Episode Transcript
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Speaker 1 (00:00):
In this episode of
the Regenerative Health Podcast,
I interview Professor JeffreyGuy, medical doctor, former
pharmaceutical executive andbillionaire philanthropic
founder of the Guy Foundation,leading what I believe to be the
world's most innovativeresearch in health that of
quantum biology.
In this interview, amongst awhole range of fascinating
(00:22):
topics, we discussed the role oflight in life, both
endogenously generated light,also known as ultra weak
biophoton, and exogenous lightin the form of solar photons and
their interactions.
I believe the light and lifestory explored through the
lenses of quantum and circadianbiology, hold the key to
(00:45):
understanding and preventingchronic disease and optimizing
human health.
Hope you enjoy this podcast.
Professor Guy, you've got avery interesting story that
started with a medical schooland detoured through the
pharmaceutical industry and hasnow arrived at this fascinating
work with quantum biology, soperhaps you could give us a
(01:06):
brief idea of that story.
Speaker 2 (01:08):
Well, I think the
briefest idea is I often
describe myself as a full-timemedical student with a giant
size, chemistry says, buteffectively.
After a few years in medicalpractice I was training to be an
obstetrician in London Idecided to take a sabbatical for
a year to do some medicalresearch in the pharmaceutical
(01:31):
industry and, very fortunately,with a pharmaceutical company in
the south of France.
It seemed extremely attractiveat the time.
I had done a degree inpharmacology during my medical
training, so that helped me alittle bit and I went off to
France to work for a drugcompany called Pierre Farbe.
I worked for Monsieur PierreFarbe at the time.
(01:51):
He had developed his businesson the back of plant medicines
but was moving into new chemicalentities and my role was to
take those first dose in man andthen up into proof of concept.
And I did that for a few years,came back to the UK and worked
for a company that developedcontrol release morphine for
(02:13):
terminal pain and other drugdelivery products.
And having worked for twoprivate pharmaceutical companies
at the tender age of about 29or 30.
I thought, well, I'd give it ago myself and started my first
pharmaceutical company which wasEthical Holdings, which was a
drug delivery business.
We did that in the mid-80s andin the 80s and 90s.
(02:36):
Drug delivery, control releasetablets, different dosage forms,
skin patches those were verypopular in terms of advancing
therapeutics and I came out ofthat company in about 97 and had
(02:56):
previously considered whichother products would benefit
from modern drug delivery and,having worked with plant
materials opiates, for example,for pain I'd focused on cannabis
and cannabinoids in the early90s but had given that idea up
(03:16):
when the regulators had told methat over their dead body would
they allow me to do anydevelopment with a cannabis
medicine.
That had changed by the late90s.
Uh, the courts and theregulators were under pressure
from a widespread use in incases like multiple sclerosis,
the arthricities, rheumatoidosteoarthritis, cancer pain, for
(03:39):
example, and her majesty'sgovernment asked me whether I
would address the issue ofdeveloping a medicine from
cannabis.
So I set up GW Pharmaceuticals,gw being my initials and those
of the surname of my foundingpartner, brian Whittle, so Guy
and Whittle.
And to cut a long story short,over 20 years we developed
(04:04):
Sativex for multiple sclerosisand then the one that most
people know about, which wasEpidiolex, which is cannabidiol
for treatment of catastrophicepilepsies in children now
extended into adults, foundourselves really at the
(04:27):
forefront leaders in cannabinoidscience uh, having turned it
from being a cannabis plantbeing considered as an illegal
plant to a medicine of thefuture, and I think it was
either scientific american Ithink it was scientific american
or nature that described thecannabinoids as the aspirin of
the 21st century.
So we enjoyed very much beingable to research this complex
(04:48):
area of medicine very complexarea indeed.
But I was always concerned that,being chairman of a NASDAQ
company, by that time we werelisted on NASDAQ, there was the
chance that one day somebodywould come along and want to
take the company over, and I wasconcerned about really falling
off an intellectual precipice.
So my wife and I, in 2018, thiswas a number of years before we
(05:10):
did sell the company weestablished the Guy Foundation
to look at the next generationof interesting research, and I
often quote at the beginning ofmy lectures that Albert Einstein
once said that if an ideawasn't at first considered
absurd, then it had no chance,and I suppose I consider myself
(05:33):
as the purveyor of absurd ideas.
The last 40 years opiates intomodern use in the hospice and
terminal care environment, thenmaking medicines out of
cannabinoids.
We thought the next step mightbe to look at something new and
something over the horizon again, and that work really came out
(05:55):
of some of the work that we weredoing trying to identify the
mechanisms of action and thepharmacology of the cannabinoids
, the pharmacology of thecannabinoids Fantastic.
Speaker 1 (06:05):
So that is quite a
pivot from cannabinoid and based
medicines and pharmacology intoquantum biology.
So what was the, I guess, sparkthat made you look into perhaps
from a biochemical to more of abiophysical and biophysics
perspective?
Speaker 2 (06:25):
Having spent 40 years
researching medicines and doing
a large amount of pharmacology,when we were looking for the
mode of action of thecannabinoids, it was difficult
to pin it down in terms ofclassical Hillian pharmacology.
And the more we looked at itand the fact that the
(06:46):
cannabinoids seem to work acrossa wide range of therapeutic
indications and work almostacross all types of mechanisms
and biological systems, wethought the link was always the
bioenergetics, that everyprocess, every mechanism of
action required energy.
And we likened it to perhapsone arriving home finding that
(07:11):
one's remote control for thetelevision doesn't work and the
transistor radio doesn't workand your alarm clock doesn't
work and you work out, you'veactually bought a box of bad
batteries.
And so we began to focus oncellular bioenergetics and I
took the view, oh, probably 15years ago, that I thought the
cannabinoids were working bymodulating intracellular
(07:31):
bioenergetics and that wouldthen be the balance of energy
production and usage, energydissipation and, of course,
inflammation, that fine balanceof intracellular energy and
inflammatory media.
(07:53):
So we began to think about, uh,the cannabinoids working at the
level of mitochondria and atcellular bioenergetics.
Initially we were producinghypotheses that worked on
standard or more classicalthermodynamics and at the same
time my colleague, long-standingcolleague, professor alistair
nunn, had started to try andimage cannabidiol inside
(08:14):
mitochondria, and we were usingtwo photon microscopy to do that
, where one?
Uh bombards the molecule withone photon uh increases
excitation state a little andthen 20 femtoseconds later you
bomb up with a second thatexcites the molecule and one
could image it.
And what we found is, as soonas we exposed the cannabidiol to
(08:38):
any amounts of light and incertain wavelengths, it
fluoresced.
And what we realized, of course, is that we were dealing with
the, with quite quite a strongchromophore, and that, very
quickly uh made us really shiftour thinking from classical
thermodynamics into quantum uh,quantum science, and it was from
that notion that we thoughtperhaps we ought to look more
(09:01):
deeply into the underlyingmechanisms of the fundamentals
of how cells work and how allcells work, and that led us into
quantum biology.
