenthe surgeon said, so six months
later, she recovered from the stroke.
Then the surgeon said, those,he angios are still there.
They could go any time, sowe should take them out.
(06:42):
Spend a year and a half since he tookthem out and she has deficits in.
Cranial nerves 3, 4, 5, 6, 7, 8, andnine that have just not recovered.
And that's, I found out thatGeorgia's experimental frequency
(07:03):
for the facial nerve works.
Yay.
The accessory nerve kind of works someof the frequencies on his list for.
Two of the cranial nerves are ab pairs,and I'm not sure what to do with those.
So it's I'm gonna put it in, but it, thequestion is, how often do people see these
(07:30):
kind of really esoteric neurologic things?
Nerve pain, everybody treats,at least I hope they do.
Yeah.
So
that's.
That will, that phrase will alwaysbring me back to my first core.
When you say nerve pain is gonna beone of the easiest things that you
treat, and I'm always a nerd and I'malways sitting either in the front row
(07:52):
or I'm sitting in the very back row,but this course I was sitting in the
very front row and I remember lookingaround being like, did anybody else
just hear her say, nerve pain is theeasiest thing that we're going to treat.
It's, it is, it's the easiest thing we
treat.
Can you, while we're on the nerve,and I'm gonna derail my own list right
now, but since we're talking aboutit, I thought I would just jump on it.
(08:15):
When you have somebody, can youexplain to, maybe there isn't an
explanation how numbness goes to hyperaesthetic and then it goes to normal.
Oh it's like the reverse process,when a nerve first becomes inflamed,
it becomes hyper aesthetic, right?
(08:36):
Hypersensitive.
So with a pinwheel, you havesharp or prickly, you have
icky, and then you have numb.
That's the progressionof nerve dysfunction.
If a nerve is numb, if you'relucky, the nerve is still there.
(08:58):
It's just so messed up thatit can't conduct anything.
Right.
Sensory is easier for us than motor.
Yeah.
But we can in, becausemost nerves are mixed.
Sensory motor, the ones that we dealwith in the arms and legs and back.
(09:19):
So when you go from normalto hypersensitive the next.
Stage of dysfunction is numb whena nerve is, when an area is numb.
So I was treating a, an osteopath ata medical meeting who had open heart
(09:41):
surgery and he had a brachial plexustraction injury and his left arm where
his arm fell off the board as theywere doing the open heart surgery.
And he said his hand was so numb.
That he would blister his fingerstaking a cup out of the microwave
'cause he couldn't feel it.
So I went, that's not thathard, is it okay if I treat you?
(10:08):
And he went, okay.
And he is looking around the booth andI just wrapped towel around his neck
and wrapped a towel around his arm.
And we talked about his kids and hisgrandkids and his open heart surgery and
my open heart surgery and blah, blah blah.
And after about 30 minutes.
His hand and his c.
(10:31):
Where I used the pinwheelon him and he went, ow.
I said, oh, that's great.
He said, what are you talking about?
I said it was numb aminute, 30 minutes ago.
The next step back towardsnormal is hypersensitive.
What this mean is the nerve meansis the nerve is still there.
It's not dead, it's not broken.
(10:52):
It just has to be repolarized.
It.
If it goes from numb to hypersensitive,it will eventually go back to normal.
May or may not stay there.
Traction injuries are the easiest becausethere's no perpetuating factor, right?
It just got stretched.
And the stretching, if you thinkof it, you take those voltage
(11:14):
gated ion channels and you justrip 'em apart so they don't work.
That's a nerve traction entry.
That's how I think of them anyway.
And then nerve traction Injury.
Do you focus on 1 24 first?
Nope.
Just 40.
Just 40.
And then 81.
I guess I could do 1 24.
I just never thought of it.
'cause I never needed it.
(11:35):
So it might help and I couldjust be, a creature of habit.
And maybe all it needs is current.
Yeah.
But I, it's, it has always been40 and 81 with nerves for me.
So yeah.
Yeah, those are the very fundamental,basic factors I think when
(12:00):
we're dealing with nerve pain.
