Obesity, Hormones & GLP-1s: Dr. Spencer Nadolsky on Ozempic, Wegovy, Mounjaro, and Women’s Health

In this episode of The Body Pod, we sit down with Dr. Spencer Nadolsky — The GLP-1 Doc to uncover the truth about GLP-1 medications like Ozempic, Wegovy, and Mounjaro, and what they really mean for women’s health, hormones, and sustainable weight loss.

We dig into the real science behind appetite, metabolism, and obesity, and why weight gain isn’t a failure of willpower but a matter of biology and hormones — especially for women in perimenopause and menopause. Dr. Nadolsky explains the myths around diet culture, Big Pharma, and compounded GLP-1 medications, plus how the medical system often overlooks women struggling with weight, fatigue, and hormonal changes. 

Whether you’re curious about GLP-1 drugs, navigating menopause weight gain, or frustrated by conflicting weight loss advice, this conversation will leave you informed, empowered, and hopeful about your health journey.

Episode Transcript

Speaker 1 (00:00):
Hi everyone. My name is Haley and this is Laura
and welcome to the body Pod. Welcome back to the
body Pod. Today we have the privilege of interviewing doctor
Spencer Nigdalski, also known as the Doc who Lifts. He

(00:20):
is an obesity and lipid specialist physician and believes lifestyle
is medicine. His mission is to help you cut through
the noise, ditch the gimmicks, and get the real science
back strategies that actually improve your health and your life.
He is also the founder of Join Vineyard, which is
an online virtual weight management and metabolic health program that

(00:43):
offers comprehensive care, including access to GLP one medications and
support from a dedicated team of doctors and dietitians. They
focus on personalized approach to weight loss, aiming for sustainable
results through a combination of medication, life style changes, and
ongoing support. Just to be clear with this episode today

(01:05):
at the Body Pod, we are no way promoting GLP ones,
but simply providing education so that you can make informed choices.
Join us for this great discussion with doctor Spencer Nadalski.
Doctor Spencer Nadalski, Welcome to the body Pod.

Speaker 2 (01:28):
Thanks for having me on we are, so we're.

Speaker 1 (01:31):
Super stoked because this conversation comes up all of the time.
So our listeners are all women over the age of
thirty five forty and pretty much it's all that I
work with in my app and just in the courses
that I run. So the GLP one doc here one
of the main ones or one of the main ones

(01:52):
that we follow. I'm super excited to dive into this
subject because I don't I'm sure you know, it's pretty
it can be pretty polarizing. Yeah, your instagrams and so this, uh,
there's some strong opinions about this. And you know, while

(02:14):
we're here and we we have you, I really want
to pick your brain and if there's anything female specific,
if it differs from you know, anything in the menopause
kind of space and that age group, or if it's
just the same for all men and women. I'm super
curious about that too, as far as treatment and how

(02:34):
you approach it. But let's start off with well, I
just have to ask you before we dive into that.
So you have the doc who lifts podcasts with your
brother and he's a physician as well. That's right, Okay,
So was your dad a physician because I saw your
post or did he take you to the teacher?

Speaker 2 (02:52):
He was a biology teacher, and my mom was an
elementary school teacher. My dad was our wrestling coach. He's
also a football coach as well. So we are really
big into athletics and academics when we were younger.

Speaker 1 (03:04):
Yeah, yeah, awesome. And then your little bro just like,
did you both know that you wanted to be physicians
or that just kind.

Speaker 2 (03:11):
Of no, just kind of happened. I wanted to be
an astronomer. And then when I went to the counselor
and freshman year in high school, they're like, they make
like thirty thousand dollars a year, and I was like,
I'm not going to be all that. My parents as
teachers make a little bit more than that. I can't
came out Yeah.

Speaker 1 (03:27):
Well awesome, So okay, I just had to get that
off my chest because I was dying to know. So
if we look at this world of GLP once and
I saw that you posted a few days ago like
that that some post about did diet? Did GLP once
kill diet and exercise? Let's just start there because it's

(03:50):
also you know, I hear about it and people with
the strong opinions will say, it's cheating. What's cheating?

Speaker 2 (03:58):
Yeah?

Speaker 1 (03:59):
Do you that way?

Speaker 2 (04:01):
Yeah? I mean, like you think about technology. Is it
cheating to use an iPhone instead of a rotary cone?
You know? Is it? Is it? You know, is it
cheating to you know, if you want to get into
the vaccine thing? Is it cheating to prevent a vaccine
preventable disease versus going it without one and getting that disease?