Speaker 1 (09:12):
And that was probably
about seven or eight years ago,
a couple of years before westarted the foundation 2024, and
you've had an absolute stellarlineup of cutting-edge
scientists present in youronline series, including names
(09:33):
like Nick Lane, doug Wallace,michael Schifra.
I mean, these are at thecutting edge of quantum biology,
and so it seems to me, from aperspective also of trying to
understand how to best optimizehuman biology and prevent
chronic disease, that this iswhere we're at collectively and
(09:55):
this is nothing that is what iscurrently being addressed in, I
guess, mainstream medicine, orcentralized medicine as it
stands, and perhaps we could, uh, start now with some real basic
overviews of quantum biologicalconcepts so that people really
understand, um, this at the baselayer.
So so maybe, um, how would youdefine quantum biology, um the
(10:21):
term maybe, to start with?
Speaker 2 (10:29):
Well, I think, in the
simplest terms, biology is a
study and really reverting tofundamental quantum mechanics as
it might impact on biologicalsystems.
I think that's the simplestapproach to take.
Speaker 1 (10:43):
And what are some of
these quantum biological
processes that are occurringinside cells?
Speaker 2 (10:51):
Well, you could start
in a number of places.
I often start at the notion ofsort of the transition between
the last 80 or 90 years ofintense pharmacology, where
medicine has been based almostentirely on chemicals, passenger
chemicals and chemicalreactions, with some
(11:12):
cross-reference to electronicsgoing on in nerves, for example.
So I think the wave-particleduality is the one that a lot of
people have a difficulty inunderstanding, that perhaps the
stick-and-ball structure of amolecule, as we know it, is only
really the most highly probablestructure when you transfer
(11:35):
from a wave to a particle.
And I think that once one cangrapple with the notion of a
molecule being more a wave, thenthis pinpoint target, this
silver bullet approach that hasbeen taken for the last 50 years
or so in pharmacology, wherethe molecule has to arrive in a
(11:55):
specific, like filling a roomwith with, with a gas, as
opposed to in individual uhcondensed particles.
So that's probably where I tendto start when I I talk to
(12:18):
people.
Then there's the issue oftunneling uh, that, uh, that,
that uh.
We can see that one wave, onewave form, can tunnel through
another and that really bringsus to the electron transport
chain and we, in the very earlytime, with our foundation, I
really set the objective oftrying to visualize tunneling,
(12:38):
and we haven't quite done thatyet.
And then there's the issues ofsuperpositioning and then the
very alluring thought ofentanglement.
Over the last six years or so,I think, we've seen a lot of
these issues that can now beplaced in a biological setting
(13:02):
where, prior to that, mostpeople thought quantum mechanics
had to be very, very cold andextremely dry.
And now we know that quantumbiology will occur in in a a
warm, wet setting of of of thecells.
And I think, probably morerecently was the notion of
(13:22):
quantum spin, and that's whatled us to thinking about the
space, the hazards of space,health.
Speaker 1 (13:33):
Do you mind
explaining quantum entanglement
and quantum spin a little bitmore?
Speaker 2 (13:38):
Well, einstein didn't
like the idea of it.
I think he called it spookymotion at a distance.
This is the notion that youhave two particles or waves
photons, electrons, protons thatare linked to one another in
some way or other andimmediately one considers the
notion of some informationtravelling between them.
(13:58):
So if one has an electronspinning in one direction and it
has its entangled buddyelsewhere spinning in the
opposite direction, if, if thefirst one changes, the second
one is thought to changeimmediately and without, without
any perceptible change in itsentropy or energy.
And the notion that a lot ofpeople have is is that there's
(14:21):
some sort of communicationbetween the two.
And I think that the bestunderstanding is that these two
entities, particles or waves,are mathematically correlated.
They originated from the samepoint, they're mathematically
correlated and therefore will bemathematically correlated,
irrespective of the distancebetween them.
(14:42):
And of course, a number ofpeople say that one of them
might might be here, the otherone might be the other side of
the galaxy, and that tends toflummox most traditional
conventional scientists straightaway.
Uh, but uh, the you know, therecent experiments, as you know,
uh, have now pretty muchdemonstrated that entanglement
occurs.
I think most people aresatisfied that entanglement does
(15:04):
occur, but it does give theopportunity for us to think
about how messaging andinformation could spread
throughout biological systemsfar quicker, with far less
energy than had previously beenthought.
As you know, you and I, ourbrains are using about 20 watts
of energy at the moment.
A computer that could do theamount of computing that we do
(15:27):
would probably need about 85,000watts.
So entanglement is veryexciting.
Also, it's interesting tounderstand how, for example,
entangled photons might impacton molecules, and I don't know
whether you know the work of TedGoodson from University of
Michigan.
He bombarded a levithlorineanesthetic molecule with photons
(15:53):
and established a, aconventional excited state.
But when he then repeated thatand bombarded them with
entangled photons, he achieved avirtual excited state which
they considered would only existabout once in every 10 million
years.
So what intrigues us is, whenwe're thinking about bio photons
(16:18):
, those ultra weak emissionsfrom mitochondria and from other
parts of the cell, whatintrigues us is to what extent
any of those might be entangledand to what extent they might be
altering the molecularinteraction of incoming
molecules.
So that's something probablyfor the next decade to work out.
Speaker 1 (16:40):
Incredibly
interesting and maybe it's a
good opportunity to brieflydescribe the the onion root
experiment and I know you had aprize and named after that, uh,
onion root in terms of thehistory of, of this idea of, of
biophoton research, and for thelisteners who previously um
listened to my podcast, myepisode with dr jack cruz, he he
(17:01):
mentioned um alexander gerwichspecifically, so, um, yeah, do
you mind sharing yourperspectives on on that, on that
experiment?
Speaker 2 (17:09):
yes, well, you know,
when we started the foundation
we hadn't been particularlyaware of Alexander Gurwitch's
work, but essentially, in 1923he was able to show that there
was information transfer fromthe one onion root to another
onion root and to increasecellular division at the second,
(17:30):
second onion root and heidentified this as as
effectively, uh, bio photonsbeing released.
Uh, and his view they wereactually in the ultraviolet
range, interestingly enough, andhe published that work in 23.
There was a lot of skepticismabout it and it was really.
(17:53):
I think it was only in the1960s that I think his
granddaughter finally producedthe definitive work to show what
had been done there, show whathad been done there, um, but the
use of um or the notion thatthat light will transmit
information between uh, betweencells or between organisms as a
(18:14):
whole, um was of course uh usedby finson, for example, in when
he received the uh nobel prizein 1903 for treating um,
received the Nobel Prize in 1903for treating lupus vulgaris
with ultraviolet light as well.