Okay.
So that's that part.
While we're on pain though, we dida lot of visceral talk yesterday.
Or last week and yesterday.
What's that?
I said it seems like yesterday.
It, it does.
And I was thinking because I'mgetting treatment on my hip right now,
(12:22):
and people are against me bringingmy machine to my own treatment.
So I'm treating myself first at homeor in the car before I get Yeah.
Treatment done.
They won't let you bring your own machine.
Okay.
Okay.
That's, nevermind.
(12:42):
It's okay.
So when people are trying toget into treat your SOAs, you
know where I'm going with this.
How do you treat theSOAs without 13 and 60?
It's just insane.
Why?
Why would you let them do that to you?
What am wait.
(13:03):
Go The time.
This is my, I'm changing hats now.
Okay.
To my patient advocate hat?
Yes.
My patient advocate hatis, they work for you.
Correct.
And so the conversation is, I wantyou, we both agree that my SOAs needs
working on, I'm going to run a frequencywhile you're working on my SOAs.
(13:27):
That should make workingon my SOAs easier.
They don't need to knowthat it's 13 and 60.
And if they say no, youfind a different therapist,
which is why I've been on the run.
Okay.
Literally, sorry.
It just,
no.
No, I, and I give peoplethe benefit of the doubt.
The problem is not really the problem.
(13:48):
It's the eyeopening experience ofknowing a, how deep the SOAs is.
Let's just think about this fora second, and iliac is, okay.
So SOAs attaches onto the lumbar vertebra
from the
front.
On the front.
And people are taking an anteriorapproach to go through everything that
(14:12):
is on top, and nobody is taking theirtime and they're just using force.
And all I can see is the microtraumathat is happening on top of everything.
And I, like I said I've been on the run
the thing that was fun with this guythat we got on videotape last week,
(14:36):
that he's got just quite a tummy on him.
But even with that, if you roll off ofthe rectus abdominis and the omentum
So you start in the middle andI'm on his right hand side.
Okay.
And.
You come to a ditch, you come toa soft spot, and that if you hit
(15:02):
it in the right place, you'reon the lateral edge of the SOAs.
Because it goes, thething is this wide and.
So you go on the lateral edge ofthe SOAs, you are on 13 and 60,
and underneath all the visceralorgans, you don't go through them.
(15:24):
You go lateral to medial and you hit theSOAs and you could, or the ureter, and
you can tell because the muscle splintsup and the patient makes scrunchie faces,
right,
and you just back off andwait for it to soften.
Then you just roll it medial and whenit gets stiff again and they make s
(15:45):
crunchy faces, you stop and you wait,and then it softens about a millimeter,
and then you roll it another three,four millimeters, and you wait.
It's, so there's no you, the,
yeah.
There's, in a perfect world, that wouldbe the approach that everybody would take
(16:08):
to treat the SOAs or the iass, however.
I'm here to say that is not theapproach that some therapists take.
And why would
you let them do that to you?
I don't let them.
I have left.
I I have said I don't think thisis the right approach for me.
Can we move on to something else andI'll get somebody else to work on my
(16:28):
So as I promise and that someone is me.
I'll see you in March.
Yes.
Yes, that's that, that'll work.
Yeah.
But it got me thinking because Idon't know about you, but when I get
worked on, I start over analyzingwhat are they on, what are you doing?
(16:50):
Why are you doing what you're doing?
And I'm thinking, why didthe tissue get like this?
Because I am a good FSM clinician,so when I'm treating others, I'm
thinking, how did we get here?
So when it comes to scarring,like scarring is one of my
obviously favorite frequencies.
(17:11):
We talked about this,I think, podcast one.
If you were on a deserted islandand you could bring five, eight
channels with you, I think 13would easily make that list for me.
Am I freezing or are you freezing?
I don't know.
I'm not freezing.
You're freezing on andoff, but not for very long.
Okay.
(17:33):
Maybe my seats.
It's electronic.
It's electricity today.