(04:22):
That type of thing. So, like we use technology and
we try to better ourselves and make our lives better
in multiple different ways. Is it cheating to use a car,
a nice brand new car or versus using a buggy,
you know, like with a horse? Is it? Is it
cheating to use a jet with nice amenities versus you know,

(04:47):
hiking across the country whatever, Oregon trail, getting typhoid, dying
in the river, whatever, So favor.

Speaker 1 (04:57):
Growing up there?

Speaker 2 (04:58):
Oh yeah, right, you don't be the banker. You'll be
the banker. You get all that money, but you're gonna die,
not like the poor farmer who figures it out anyway,
So it's not cheating, it's it's it's also if you
say it's cheating. We're moralizing weight. It's not like it's

(05:19):
some sort of competition, you know, where it's like you
try to keep it fair because then the competition then
if you cheat, you can actually cheat in a competition,
you get an unfair advantage that the other person didn't
have that isn't allowed. So performance enhancing drugs would be
cheating in the context of a competition. Are anabolic steroids cheating, Yeah,

(05:43):
in a competition when they're not a use, when they're
not supposed to be used, But in general sense, they're not. Really,
it's not cheating unless we're moralizing muscle building or something
like that. So I don't like it when people say
it's cheating. It's just completely negates the idea of this
idea of obesity as a chronic disease. Uh, it completely

(06:06):
ignores the biological aspects of obesity. It doesn't mean and
this is what people say, but it's a calorie deficit.
I'm like, yeah, yeah, it's a calorie deficit, but they're
biological drivers to push you away from a calorie deficit,
eating fewer calories and you burn. So no, it's not
it's not cheating. Yeah, that's ug enough myself the soapbox

(06:26):
for a second.

Speaker 1 (06:28):
No, I love that. And when you like, let's go
back to that first question of did the GLP one
killed diet?

Speaker 2 (06:35):
Oh yeah, I did it kill it? You know, so
like here's the thing it changed. It changed it because
here's I mean, you know, I worked at Weight Watchers.
They bought my first company, Sequence, which is kind of
a way to get access to these medicines. Uh, you
see Neom. You see all these other diet companies out there,

(06:59):
They're struggling. They're all struggling. And the reason is is
because people aren't just gonna sit here and white knuckle
and will power their way through diet and exercise anymore.
There is a small percentage of people that will be
successful doing that alone, but in general, people are kind
of fed up with having to kind of white knuckle

(07:21):
themselves and feel miserable while trying to lose weight and
keep it off. So it doesn't negate it though, because
even with the medicines, like, you still need nudges in
the direction of optimizing your nutrition. I mean, you don't
have to if you want to just take the medicine
and just eat fewer calories of whatever you're eating without
exercising technique, You can technically do that. I strongly recommend

(07:46):
not doing that for optimal purposes, but you could technically
do that. So if that's if we're just trying to
change the scale and just eat fewer calories and not
pay attention to the quality your composition of our diet
or physical activity. Yeah, if that's all at the window,
then yeah, sure, I guess it killed killed diet and

(08:08):
exercise because we're just not gonna we're not going to
look at those other things. But I think it more
changed it to where like, hey, now you don't have
to white knuckle and grin and bear it, and now
you can focus on the quality of your lifestyle habits
and as opposed to just like grin and bearring being
miserable while doing it. So like it changed it. So

(08:29):
there's no exercise in a pill or injection. They're trying
to make it. By the way, there's new injections. They're
combining them with these GP one medicines to basically block
one of the governors on our muscle growth called myostatin.
And these have been big. This idea concept has been
big since the nineties. Once I was in high school.

(08:51):
They're trying to figure out supplements to block myostatin, and
everybody talked about it. You can see these animals that
had their genetics modified to where it blocks their milestat
in gene and they're just huge, just jacked animals. Wendy
the whip It dog was just this jack dog. You

(09:12):
can look up and in Belgium blue as a type
of Uh, everybody's seen it. I mean you col probably
you've probably seen him. Some people would think that they're fake,
but they're they're real. You can modified and some humans
probably have some modifications. I always joke about my brother
having lacking his milestand because he's short and stocky and

(09:33):
just yoked. He works really hard though, but he's always
been pretty pretty pretty checked. But anyway, they're trying to
they're trying to develop this. It's not but right now
we don't have any replacement for exercise. And I always
tell people we need to brand exercise differently. My buddy's
an OBC doctor Yoni Friedhoff up in Canada. He kind

(09:56):
of said this, we need to rebrand exercise. Instead of
thinking about it as like a weight loss endeavor, we
got to think of it as a cardio respiratory fitness endeavor,
a body shaping composition endeavor as opposed to US pounds
on the scale endeavor. So they you can't get those

(10:17):
things from an injection or pill yet. I mean maybe
in the future, maybe maybe we'll see it someday and
nobody will have to do anything other than take all
a few different injections. It's very it's not completely out
of the realm, but.