So in the early part of lastcentury there was quite a lot of
interest in the use of light interms of biology, in terms of
(18:36):
therapeutics, but that, I think,got very much lost once we got
into the post-war era of thepharmaceutical industry's
patenting molecules and most ofmedicine, and most medicine
research for the last 80 yearsor so has been based on purely
molecular interactions.
Speaker 1 (18:55):
And that's a point
that I really want to emphasize,
which is that this storyappears to be one of both
external light, which is theinteractions of, say, solar
photons on biological systems,but also one of external light,
which is the interactions of,say, solar photons on biological
systems, but also one ofinternal light, which is the
light being endogenouslygenerated by structures like the
mitochondria, and I believeGerwitz he used a piece of
(19:18):
quartz to show that thebiophoton emission was in the
ultraviolet range and that wasactually blocked by glass.
What's your perspective on this?
I guess, distinction and therole of, perhaps, solar photons
versus endogenously generatedphotons in the quantum biology
(19:39):
story.
Speaker 2 (19:40):
Well, biology is
pleiotropic.
It will use every asset that'savailable and time and time
again, and some processes areconserved very highly throughout
evolution from much lower orderorganisms.
The sun and solar photonsrepresent almost entirely the
(20:06):
source of energy that we haveused to evolve and therefore it
seems to be no surprisewhatsoever that there are very
many systems in our body thatare either using photons,
communicating with photons, orindeed, as part of their process
, producing photons.
(20:26):
Now, solar photons, of course,of the farm, more of them,
honest, sort of sunny day.
Here in England we've got about10 to the 14 or 10 to the 15
photons per square centimeterper second in a normal daylight.
I suspect in Australia it mightbe a bit more.
And the bio photons, or theultra weak emissions, we're down
at 10 to 100 or so.
(20:47):
So they are very, very uh weakindeed.
So I think that the biophotonsare really acting on on
structures very, very close uhthemselves if they're acting
physically, but of course thereare the opportunities that they
may well be entangled.
Speaker 1 (21:06):
The solar photons, of
course, in different
wavelengths, can penetrate cellsand whole organisms quite well,
especially in the red range andspecifically with regard to the
frequencies or the wavelengthsof light of these, of these bio
photons.
As you mentioned, Gerwitz'swork suggested there was
(21:28):
ultraviolet range.
What is your perspective orwhat have you noted in terms of
what ranges of light are beingemitted by different parts of
the cell?
Speaker 2 (21:42):
Well, different
authors have produced different
estimates there and it seems tobe across the range and I
suspect it might well be horsesfor courses.
It may well depend on thepurpose, function and downstream
objectives of a process, aprocess.
So we're still in the earlyphase of really trying to
(22:07):
characterize these emissions.
With our facilities now atHarwell, where we work with
central laser laboratories,we're able to detect down to a
very, very small number ofbiophotons.
(22:27):
It the equipment is in abasement, in a box, in another
box, another box to exclude allour extraneous light, and we
have to settle the systems down.
Um, we have to be be carefulwe're not just detecting delayed
luminescence that can confusethe situation.
So we're wanting to characterizethe biophotons and the ideal
(22:47):
outcome for us would be able tocharacterize biophotonic
emissions from mitochondria that, for example, are distressed
and are communicatingmitochondria next to them.
Now we demonstrated that in astudy that was published earlier
in the year Reesoulton et alwhere we showed non-chemical
communication betweenmitochondria.
(23:07):
So if that communication iswith biophotons, it would be
very, very interesting tounderstand if there is a
specific characteristic and whatinformation is being carried by
a biophoton and if we cancharacterize biophotons which
are being released frommitochondria, perhaps saying,
perhaps saying look here we havea problem.
Saying to the next groupmitochondria, here is the
(23:30):
solution and, by the way, here'sa small packet of energy to
deal with that problem.
If that's what's going on,could we then, with our own
lasers, emulate uh, that by aphotogenic emission, and re uh,
re--impact or re-targetmitochondria, so that we could
produce something that we'vecalled for the last 10 years
(23:51):
mito-tuning, and we retunemitochondria by not only, not
only, directing a range ofphotons at them, but photons
that are specificallycharacteristic of those type of
biophotons that would have beenemitted by the mitochondria.
Speaker 1 (24:11):
So that's one of our
objectives over the next few
years.
Fascinating, and it gets tothis idea of the function of the
mitochondria and I think, atthe really superficial level,
what people are taught inuniversity and school is that
these are just power plants ofthe cell.
You know using that term.
But looking at what's occurringon the electron transport chain
(24:34):
, the kind of the exhaust ofthis mitochondrial engine
obviously is water and it's alsothese biofotons.
I mean, they're actually beingspewed out, so to speak, but
perhaps not as waste.
But what you're suggesting isactually perhaps as a signalling
(24:58):
and information transfermechanism.
Speaker 2 (25:01):
Yes, look, at school
we were all told that
mitochondria were the powerhouseof the cell, and I think most
life scientists would think ofit as like a sort of a brazier.
If you imagine, on a picketline, everybody around warming,
warming their hands around a, abarrel with a, with a fire in it
, and just the energy is justbeing distributed in any, any
direction.
We initially began to learn itwas really more like a sort of a
(25:26):
mobile phone battery which wasdiscriminated as to what, how,
the amount of energy it wouldprovide.
At certain times Mitochondriaseem to be able to discriminate
which systems do need energy andwhich ones don't.
And the example we give is ifone has two neurons requiring
(25:47):
energy, if one gets energy andthe other one doesn't, the
mitochondria can actually alterbehavior.
There has been work associatingmitochondrial function with
memory, for example, and we'vemoved even further on.
And if you go to a power station, yes, yes, there's a lot of
energy generation, but probablythe most important room in the
(26:08):
entire power station is the oneswhere they have all the knobs
and dials to decide how muchpower to produce, how much to
dissipate, because they don'twant to overfry the system.
Uh, you know, removing the rodsfrom your, your nuclear power
plant, just so that you'reproducing just the right amount
of energy and directing it tothose parts of the grid that
(26:31):
need it.
And it's that information roleof mitochondria that seems to
come to the fore.
And, as you know, with plants,of course they use the
chloroplasts to produce theenergy, but they also have
mitochondria.
So it seems that mitochondriaare communicating not only with
other mitochondria within thecell, but of course cells
(26:57):
exchange mitochondria,mitochondria exchange throughout
the body and there are actuallyfree-flowing mitochondria in
the plasma, although the jury'sout as to whether those are
fully functional or not.
So part of the process ofelectrons huddling their way
down or flowing down theelectron transport chain is that
needs good quantum coherence.
(27:17):
If there's any interruption orperturbation of that, then one
has this sort of visualizationof electrons spilling off the
side and that get doing a coupleof things, producing reactive
oxygen species and possibly abyproduct or a primary product,
that is, the release of photons.