But now that I'm thinking more andmore, how did the scarring get here?
Why did the scarring get here?
13 isn't the holy grail that it once was.
It's necessary and it's useful becauseit will help with the adhesions and
(17:56):
it will help with the scar tissue,but I have to think more now.
How did we get here?
Was something inflamed first?
Did something, tear first was somethingtraumatized first was something.
I've got one for.
Patient's presenting complaintis swelling and pain.
(18:19):
She's a runner.
Presenting a complaint isswelling and pain in her knee.
And the MRI shows the MRI reportsays an intact meniscus, but she
has pain on the medial side ofher knee, so I don't believe it.
And conia patella.
(18:39):
When you look at her knee, it's acentimeter larger than her right
knee, and the patella is pulledover to the lateral side, so the VAs
later lysis too tight pulling it out.
The quadriceps is too tight.
(19:02):
She's got hyperesthesia in allof the cervical dermatomes and.
Is very tick Oliver.
It's this is interesting.
So I'm working on her leg andfind out that her hamstrings
are like guitar strings.
They're really tight.
So the first visit I ran40 and 10 and 91 and 10.
(19:25):
Why are you doing that?
Because your hamstrings and yourquads shouldn't be that tight
and the knee pain didn't start.
Until after she had a tummy tuckand liposuction, and the nice man
did a liposuction on her back,and L one was numb from where
(19:49):
the incision was in her abdomen.
But L two and three and fourwere all way hypersensitive.
She said, I want you to fix my knee.
And I went unfortunatelyyour knee is not the problem.
It's the symptom, but before thesurgery you didn't have any knee pain.
(20:15):
And after the surgery, your patellais tracking to the left, and I'm
thinking to myself, you really needthe Livermore to go to Livermore and
see Kim, but she's got two kids and shethree kids and she doesn't have time.
So I had to channel you and I thought,wait a minute, what did he do?
(20:40):
He.
Did a liposuction around the lumbar spine.
And L two was, I think thetechnical medical term you use
is pissed off, was irritable.
That would make the vastlateralis cranky as well.
(21:01):
So the day we made the most progress.
It was the next day when I ran 124 and the meniscus for an hour.
I ran so torn and broken repair and healedthe meniscus 'cause she has joint, she has
(21:24):
pain at the joint line on the medial side.
The only thing that does thatis the meniscus and then the.
Patella is just gettingground down in the groove.
So I did inflammation and I thinknecrosis in the patella, but
inflammation in the patella for an hour.
(21:46):
Inflammation in the periosteumfor an hour, and in inflammation
in the nerve for an hour.
So she had four machineson her left side and on.
I went over on her right side.
And started taking apart the scar tissuefrom the tummy tuck and liposuction.
(22:08):
Right.
And at the end of it, for thefirst time, I think it was the
third session, she got up and she'swalking around and she's so excited.
She said, it doesn't hurt.
It's still half a centimeter bigger.
They're still diffuseswelling, but it didn't hurt.
(22:28):
They did an MRI of her knee.
Nobody's, nobody, none of the physicaltherapists, none of the surgeons,
none of her gps, nobody that's seenher has te tested her sensation.
Nobody has done her reflexes.
(22:48):
So that's, that is why, that's the otherreason FSM practitioners are special.
It's like you can learn anawful lot with a pinwheel.
Yeah it's been a huge adjunctto my assessment findings.
I'm biomechanic geek.
I'm also adding fascial linesand that kind of movement
(23:09):
pattern in, and, everythingcompliments the other thing, right?
The thing with FSM is itforces you, allows you.
To think outside the box andcross reference, and you said it
before, it's pattern recognition.
So once you see something, onceit's impossible to unsee it, and
(23:31):
then you start seeing it everywhere.
So I do think it it always hasbeen beneficial when somebody
comes in with pain in their kneeto not be so myopic and just look
at the knee to do your assessment.
And think, could it becoming from the hip?
Could it be coming from the back?
Could it be coming from?
(23:52):
And the other question you ask is,what happened just before this?