Speaker 1 (10:30):
It's not terrifying. I feel like that's wally.

Speaker 2 (10:33):
It's scary. It's scary to think it's a good thought process.
Though it starts getting your mind going, like what like
what if we just injected and that's it. I don't know.
I enjoy the process of it.

Speaker 1 (10:45):
Yeah, yeah, So these drugs are not going away. Do
you foresee them going away at any point?

Speaker 2 (10:53):
No, So everybody's been waiting for the other shoe to drop,
because what it seems like is like these things will
do everything, including doing your dishes and taking your doing
your laundry and taking out the trash. Because like it's
it's like, okay, how can these drugs be so good
at everything? And everybody's waiting, whins the weird cancer going
to come out? Win's the weird x y Z side

(11:14):
effect coming out. There are a few, you know, you
see these case reports, you see some rare things of
like different causes of blindness that they're that they're trying
to look extremely rare things. Though, in large clinical trials
that are randomized against placebo, we see a lot of
benefit and these are in thousands of people. When you
start putting them on millions of people, you start seeing

(11:35):
these rare things like, oh, that's interesting, there are a
few more people that got x y Z because we
wouldn't have picked it up in the thousands of people
randomized trials. You start picking up when millions of people
are on them. But in general, benefits are just vastly superior,
higher than the risks. So these rugs I don't think

(11:58):
are going anywhere that The first one has developed FDA
approved in like two thousand and five, called Bayeta, So
we've had them for two decades now they are different.
Now they're bigger, stronger, bigger, faster, stronger than you want
I want to say that. Yeah, So that these things
are only going to get better. They're going to find
ways to hit the receptors in different ways that may

(12:18):
not cause that nausea that you get. So you know,
so for anybody listening, the GLP one it's a natural
hormone that comes from our own testines. The issue is
that we break it down within a minute or two.
Our own bodies have enzymes that break it down so quickly.
So researchers found ways to modify it. And now we
can modify it in ways that it's less longer in

(12:42):
our body and maybe hit the receptors that then have
downstream effects different than our own human natural GLP one.
So what that means is that maybe it maybe it
goes down a different stream once it hits that receptor,
it gets really complicated. But like essentially it's there. Like

(13:02):
you said, they're going to get smarter and better, and
then they're going to hit different words, They're going to
find all that they're finding all these different things that
hit different similar types of receptors that will then have
different downstream effects. And you can imagine the permutation se
multiple different types of combinations you could come up with.
And I I don't take the medicine, but it's possible

(13:23):
in the future. I'm taking one. People that don't have
much weight to lose one I know a lot of
people are doing it now, but I think what we're
going to see is all that research on it, and
I think in the future, might we might all be
taken one.

Speaker 1 (13:36):
I was just going to ask you that, do you
see us all on this like in the future, Well
do you?

Speaker 2 (13:43):
Yeah? Because Okay, So I was going to say, imagine
so like because people people get really upset by that
idea because they want what we should be just doing
diet exercise. I agree we should all be doing diet exercise,
but think about the reality is that. Let's imagine you
get two parents to struggle their weight, maybe it's genetic whatever,

(14:04):
and you see that the kid is they're eighteen twenty
years old and they don't have technically that obesity yet,
but you can see they're going on that trajectory zoop.
You put them on the medicine, you prevent it. All
of a sudden. You just prevented obesity and probably prevented
heart attacks and strokes and kidney disease and liver disease
and every other disease that's related to obesity. We don't

(14:27):
use them for prevention yet, but I could totally see that.
So that's that's kind of a world, I think, unless
another shoe drops and we notice, oh god, yeah, this
is another fen fen. But I don't. I would put
the farm on it really well.

Speaker 3 (14:42):
I feel like it would be very powerful for prevention
and people who are truly obese how it's changed their life.
I mean, I think the strug is incredible. I have
a question, do you think that. Do you have a
lot of clients that are at their ideal body weight

(15:02):
and they're taking it for other benefits? What other benefits
do you see people taking the drug for because I
know people who are microdosing for the other benefits.