So every time you get Rossproduced, you'll get photo that
(27:40):
by photons produced.
And it certainly seems to be acase that when mitochondria are
distressed or reactingadaptively, a lot of people
describe mitochondrialdysfunction.
What they really mean is thismitochondria is functioning
perfectly well, but right at theextremes of their flexible
limits.
When they're distressed, itseems that there's a much
greater release of theseultra-low emissions, these bio
(28:04):
photons.
Speaker 1 (28:06):
It prompts a
fascinating question, and I had
a funny image in my mind of theSimpsons and Homer Simpson at
the nuclear power plant with thedonut and red lights going off.
But where, do you think in themitochondria, might that
coordination decision be made interms of, perhaps, the
wavelength or the direction oflight emission?
Speaker 2 (28:28):
Oh, I think the
answer to that is we don't know
at the present.
One can begin to sort ofreverse engineer and speculate
in so much as can we identifypathophenotypes that may be
associated with irregularitiesat different parts of the chain,
and one of the culprits in theelectron transport chain, I
(28:53):
think and if you listen to NickLane's talks on this would be
complex I.
So I think complex I seems tobe or the abnormalities or
perturbations of complex, I seemto be responsible for a number
of pathologies.
And then, of course, down atthe other end of the chain, the
ATPase, of course, there are anumber of mutations in the
(29:16):
proton channels there that arealso associated with illness.
So we could sort of derive fromcircumstantial evidence which
parts of the chain might be moreimportant than other in
communicating messages, in termsof homeostasis and in terms of
adaptive responses, by when theyfail, when the adaptive
responses fail to adaptappropriately, and then that
(29:40):
appears as illness or disease.
Speaker 1 (29:43):
But think, uh, we're
not, we're not there yet, unless
you have a a better notion no,no, no question just came to my
mind as as you were speaking,and a question, another question
that really uh sparked my mindin terms of the production, the
endogenous production of light,and and really, when you
(30:03):
mentioned the neurons, is uhthis idea of the endogenous
production of light?
And really, when you mentionedthe neurons, is this idea of
endogenous light production as asignaling mechanism in the
brain?
And obviously clinicians in theaudience will know that the
brain exhibits a form of melanincalled neuromelanin, which is
(30:24):
in uh in alzheimer's disease issorry, parkinson's disease, is
is a pathologically lost and thetheory that um dr jack cruz has
proposed is that that thatinternally uh exhibited
neuromelanin is acting as a formof um way of capturing
indulgently generated light, umin terms of signaling and other
(30:44):
functions, and and further tothat point, is that we have
expression of these non-visualphotoreceptor proteins like
melanopsin, throughout,throughout the brain.
Do you have any insights orthoughts on on those those
points particularly?
Speaker 2 (31:00):
well, I mean clearly,
um, our, our view is that light
is important for the normalfunctioning of the brain, both
endogenous and exogenous.
To deal with the endogenousinitially, some of the molecules
to which you're referring couldpossibly better be thought of
as antennae to really detectincoming photons and deal with
(31:26):
them and respond in thatappropriately.
There's a lot more work thatneeds to be done to drill down
and to silo down in a sort of anold-fashioned reductionist
basis to work out what's goingon immediately.
But what we do know is is that,um, with exogenous light, there
are a number of good studiesnow showing the change in neural
(31:51):
function, neuropsychiatricfunction, with exposure to
different wavelengths of light,mainly of course in the red,
near of red and of course muchhigher wavelengths near infrared
and of course much higherwavelengths.
I always think it's strange thatone of our most precious organs
, the brain, isn't buried deeplyin our body, surrounded by
(32:17):
layers of fat as a shockabsorber, but it's sitting
exposed with a very small, thinpiece of bone around it, and we
now know that of course, certainwavelengths of light, certainly
the red light, will pass wellinto the brain, if not all the
way through.
And so to have an organ likethe brain that is exposed to
(32:39):
sunlight for the entire lengthof our evolution, and to know
that also, that the cells withinor the organelles for the
entire length of our evolution,and to know that also, that the
cells within or the organelleswithin the cells within the
brain are also emitting lightthemselves.
It seems to me that we'resomewhat two or three decades
late in thinking about this inthe context of advances made in
(33:01):
medicine elsewhere.
Speaker 1 (33:04):
Certainly, and that's
the work of scott zimmerman,
who's an optics engineer,showing that the uh, the sulca
and the gyria really opticallyoptimized to to concentrate near
infrared photons um into the,into the gray matter.
That that really makes sense,um as well to me, from from a
longer wavelength light point ofview.
But I'm curious, uh, if youhave some thoughts on how
(33:26):
shorter wavelength light, sayultraviolet, could be
penetrating into the deeperinside the brain regions when we
know that they're moresuperficially absorbed because
of its wavelengths.
Do you have any thoughts onthat?
Speaker 2 (33:42):
Yes, well, I think
the shorter wavelengths scatter
far more readily and I know youspoke with Bob Fosbury and he'll
talk to you about first-orderscatter and so blue and
ultraviolet will not reallypenetrate very far at all into
biological systems, whereas thereds and infrared and the
(34:07):
invisible, non-visible lightphotons will pass all the way
through.
It would seem, therefore, thatover evolution, those systems
that are going to use the energyof light and the information
that can be carried by it andproduced within it, within light
(34:33):
, will have effectively evolvedaround those, um, those
wavelengths that can, uh, thatcan penetrate the whole of the
organism.
Um, and equally uh.
It's very helpful that,although ultraviolet light was
probably extremely important inthe very early days of evolution
to be the very high energysource, of course as soon as DNA
(34:55):
became available to organismsit was extremely damaging, and
so one of the earlier processesin evolution must have been a
way of dissipating energy notrequired.
So if early organisms wereutilising and manipulating light
(35:16):
energy and light information,there would have to have been a
very good system to dissipate itso that organisms effectively
didn't fry themselves.
So we I think medicine isbeginning to understand a lot
more about the therapeuticimplications of anything above
(35:37):
600, 630 nanometers, and I thinkthere's some catch-up needs to
be done in terms of theultraviolet spectrum.
Speaker 1 (35:47):
Yes, and the point
about dissipation.
I mean, I think that is that'sa pretty critical again concept
in quantum biology.
Can you explain dissipation andthis idea of a dissipative
system?
Speaker 2 (36:07):
explain, uh,
dissipation and this idea of of
a dissipative system.
Yeah, I mean, I think if, if,if you're going to tap into an
endless source of energyemanating from the sun, um, one
has to have a way of having asafety valve in terms of not
being exposed to too much.
Now, of course, withmitochondrial function, the
mitochondria can uncouple sothat you get futile cycling,
(36:29):
usually resulting in increasedheat in the cell, and it's often
said that mitochondria areworking at a much higher
temperature than surroundingtissues.