I had a tummy tuck in Novemberand I got knee pain in January.
Those that, that, those are not unrelated.
It's not, what is it?
(24:13):
Be before, therefore, becausenot necessarily, it's not
always this happened before.
Therefore, what happened afterward?
It has been caused bywhat happened before.
But in medicine at least,they're usually related,
right?
Yeah.
Side note, I've been binge watchingthis old show on Netflix called The
(24:36):
Resident, and I love medical dramas.
It's called the what?
The Resident.
Oh, good.
Yeah.
Okay.
I've never seen that.
He's an internal medicine doctor and heputs together he does pattern recognition.
I just keep thinking, he wouldbe so good at FSM and I realize
he's not a real doctor, but Icredit that type of thinking.
(24:59):
I value that type of thinking somuch more because, and you've said
it time and time again, you don'tthink about these areas because before
you never had a tool to treat it.
So who cared about the Durham really?
Yeah.
Who cared about the periosteum, whocared about the fascia, who cared
about the mentum, we knew it was there.
(25:20):
But the respect that you have ismore profound, I guess with FSM
you have a way of affecting,you have a way of affecting it.
So I just I love my dearfriends who are fashionistas.
They think it's the fascia.
They think it's the muscle.
(25:41):
But, and this is why I say, with allhonesty, I'm a terrible chiropractor.
'cause I don't think bones do anything.
They either move or they don't move.
And if they don't move, there'sa reason they don't move.
Muscles move bones.
Nerves move muscles.
The brain moves the nerves, right?
(26:01):
And it's like some.
You just have to find outwhere along that chain Yes.
Things went wrong.
Yeah.
And I don't think that is far off.
I think a lot of therapists do, whenwe do our assessment we're trying to
figure out why isn't this working?
Why is this not firingthe way that it should?
(26:22):
But now you just have so muchmore support in your treatment
plan after figuring out the cause.
Yeah.
We have, I've seen biomechanical feedback.
People do some amazing things here,push on this and then do that, and
then do this, and then do that, andwhatever the problem was goes away.
(26:45):
The question is how long will that stay?
Did you fix it?
Strain, counter strength.
Great.
Terrific.
Yeah.
Will it stay fixed?
I don't know.
It's, yeah,
The staying fixed ismy, it's my love affair.
That's where I I takethat part very seriously.
(27:07):
I guess once you start getting goodat what you can do with your hands in
the clinic the real challenge is howcan we keep these results as permanent
or as long lasting as possible?
And I do believe with everything in myheart that it comes down to feeling safe.
Body has to feel safe tocreate the movement and the
contraction, otherwise nothing.
(27:29):
So the real work, I think, beginswith so I think, yes, what's
happening, how did we get there?
And then after that is.
How can I create a safe environment?
How can I get this nervous systemto believe these changes are safe
and the movement is gonna be safe?
And this lady with the knee said, I'mjust, I've just really ticklish while
(27:58):
ticklish is, goes up the pain pathways.
So when I did the sensory exam, she washyper aesthetic from C two to S two.
Like she had two nerves thatwere not hypersensitive.
Wow.
So I ran 40 and 10 and 40 and 92, andthat wasn't till the second day that
(28:25):
I needed to run 81 and 10 toloosen the hamstrings, but.
Safe is ticklish is not safe.
So she said, how are yougoing to make me not ticklish?
And I said we just talk to thesensory cortex and tell it's safe.
(28:47):
We just dial it down a little bit.
And so you run 40 and 92, quiet, thesensor motor cortex, and I've never.
Not work.
It's like I couldn't touch her abdomento work on the liposuction scar
(29:08):
'cause I couldn't touch her abdomen.
She's so tickly.
You can't treat somebody that'slevitating, so I ran 40 and 92 for,
I don't know, it took 20 minutes,which is a really long time.
Usually it's two or three.
That's what made me have 40 and 10.
It's interesting.
Yeah.
So it's like the body had to befeel safe and there's something
(29:33):
about nerve hyper irritability orhyper sensory awareness, whatever
tickler is about, and that is unsafe.
Yeah.
And there used to be a trick thatwe would do, especially again, it's
like the SOAs going into the abdomen.
(29:55):
If they were ticklish goinginto, rec femme area, iliac is
to put their hand down and thenyou put your hand on top of it
that works.
Yeah.
But.
It's just nicer if you don't haveto involve more things on top
of where you're trying to go.
Sometimes.
There's a lot of questions.
(30:15):
One are like a couple case studies.
So I just, I wanna jump to summersreally quick because we're on the
nerve sort of train right now.
She says on nerve inflammation,you said 43 96 polarized.
And are you also seeing81 3 96 polarized too?
Just want to be sure I understand that.
I have a client withserious leg nerve pain major
(30:36):
fasciculations from back surgery.
So I imagine scar tissue involved too.
So 13 3 96 2,
Run 40 and 3 96 first so that youcan touch the nerve and then take the
scarring out, and then you can try.
(30:57):
Fasciculations is.
When the nerve is, whenthe muscle is denervated.
So then you do 81 and 3 96,or even sometimes 81 and 10 to
get rid of the fasciculations.
That's a, that shouldn't be athing after low back surgery.
It's, that's curious.
(31:18):
Yep, you're on the right track.
Keep in mind, you mighthave to run 81 and 10.
I know it sounds like I know whatI'm doing, but you guys understand
I make this up as I go along, right?
When it comes to nerve pain,we just talked about two,
two a channels, 40 and 13.
How do you discern which one to use?
(31:40):
Oh, you always use for 41st becauseany nerve that's scarred is inflamed.
Yeah.
So one of the.
Techniques and research for creating nervepain or simulating nerve pain is they go
in and they just tie a knot around it.
They put a piece of thread around.
(32:00):
It basically creates scar tissueon the nerve, so you have to
run 40 so that you can touch
nerve.
And then on the secondit was 13 and or 81.
But, and then you follow the nervealong its path and release it because
(32:21):
it can be scarred just about any place.
What'd you do
with your hand?
Oh I burn myself.
You have a bandaid on.
I burnt myself.
Oh, okay.
As long as, yeah, I got actuallygot a cold, a flu, a sinus
infection, all those things.
(32:41):
It's the bug is that's going around.
Just in case it's viral.
I've been running long covid every night.
Yeah.
And
then just in case it's bacterialon top of viral, I started
on Augmentin yesterday it's.
We look,
we look, you look in shock.
More than one thing.
(33:02):
Can talk summer.
Asked about timing.
Say again?
Summer.
Just asked about timing.
I'm guessing about the nerve frequencies.
Summer asked about, I lost summer.
Oh, what about timing?
Yeah,
I erased it.
You run 40 and 3 96 until you can touchthe nerve without having the patient jump.
(33:22):
So you run 40 and 3 96 for maybe threeto five minutes while you're setting up
the second machine to run 13 and 3 96.
So if you're moving anerve that's scarred.
It's a pretty good chance you'regonna traction it, which will increase
(33:43):
the pain.
So if you have ones running 40 and3 96 constantly, then you can treat
the scar tissue easily and that wayif you tug on the nerve and create
a traction injury, it's not gonna
hurt anything.
So it's where multiple machines comein handy, even if they're custom cares.
(34:05):
You have one that's 40th through 96.
The second one we're singlefrequency combinations.
Yep.
I'm gonna jump down to Jennifer'squestion 'cause it's a little bit
easier to read than the other ones.
Just quickly, she has a client who hasataxic gait works as a pool cleaner.
(34:26):
Undergone MRI showed noabnormalities in his brain.
According to pt, x-rays are good.
She does an assessmentand there are no reflexes.
Sensory, everything is sharp touch, supersensitive on paraspinals and stomach.
C3 is in an anterior position.
(34:47):
He has shallow breathing when attemptingto stand and stabilize his bounce.
He seen the PT for that.
Began treatment withconcussion sympathetics.
Vagus nerve emotions,inner ear frequencies.