There was paper a couple ofyears ago saying the temperature
is nearly up to 50 degreesinside those, a couple of years
ago saying the temperature isnearly up to 50 degrees inside
those.
So I think any system that isusing a freely available,
abundant source of energy has tohave a way of capping that
(36:53):
energy and dissipating itappropriately.
And obviously, in terms ofoxidative-phosphorylation, that
would be done by uncoupling theenergy production.
Speaker 1 (37:09):
And a point to
revisit quickly the quantum
coherence state.
There's a suggestion that oneof the roles of sleep and
stillness at sleep is tofacilitate quantum coherence.
Do you have any thoughts onthat?
Speaker 2 (37:28):
Oh, I think that's a
very interesting notion.
The brain, modern humans, are,I think, subclinically inflamed.
I often say that the averageRoman soldier was about 7% fat
or lipid, whereas the averagehuman today is more.
(37:48):
So we have a lot of ectopiclipid that leads to increased
intracellular inflammation and Ithink it's fair to say that the
humans as a whole, throughtheir lifestyle, have mild
mitochondrial dysfunction andintracellular inflammation.
So we live in a far moreinflammatory milieu than
previously.
The brain will also be the same, will be slightly inflamed, and
(38:12):
it's only during sleep thatthat process can be reversed, I
think, and certain times duringsleep and that's very important
to to, to to, to understand whenthat might be another.
The work is only in thebrilliance, only early phases,
how sleep and quantum coherenceget.
Speaker 1 (38:34):
Yeah, that's a very,
very interesting thought and
something that we're looking at,looking to and so I guess, uh,
understanding that themitochondria really, to me, is
the, the logical focus should bethe logical focus of a medicine
, health science, uh, as itstands, and and, and you know, I
(38:58):
like to often reference dr dougwallace because his concept of
this bioenergetic etiology ofdisease was one that posited
that we shouldn't be looking atindividual organ-specific
problems.
We should recognise diseases ofchronic nature in the kidney,
in the heart, in the brain, assimply organ-specific
(39:20):
manifestations of a bioenergeticfailure.
Organ-specific manifestationsof a bioenergetic failure.
So how would you describe thatkey concept in terms of, yeah,
focusing on chronic disease andpathology.
Speaker 2 (39:37):
Well, I think I agree
entirely.
When I was a medical student,and I think even now, one is
really taught that the body isself-satisfying, it provides its
own homeostatic environment,and that illness and disease is
sort of the addition ofsomething new.
Something extra has been addedand that's what's causing the
(39:59):
problem.
We take a slightly differentview and that is we're probably
getting ill all the time.
You've probably done that a fewtimes since this interview
started but we have theprocesses to correct, to
regenerate and to repair, andwe're doing that all the time,
and that the emergence of somediseases and a lot of diseases
(40:23):
is not really something new.
Has turned up Now we can thinkabout, you know, if one has been
shot or there's a virus orsomething turns up, but
generally it's the loss of thatability to continue to adapt and
regenerate, which tends to getworse as one gets older, and all
of those processes that we seein terms of mitophagy, in terms
(40:45):
of apoptosis and regeneration ofcells and reusing some of the
waste that is produced by aftercell death.
Those all roads lead back tothe mitochondria, not only in
terms of providing energy to doit, but the signaling and the
messaging as to when to do it.
I think that having a sort of auniversal notion that
(41:11):
mitochondrial dysfunction wouldlead to certain diseases and
organs, I don't disagree withthat.
But we also know thatmitochondria tend to be somewhat
different.
If you look at the mitochondriain the retina or in the heart
or in the brain, their form andfunctioning can seem to be
different to other parts of thebody.
(41:32):
So there is some organ or organor function specificity that
would add to another layer.
Speaker 1 (41:39):
if you've got a
generalized mitochondrial,
dysfunction and this concept ofmitochondrial heteroplasmy I
mean, as I understand it, it'sthe accumulation of
mitochondrial DNA mutations thatare affecting essentially the
translation of key proteins onthe electron transport chain and
therefore the ability of themitochondrion to produce energy.
(42:02):
And if there is and I guess theother point to make here is
that if these are ancientbacterial remnants, which you
know, so we understand them tobe, then their circular DNA is
not protected by a nucleus andthe accumulation of reactive
(42:23):
oxygen species from a leakyelectron transport change is
therefore potentially renderingthem more susceptible to damage
and therefore accumulation ofmutation.
So can you speak tomitochondrial heteroplasmy and I
guess, the role it plays inpotentially dysfunction or what
(42:47):
you described as acting at theedge of its capabilities?
Speaker 2 (42:53):
Yes it's an area
where very few researchers and
clinicians really have areasonable understanding.
But it would seem that thehuman body adapting, especially
if you've got a genetic disorderthat is leading to a high level
of mutation.
(43:16):
It seems that we can toleratereally quite a high amount of
mutation.
Probably 50-60% of themitochondrial DNA could be
mutated to maintain somefunction.
But I think if we look moreclosely we will see that more
generally amongst the population.
But it just doesn't, it doesn'trepresent itself as a, as a
(43:38):
pathophenotype um.
What we do see, of course, is uma very close association now
between mitochondrial uh dnamutation and and tumors and
cancers.
So when we look at cancers now,we can identify some
mitochondrial DNA mutations very, very regularly in those areas.
(44:03):
So this is an area I think thatis going to need a lot more
research in terms ofunderstanding how much
background heteroplasmia thereis and at what point is there a
threshold above which themutated dna determines the
ultimate function in terms notonly in energy production but
(44:27):
probably in information transfer, and it may be in a lot of
conditions that the informationtransfer is the thing that
suffers first, for example intumors, in cancer cells, where
the possibility is that energyproduction continues, but
information transfer issuppressed, which allows then
(44:50):
the tumor to grow somewhatrampantly without getting the
feedback from surroundingsystems and organisms.
Speaker 1 (45:00):
And on that topic I
think it was Michael Schiffer's
presentation where he describedthe charges of different cell
types, and in tumourous cellsthey notice a loss of negative
charge.
Can you explain that concept orhow you think about it?
Speaker 2 (45:18):
It was fairly new to
me that he was talking about
both that in tumors, but also inwound healing as well.
the differential charges sowe're beginning to understand a
lot more.
And this again comes away fromthis idea that everything is to
do with the transfer ofchemicals, the old view of a
ligand sitting on a receptor.
And that's not really the onlyway that biology and medicine
(45:41):
works.
The charge across membranes inhumans, or in any organisms, is
enormous.
The charge density across amembrane is something like three
times that of lightning.
So you know, michael, you know,I think that's absolutely right
in identifying that there isthe differential charges.
(46:04):
And if they change in, say intumors or inflamed tissues or
where repair is occurring, wherethere's been damage to tissues,
then this is far more importantthan has been thought of before
.
We have to somewhat get togrips with what's going on and
how can we either manipulate orenhance the appropriate changes
(46:30):
in charge to accelerate or toimprove healing and improve
therapy.
Speaker 1 (46:38):
Yeah, and that makes
sense to me and I think you also
.
As you.
As you mentioned, he describeduh stem cells and they have a
similar d d uh lack of or lossof um of charge.
So there's uh, there's, there'sobviously a very intentional
effect of the human body's usingum to to uh is using when it
comes to that, and the otherreally big concept that I'd like
(47:02):
to hear your thoughts on isbiological semiconduction and
this idea that proteins that arein the body are essentially
semiconductors.
Can you speak to that idea?
Speaker 2 (47:18):
Not greatly.
It's not a very specific areaof my knowledge at the present.
We had a very good lecture byJudith Klinman a couple of years
ago on that.
What we are looking at, though,though, in terms of the
(47:38):
interaction of protein with itsenvironment and the notion of
semiconductor, is looking at thelayers of water, the
interfacial water aroundproteins, and how that extends
out way beyond the sort ofnormal distances that you would
normally see bonding charges,for example and our symposium
(48:02):
series actually next spring willconcentrate entirely on water
order and the quantumcharacteristics of water.
I think it's been completelyoverlooked in terms of medicine.
We sort of think water's waterand nothing's going on there.
So let's think about theproteins.
So I think, when you look atthe protein and proteins, how
(48:24):
they interact, we have to lookat the interfacial water around
the proteins and understandwhat's going on there as well.
And, of course, as you bombardthat water with photons, you
alter some of its characteristicand including some of the
thought, also the viscosity,which some people say has an
(48:45):
impact on the speed or thereadiness of which the ATPase
will nanomotor will rotate, forexample.
So we're going to be lookingclosely at water and certainly
how it interacts with proteins.
In terms of proteins andsemiconductors, you'd probably
have to talk to my scientistsabout that, I think.
Speaker 1 (49:07):
Yeah, no worries, the
water and light interactions
are absolutely fascinating and,yeah, the idea that different
light frequencies are changingthe biophysical uh function of
water, I mean that that isincredible.
And the, the idea that evenwater is potentially uh, has a
storage and information storagecapacity, I think is is one of
(49:29):
those corollaries which uh,which is is mind-blowing and
again, so far beyond the, thecurrent state of the art in in
clinical practice, and and um,and and medicine, uh, maybe one
more, one more questionspecifically on, on the, I guess
, the biophysics of this, beforewe uh, we change tack and do.
(49:50):
Can you speak to temperatureand and how temperature is
affecting quantum processes andperhaps mitochondrial function?
Speaker 2 (49:59):
Yes, it's a very
interesting thought.
For the last 20 years I'veasked all my colleagues a simple
question, that is, how does acell know what temperature it
should be inside?
Is it managed by a series ofthermostats or whatever?
Or is the temperature within acell or within an organelle
really the the result ofstraightforward physics, of a
(50:21):
group of molecules in a vacuolebeing with a source of different
sources of energy, bothchemical energy and photonic
energy, um, uh and electronenergy.
When you mix all of thosefactors together, does that
determine what that, what thetemperature would be?
(50:41):
What is the set point of thecell?
Because if you think aboutconventional medicine and
conventional chemistry, if youchange the temperature, the
equilibrium within all of the uhchemical reactions is going to
change, and so we have this ideaalso that temperature is more
(51:05):
important to quantum effectsthan heat.
So I think it's reallyimportant to understand how
temperature is regulated.
Is it regulated or is it just aresult of the physics and
chemistry that has been producedwith the exogenous and
endogenous energy supply?
And, as I mentioned early on,it's thought that the
(51:27):
temperature within mitochondriais much higher than the
surrounding materials.
And we do know, for example,quite a number of plants.
Certain plants are exothermic,can produce temperatures
regionally within themselves ofsix or seven degrees higher than
the surrounding ambienttemperature.
So temperature very important,but it's really understudied.
(51:50):
And the understanding of howtemperature is maintained or
modulated within the cell is itreally needs further study.
And I've come back to thingslike uncoupling.
The result of uncoupling in theelectron transport chain will
be an increase in temperatureand that might be not a
(52:12):
byproduct, but that might be theprime reason why we see
uncoupling.
Speaker 1 (52:16):
If one alters
temperature then the quantum
processes about which we'retalking will themselves alter
yeah, and it makes me think ofthe, the clinical implications,
which is people and when theyget cold quite often and
essentially stimulatethermogenesis and in the body
and the development of thingslike brown adipose tissue.
(52:37):
And this has been used as anintervention to reverse
metabolic syndrome and type 2diabetes and it's basically
upregulating the thermalgenerating effect and helping
pull things like that ectopiclipid out of the wrong place and
essentially burning it.
So I'm definitely intrigued atthat temperature and how that is
(53:00):
affecting things.
And the other real interestthat I have is the circadian
system and circadian biology andobviously it's critical to the
whole organism function.
Do you have any thoughts abouthow circadian biology and I
guess, the clock genes, whichare nuclear in nature in the
(53:23):
nucleus, how are theyinterfacing with the
mitochondrion?
How are they affectingmitochondrial function and
perhaps optimizing mitochondrialfunction?
Speaker 2 (53:35):
An interesting
thought.
I mean just going back to thepoint you made before, though,
not all humans share the samecold genes and therefore, for
example, the Arabs, the PimaIndians, those that are
suffering very high levels ofmetabolic syndrome, are unable
to futile cycle, so when theyget cold they need to shiver.
(53:55):
So it's interesting that it'snot the same across all In terms
of we're just, I think, at thebeginning of understanding the
more important role thatmitochondria have in determining
circadian rhythm, determining acircadian rhythm and the
(54:20):
downstream effects that mighthave on on aging.
Uh, again, most physicians ofour generation would have been
taught, told, that melatonin,for example, comes from the
pineal gland and that's the endof the story.
We now know probably most of itcomes from mitochondria, uh the
.
So mitochondria clearly very,very strongly involved in
circadian, in the controlcircadian rhythm.
(54:43):
Now, how that interacts withthese nuclear clocks, I'm not
entirely sure, but I'm sure youhave a lot more scientists with
whom you speak which willunderstand that better than me.
But what we do see is the linknow between the alteration in
(55:03):
mitochondrial functioncontrolling circadian movement
and the red-blue balance towhich we're being exposed, and I
think you've spoken about thisand some of your guests have
spoken about this that thereseems to be a sort of a red
starvation and a sort of a shifttowards blue, which is
inappropriate for where theperson was born or lives.
(55:25):
And as we've come under that,comes under stress, we might
revert back to the red-bluebalance that might have been
needed nearer the equator acouple hundred thousand years
ago, before humans were startedto migrate.
But that's the area thatinterests us in terms of the
effects on mitochondrialfunction that have an effect on
(55:46):
circadian rhythm, which havedownstream implications for
cellular and organism ageing.