However, even with light touchon the neck, he has discomfort
raising his chin, reporting asensation of suffocation muscles.
(35:12):
Do not feel particularly tense.
Did not observe the expected smush effect.
Did he?
They did an MRI of the brain,and it's normal what it says.
23 years old.
It just so everybody knows, ataxia, whenI had that herniated disc in my neck
(35:36):
that was pushing on the motor pathways
Yeah.
And I had a positive binky on the leftand spasticity or increased tone on
the left one of the neurosurgeons said.
Oh,
some people wait until they'reataxic before they come in.
So ataxia does not alwayscome from the brain.
(35:58):
I don't know that much about it.
All I know is people that comein with an ataxic gait, it's
coming from the spinal cord.
But if the spinal cord is compressed.
He'll have hyperactive reflexes.
This guy needs to see aneurologist that isn't an idiot.
(36:22):
But really, it doesn't sound somebodydid an MRI of his brain, but.
How about an MRI of his neck?
MRI of his neck would be good, buta neurologist would also be good.
It's the, you did all the thing.
The only thing I would trywould be 40 and 10 or 81 and 10.
(36:47):
Raising his chin, suffocationmeans his, that's the modela.
Muscles don't feel tense.
That's weird.
Also expected, you said
you ran 40 and 10, but only for 15 minutes
and it didn't do anything.
(37:09):
Yeah, it's in a 40 and 10 patient.
The patella reflexes are
hyperactive.
And you're saying that youdidn't get any reflexes anywhere,
and there's two possibilities.
You're a massage therapist, soone is you might not be doing 'em.
That's a possibility.
Even for me, I.
(37:30):
And
having no reflexes, is it doesn'tgo with what I know about ataxia.
Wait, Jennifer said what?
Oh.
It's not on the QA.
Oh, she wrote in the chat.
She agrees, but so did pt.
(37:51):
Yeah, that's weird.
I don't know.
Yeah.
It's getting reflexes is not always easy.
Took us, three 10 weeks.
Even now C seven is onethat escapes me sometimes.
Okay.
So they only did a neck,MRI, an neck x-ray.
(38:16):
They did a brain MRI
neck, MRI might be a good idea.
C3 is anterior, whichmeans it's translated.
Is, and the only way to tellif there's cord compression is
an MRI of the cervical spine.
Raising his chin.
(38:37):
Sounds like dura, doesn't it?
Yeah, I was say I was, I have thatimage right of trying to keep right.
Yeah.
C3 is anterior.
And the other thing is that a C3disc is like if you do the little
(38:58):
pinwheel thing, C two, C3, C four.
What are these nerves like?
Sensory, everything is sar touch.
Super sensitive on theparaspinals and any stomach area.
Wait.
The thoracic paraspinals arehypersensitive and the, so the stomach
(39:22):
area is the thoracic paraspinals?
Okay, so
take two.
Go back and find out whathappened before all this started.
The thoracic paraspinals super sensitive.
So Hyperesthesia on the thoracicparaspinals and on the stomach
(39:43):
area, those are thoracic.
And you can actually get athoracic disc herniation.
So the question is, didhe fall on his butt?
He's a pool cleaner.
So that's what I'm thinking of.
Slipped on the pool deck.
Yeah.
Oh, he wore a helmet when he wasa kid because he always fell.
(40:05):
So has there been like a balanceissue since the donna time?
I'm just thinking vestibular.
It's the thoracic paraspinalsthat makes me nervous.
The only patient I've ever.
Treated that turned intoan overnight quadri.
Paraplegic was a guy with a Tseven eight herniated disc that
(40:26):
was pressing on a spinal cord.
And the surgeon didn't use enough steroidswhen he took the vertebral body or the
spinous process and the lamina off.
He didn't use enough steroids.
The disc and the cord blew upand sheared all the nerves.
So sounds to me.
(40:49):
But see, that should make hispatella reflexes hyperactive.
That should make thepatella is hyperactive.
Right.
Somebody needs thoracic.
MRI.