Speaker 1 (55:54):
Yeah, thanks.
It's such an interesting topicand I really think that these
type of questions are the onesthat need to be funded and
answered, especially in this dayand age where we've electrified
and lit up the whole of ournighttime in a very evolutionary
and ancestrally inappropriateway.
I want to ask you what are someof your favourite or
(56:18):
interesting insights from theseries that you've done, most
recently with the amazing guestsand researchers?
I don't mean to ask you to pickfrom your favourite children,
but which are some of the mostenticing of these so-called
crazy ideas that are alluringfor you?
Speaker 2 (56:38):
well, I mean, one way
of uh about that question is
you perhaps ought to read thenew book that we've written, the
quantum biology book.
That sort of really coversthose.
What I what I see is a bringingtogether of of vastly different
disciplines in medicine.
That's one of the things thatyou've mentioned.
(56:59):
Our faculty has quantumphysicists, mathematicians,
astrophysicists, biologists,quantum biologists and more
conventional pharmacologists.
And you know, at one end we wethink about the work of mike
levine and how it seems thatit's not just genetics that are
(57:24):
controlling shape and functionand to certainly say it may not
be genetics at all.
In some instances he's workedwith planaria where he can
change the shape, the functionof planaria without altering
their genetics, where we cantake a part of a planaria, can
be taught certain behaviors.
You cut it in half, the headend grows a new tail.
(57:45):
We will know that the tail endgrows a new head.
But what happens is the tailend, when it grows a new head,
it exhibits the same behavior asit learned before it was cut in
half.
So we think about memory as canbe held somatically as opposed
to just in the brain.
So the notion of cells beingfar, far smarter in terms of
(58:08):
memory and in terms of thestimulus for shape and
production possibly coming fromthe energetic energetics
surrounding them, and I talk alittle bit about the quantum
fractal does, does the quantumengine of life in a way produce
a shape around which that thatorganism grows?
(58:28):
And each organism, uh has aunique um, each species has, has
unique atpas, for example, andwe have a study running with
mike and wayne levine at themoment, where we're looking to
transfect uh atp subunits andatp atpa subunits in a whole
from one species to another tosee whether we can transfer
(58:52):
shape and function withoutaltering genetics, so resetting
a balance between the role ofbioenergetics in producing form
and function.
And you know, it was said thatlife is just an electron looking
for a gradient.
So to understand thosegradients, how you drive those
(59:13):
gradients, and then the thoughtthat not only can these cells,
small numbers of cells, rememberprocesses which would lead to
behavior, and that might bepurely back to the point I made
about allocating energy todifferent neurons or different
neural processes, processes todetermine behavior, but of
(59:34):
course the work of Sartre andAigné, the very early lecture we
had, looking at the physorumand the slime mold being able to
effectively solve the travelingsalesman conundrum.
So the single cell,multi-nucleated, single cell
(59:56):
slime mold was actually able tosolve a problem that has
flummoxed most of the world'smost powerful computers.
Although it's not a quantumcomputer, it was behaving as a
quantum computer.
So now we see that cells notonly have an ability to remember
something and pass that on, butactually they can compute
(01:00:17):
problems themselves.
So computation may not onlyoccur in the brain and through
quantum wave theory, as you see,in the brain, as John Joe
McFadden will talk about, butthat may also occur in other
parts of the body.
You see that sort of in otheranimals, things like squid and
things like that, where quite alot of processing could go away
(01:00:40):
from the brain.
And then we think about talkingabout the work of Michael Sifrin
and the charge changes aroundscar tissue and around damage to
tissues.
You've probably seen the videoof Mike Levine's anthrobots,
(01:01:07):
which are just cells that havebecome like his xenobots and
that they, without any priorknowledge, will run up and down
a damaged tear in a neuron and,over a 72-hour period, repair it
.
And now what attracted them?
Was that just a sort of anormal physics-related response
(01:01:34):
to a change in charge across adamaged membrane and the natural
response of the anthropos wasto do something with it.
So we then think about what isthe origin of the bioelectric
templates which Mike talkedabout, what is the fundamental
(01:01:55):
generator that can generatethose bioelectric templates?
And in generating the energy,does it generate a shape form?
And I talk in the new book alittle bit about what I call a
quantum fractal in terms of, Iexpect it, like most people
understand, if you put some ironfilings on a piece of paper
with a magnet underneath, theiron filings will take up the
(01:02:17):
shape of the magnetic field.
Well, in quantum terms, isthere also a shape of these
quantum fields?
And is it around that thatbiology forms life?
(01:02:38):
Existed before biology,self-replicating,
non-dissipating or aself-replicating, dissipating
process which decreased ordidn't increase entropy as much
or as little as possible, andthat biology just made that
process portable.
And this also leads to thenotion that when we're looking
for life outside the earth, theproblem is, most people are
(01:02:58):
looking for biology, but it maynot be biology at all.
It may just be a very, veryspecific, uh, energetic entities
that are else able, able toself-replicate and carry
information with them and, asyou know, the physical data hold
(01:03:19):
back entropy.
You need energy and information, and that's why we think
mitochondria is so important inthat role.
So we then link that into intowhere is the seat of this energy
and information production, andall of those roads lead, in our
mind, back to mitochondria,although of course there is
(01:03:39):
still there's a very good notionnow of non-mitochondrial ATP
production, and so that'ssomething else that we have to
think about again.
So ATP production may be morewidespread than just purely in
mitochondria.
And then the next link is howwe then link those bioenergetic
(01:04:06):
processes to the myriad ofpharmacological or biological
systems that have been describedover the last 60 years.
That's what I'm trying to bringtogether, and from that come up
with new, obviously, diagnosticmodalities and new therapeutic
modalities, which I think willoccupy us for the next couple of
(01:04:26):
decades.
Speaker 1 (01:04:28):
Yes, absolutely
fascinating stuff, and I'd also
point the listener to your 2016review paper, the Quantum
Mitochondria and Optimal Health,and we touched on a couple of
the topics in that paper, but Ithink there was still a lot more
that we haven't, so I'mobviously mindful of your time,
(01:04:48):
professor, guy, so I reallyappreciate you coming on and
speaking with me.
Where can people learn moreabout the book that you're um,
that you've written, andobviously more about the guy
foundation and and the theseries that you do?
You operate?
Speaker 2 (01:05:03):
So the guy foundation
just Google the guy foundation
and you'll land on our homepage.
Uh, we have information thereof our symposium series, the
next one and the last ones, allof the symposia that we run.
We run two series per year, onein the spring, one in the
autumn.
I think there's about 55 videosnow.
(01:05:26):
They're about an hour each,with a range of scientists that
form our faculty globally.
Those are all on the website,but they're also on YouTube.
There's a YouTube channel andwe also on the site describe the
research that we funded, bothin the UK, us and elsewhere, and
(01:05:46):
we curate the researchprogramme to try and guide the
process forwards.