I think something ispressing on his spinal cord.
And if it, if the ataxia isnot coming from the brain, then
(41:12):
it's coming from someplace.
And you said that he fell allthe time when he was a kid.
So this is something he could havelived with for a really long time.
You can.
Try 81 and 10.
(41:33):
Alternating, alternating.
Don't polarize it if he's bad enoughto be ataxic and if it is coming from
a disc, it there, it's something youprobably ought to run alternating
from next to feed.
Don't polarize it.
(41:53):
If there's cord compression enough tocause ataxia, there's cord compression.
Enough to cause problemswith polarization too.
Suzanne, chronic lymph, 66-year-oldmale with chronic lymphocytic leukemia.
What.
Okay.
Treating the lymphocytic leukemia,they're waiting for it to get worse.
(42:17):
We just have to move past that.
August fell with vasovagal syncope,
er intubated heart attack,and cancer before Id.
Infectious disease infectiousdisease, confirmed West Nile virus.
Two weeks in the hospital,one week in rehab.
(42:38):
Walker couldn't lift his arms
through miles, just assume.
Okay.
Okay.
You.
You can treat the nerves.
Everybody else seems to beignoring the chronic lymphocytic
leukemia, so I guess you can too.
Don't treat the immune systemand west Nile virus, I would.
(43:04):
The only thing I think wehave that's close, it's not
in the herpes family is it?
No idea.
Epstein Barr family, I don't remember,but I go through all the viruses.
We have 38, 41, 44, 56, 1 60, 180 9,and do it with the con spinal cord.
(43:26):
The nerves.
Yes.
There you go.
Facebook suggestion one60 for viruses in 10.
3 96 1 0 9. Okay.
But put a pulse oxometer on him.
Initially first treat the right arm.
No treat the left.
That has more deficit.
(43:47):
I'd go with that.
It's gonna be the easier to fix probably.
Or you could start in the better one.
Ask him.
Basically ask him if, is heleft-handed or right-handed?
Which one does he want fixed?
Which one bothers him more?
'cause we do work for them.
Oh,
(44:08):
okay.
We're gonna leave this client number two.
Alone and she is so faroutside your scope, you really
chronic pain, mono, herpes.
Yeah, it's that one's, I'd find a, do a PT
(44:32):
or an MD to take this one chronichip, back, neck, elbow leg and
okay, that sounds like 40 and 10.
She's had six auto accidents,so that sounds 40 and 10.
And Vegas concussion in Vegas.
(44:53):
You could do that.
Easily uncontrollable seizures.
That sounds a little scary.
Yeah.
I It's
knowing when you're outside your scope.
Oh, yeah.
Alf if they did an MRI ofthe brain, the radiologist.
(45:17):
Would have checked for Chiari, 'causebrain MRIs stop at C one or two.
So Alpha's talking aboutthe guy with ataxia with the
with the normal brain, MRI and nobody'sand hyperesthesia in the thoracic
spine and nobody's done a thoracic.
(45:40):
MRI,
but they did do a brain MRI, which thena Chiari and tumor in the modela will
cause ataxia and the cerebellum as well.
If they did an MRI of the brain,they would've checked for a qi
and.
(46:01):
Catherine, that's whatquestion am I answering?
That's a really good question.
It's, we just I justwent all over the place.
Sorry about that.
Did I lose everybody?
You're
muted.
I was, my dog started barking, so Ijust put myself, I just it's easy for us
because we see the questions popping up.
(46:22):
So I think the question we were, oh,
nobody else sees them.
Oh, wow.
I don't know if everybody can seethem and or if they just check on it.
Yeah,
so summer is correct.
Chronic lymphic lymphocytic leukemiais cancer and we don't treat that.
So as long as you.
Don't treat the immune systemor the bone marrow or the blood.
(46:46):
You're fine.
People on YouTube can't evensee the questions though.
Say again?
The people on YouTube,
people on YouTube can't see the questions.
Oh, the people on
YouTube can't see the questions.
It's
our little immediategroup that gets to see it.
So that's why we have to like,read them and talk about them.
So sorry, YouTubers.
But yeah, I think one of thecommon things while we're on it is.
(47:08):
Is Can, is treating people withcancer completely contraindicated?
Say again?
You froze.
I did freeze.
I say that.
You did freeze.
Yes.
Oh, we're almost done.
Sorry.
No.
One of the questions that we get alot is people with cancer is treating
them completely contraindicated.
(47:31):
No, you can't treat the cancer, right?
You can treat concussion.
Yeah.
You don't treat the vagus 'cause thevagus suppresses the immune system.
So you don't want to treat the vagus,but you can, according to Harry.
Treat concussion.
Support the adrenals, treat the emotionalfactors because they're scared to
death if they're refusing medical care.
(47:57):
You can treat bone mets and youcan treat the pain from the cancer.
If they're having traditionalcancer therapy, you can treat
toxicity from chemotherapy.
You can treat radiationburns during the radiation.
(48:20):
To prevent scarring.
You can treat preemptively, you can treatperipheral neuropathy from chemotherapy.
So
the cisplatin of chemo agents universally,I can't remember what the percentage
(48:41):
is, but it's it's, pretty high.
So we had one practitioner whotreated his blood vessels and
from knees, feet with chemotherapyperipheral neuropathy from toxicity.
And these days they are alsoduring chemotherapy using ice.
(49:07):
On the lower legs to reduce thecirculation in the lower legs and
reduce the amount of chemo agent thatgets to the blood vessels in the lower
legs so the nerves don't get damaged.
Carcinoma toxins, there is a numberfor that, but we don't use it to treat
cancer 'cause we don't treat cancer.
(49:27):
So I'm not even sure whatcarcinoma toxins are.
Leaf, I'm gonna see you next week,I think, or the week after, so I can
ask you what Harry thought about that.
But I, once a patient has beendiagnosed with cancer, or once
I suspect cancer I'm done.
It's, I'm not done.
I do concussion.
(49:49):
I do the adrenals, I doemotions, I do immune support.
I think those are the four thingsthat Harry did adjust the spine.
That's the other thing he did.
Yeah.
We have a minute left.
I'm going to, I'm going to bring thismeeting to a close and say if you have not
(50:10):
yet registered, if I'm frozen, I'm sorry.
For the advanced, the sportscourses are almost full.
I think there's two spots left andbecause we're in the one room we
can't pack very many people in.
Please sign up if you haven't yet,and please come because it's in person
and I can't wait to see everybody.
It's gonna be so much fun.
(50:31):
We have 28 people already for the core.
Wow.
We have 28 people for thecore, so we have room for.
How many more?
Two more.
10 more.
I'll take.
I'll take up to 40.
Kevin says he'll take up to 40.
I love the, whoever it was thatsaid, after six auto accidents,
(50:55):
perhaps slipping, TTH, and one ofthose sessions should be considered.
And.
Yeah, I'm excited for Phoenix too.
I'm working on my slides.
Yay.
Yay.
Alright everybody,
we made it.
It's four o'clock.
Sorry about my glitchy connection today.
(51:17):
Internet is internet.
Yes.
I'll see you next.
Bye.
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The information and opinion providedin the podcast are not medical advice.
Do not create any type of PO doctorpatient relationship, and unless expressly
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Crime Junkie
Does hearing about a true crime case always leave you scouring the internet for the truth behind the story? Dive into your next mystery with Crime Junkie. Every Monday, join your host Ashley Flowers as she unravels all the details of infamous and underreported true crime cases with her best friend Brit Prawat. From cold cases to missing persons and heroes in our community who seek justice, Crime Junkie is your destination for theories and stories you won’t hear anywhere else. Whether you're a seasoned true crime enthusiast or new to the genre, you'll find yourself on the edge of your seat awaiting a new episode every Monday. If you can never get enough true crime... Congratulations, you’ve found your people. Follow to join a community of Crime Junkies! Crime Junkie is presented by audiochuck Media Company.