What we're trying to do with thefoundation not only is funding
research and leading it forwardand bringing people together
into a room being in a virtualroom, of course that would never
normally find themselves in thesame room.
(01:06:06):
That's the important thing isto try and overcome some of the
really interesting thoughts, andwe have quantum physicists and
physicists and on the whole,physicists do a lot of
hypothesis generating, butoccasional uh experiments.
(01:06:28):
I think it took what?
40 years for the higgs bosonexperiment to be to be
undertaken.
Um, but once they've done theexperiment, if the experimental
findings correlate with themodel, then it's given a tick
and they move on.
Life scientists are driven verymuch by experimentation.
(01:06:53):
I think it was Boyle who saidthat science is animated through
experimentation.
But you have to think about ita bit more.
And of course, the grants andthe ways life science is funded
is funded on the back ofexperimental proposals for
experiments, but probablythere's not enough hypothesis
generation thinking about it,life scientists.
(01:07:13):
So initially we had all thesedifferent scientists all with
the same notion that quantumbiology was of great interest,
but the physicists saying youhaven't thought about it enough
and the life scientists sayingwhere's your data?
yeah, and now what we'vehopefully managed to do is
create those stepping stones inbetween to be able to have a to
(01:07:34):
bring.
Bring these together's, I thinkis the fundamental role of it.
The book.
By the way, here's a revisedcopy of the book Quantum Biology
.
We wrote a book a couple ofyears ago about how we developed
cannabidiol CBD, which I thinka lot of people around the world
will know a lot about.
(01:07:55):
But when I first started withour cannabinool cbd which I
think a lot of people around theworld will know a lot about,
but when I first started withour cannabinoid program,
cannabidiol was described as aninert component of the cannabis
plant and so we wrote a bookabout it and there was a chapter
in that book.
That book was called aworthwhile medicine and we wrote
a chapter about quantum biology.
And this was a few years agoand the publisher said nobody
(01:08:16):
understood it.
Could you write a book that thelayperson could understand?
So we've written this book.
It publishes in England, Ithink next week, on the 28th of
November, and I actually sawit's on Amazon if people want it
.
So it's quantum biology by me.
Speaker 1 (01:08:35):
Fascinating.
Well, I'll definitely sharethat link out and I'll
definitely be grabbing myself acopy.
So I agree that you know thisis such a sorely needed area of
education and research and, likeyou, I share the thought that
this is how we help optimizehuman health.
So any final parting thoughtsthat you have for the listeners,
(01:08:58):
and maybe medical practitioners, doctors, medical students who
might be listening yes, I thinkthe important thing is to level
the playing pitch of knowledgeand understanding.
Speaker 2 (01:09:12):
When we thought about
the book, I found over the last
six or seven years it is much,much easier to explain quantum
biology to a layperson who baseswell, that's extremely
interesting or that'sfascinating, and this happens,
and this happens, whereas whenwe start with conventional,
traditional scientists, you cansometimes not get past the first
(01:09:33):
sentence before the scientistsays, oh, whoa, hold on, that's
not what I learned.
So what I would say is levelthe playing pitch, just sort of
set to one side the moreconventional thoughts about
biology, about physiology, aboutpharmacology, and understand a
little bit of what reallyabsolutely drives us.
(01:09:55):
And I often say to peopleimagine you and I had been
having this conversation and say, for example, this cup, for
example, had hovered there forthe whole conversation.
Okay, someone might say to youwell, what did, uh, what did dr
guy, what did professor guy sayto you?
And you say I have no idea.
I couldn't take my off this cup.
It was suspended there.
(01:10:16):
I had, I had no idea how it wassuspended there.
It absolutely was miraculous.
And I say to physicians, I saywhen you look at life, when you
look at humans.
It's as confusing, as complexand as miraculous as that cup
being suspended there.
And it's worthwhile spending abit of time if you're a
physician or a life scientistnot just to dig straight into
(01:10:38):
what's a disease and what's anillness and how we treat it,
because medicine is mainlypattern matching.
It's a lot of pattern matching.
But actually go back tofundamentals, say, well, if I'm
treating these organisms, thesehumans, it might be worthwhile
understanding what drives them,what actually makes a cell work,
actually makes a cell work.
(01:10:59):
And what we now know is itdoesn't stop just at chemicals,
it doesn't stop just at nervestraveling rather slowly up up
and signals traveling ratherslowly along nerves.
But the quantum biology orquantum science and quantum
mechanics has a fundamental rolein the fundamental workings of
us, allowing electrons to flowdown a gradient.
(01:11:21):
That's what life is all about.
And I say to these doctors,everything from the chin
downwards is orientated tofinding glucose for the brain,
because the brain needs glucose,we need oxygen, and that those
two are incorporated in inmaintaining uh, in maintaining
(01:11:42):
these gradients.
And so that's what I'd say toto doctors is sort of just open
your mind a little bit and thinkabout uh, and read this quantum
science and try and escape thenorms of of medical teachings of
the last 50 years or so, whichhas been extremely syllogistic.
There's a lot of syllogism,there's a lot of if this, then
(01:12:03):
that, and a lot of medicalthinking relies on the
modern-day doctor, consideringthat they understand the
mechanism of action and, frankly, if they don't understand the
mechanism of action, they can'tget involved with therapy.
I'm tend to be aphenomenologist.
I observe phenomena, I believethe patient.
Why doesn't somebody else andbegin to manipulate and use that
(01:12:26):
phenomena and see if we canalter it, see if we can emulate
it and and harness it fortherapeutic or diagnostic
purposes, uh, or prophylacticpurposes, and I so.
I think one has to set asidethat syllogism of intellect and
and just be rather moreaccepting that these rather odd
(01:12:49):
things, very weird things thatgo on in in in quantum physics,
just accept that, okay.
Okay, that's fine, let's acceptthat happens.
Now what is the impact of thisin human physiology and human
biology and to what extent canwe understand it, grapple with
it and manipulate it?
And that's obviously somethingthat we've published recently in
(01:13:13):
our space report.
Speaker 1 (01:13:16):
Yes, and we didn't
get into the space report.
We were, we, we.
Maybe that's a topic foranother episode and and I'll
point people to my previousepisode, I did a presentation
about the space report, but butreally, um, I I couldn't agree,
agree with you anymore and I Ireally think what, what you're
doing and the emerging field ofquantum biology is the core of
(01:13:38):
what is a decentralized movementin science and health, and that
decentralization is answeringquestions that centrally funded,
centrally administered, maybeeven gated institutions haven't
given us answers to thesecomplex questions.
So, and yeah, very muchappreciate your work and the
(01:13:58):
work of the foundation.
I'll be be uh, following,following it very closely um,
into the future.
So thank you very much, uh,professor, guy, uh, for for your
time.
It's been fascinating andintriguing.
Speaker 2 (01:14:11):
Thank you.