Speaker 2 (15:18):
Yeah, So my practice mostly revolves around those with an
indication of obesity or type two diabetes the FDA indications.
Now having said that, those patients when like, I'll take
a patient with soriatic arthritis, for example, she's on multiple biologics,

(15:40):
different drugs that are out there for her storiatic arthritis.
And it wasn't until she started toure Zeppetite, which brand
name is zep bound or Manjaro Monjaro's type two diabetes
version but set bound for the weight management version, and
it wasn't until that where she noticed a large improvement
in the storiatic arthritis symptoms, you know, and when you

(16:04):
see these and all sorts of autoimmune inflammatory disorder that's
with the chrones, all sort of colitis, rheumatoid arthritis, all
those different things, I'm seeing the same thing. So I
think Big pharm is smart. They want to make but
they're making a ton of money right now. They want
to make more money, and they're starting to see it.
I know they are, because if I'm seeing it, they're

(16:26):
definitely getting reports of this, and they are studying these things,
and so what they're going to try to do is
find all sorts of indications. They want to put everybody
in the drug too, so because it's incentivizing to them
to make more money and put their drug benefits. So
I think that you are seeing Yeah, So I think
these anti inflammatory autoimmune types of improvements. We've already seen

(16:50):
the cardiovascular disease reduction event reductions in people with a
history of type two diabetes and cardiovascular disease, and that
is likely separate from the weight loss that occurs from it.
So it's a it's a weight It likely has some
weight dependent effects for the cardiovascar event reduction, but it's

(17:16):
a lot of it's independent, meaning you don't have to
lose weight, you'll just protect yourself from a heart attack
in the future from taking this medicine. So those types
of things I'm obviously addictive, like behaviors. Alcohol cessation is huge.
I get a lot of patients that love it for that.
They don't even want alcohol anymore, even thinks biting their nails,

(17:37):
all sorts of different things.

Speaker 3 (17:41):
So what about improved cognitive fund.

Speaker 2 (17:44):
Yeah, not so much. It's not as much. They some
people feel anti anxiety properties from it. But I believe
the anxiety improvement comes from a lack of worrying about
food all day. So people don't think about food all
day anymore. That food, it's what everybody talks about, is
that it's not a hunger craving issues. It's literally just

(18:05):
when am I going to eat? How much is going
to be? Is there going to be enough food for
me to eat? Then? What is it going to be?
It doesn't have to be anything. It could be a
healthy food, like a not junk food or anything like that.
It could be just any food. So I believe a
lot of the anxiety improvement comes from that. All quieting
down whether it's a true anxiolytic anxiety reducing effect beyond that,

(18:31):
I don't know, but that's we see a lot of that.
I do caution though, high doses sometimes we see a
dampening of the mood and motivation. So those cognitive effects
can actually it can put not like thinking straight, but
like motivation wise can be reduced when you get to

(18:52):
high doses of tures appetite and some aglutide. I see that.

Speaker 1 (18:57):
Okay, so I'm going to take a detour really quick.

Speaker 2 (19:00):
Yeah, detour away.

Speaker 1 (19:03):
And I know this is this is a very this
could come across as very political, but the perfect well
let's go, I know what what is like? Is it
Is it terrifying that big pharma would have this much control? Yeah,

(19:24):
from the big pharma that we know in the United
States today.

Speaker 2 (19:28):
Yeah, I think I think that's that's everybody's concern. That's
why they're like, man, you're a big pharma shill. I'm like, yeah, Like,
I don't know what to tell you. It's and people
are like, oh, they must be in cahoots with big
food because in the big food and then you bring
in the big farm and then they're all making tons
they are making tons of money. It is, it is.

(19:50):
It would be concerning that they have that much power,
and that's that is the issue, because we want them
to be incentivized to come up with these new therapies.
But then all of a sudden, it's like you're charging
what for these life saving therapies? Like, come on, you
guys would be just ridiculously rich. If you lowered the
price tenfold, they could make some I mean it was

(20:12):
twelve hundred dollars to go to the pharmacy. Now they
have this Lily Direct thing. Eli Lilly and Novo Nordice
did this too, the two big ones. But twelve hundred
dollars at the pharmacy. The net cost is something like
six hundred dollars if you don't have the PBM. So
then they do this thing called Lily Direct and you
can get it for three hundred and forty nine dollars
for the lowest dose and then four hundred and ninety

(20:33):
nine dollars a month for the other doses. So it's
cheaper than twelve hundred dollars.

Speaker 1 (20:38):
But that's not cheap, No, that's still a lot of
money for.

Speaker 2 (20:41):
Its still out of reach for most people. So you know,
they're they're how much it costs them to manufacture. There
are reports out there it might be like five to
ten dollars a month for them at the most. Of course, Yeah,
so you can imagine they could go, all right, why
don't we just they can make it ninety nine dollars
And that's what you see at other countries, ninety nine

(21:01):
bucks for a month, and here we are suckingd wind
in the United States. So that is a concern. What
I would hope though, is capitalism kicks in, we get
these other I mean, there are tons of these things
on the horizon. What's funny though, is that you see
Lily and Novote they're trying to jump in and buy

(21:22):
some of these places out. You kind of see it.
But I believe what we're going to see is a
few other players jump in. They're going to undercut them,
and then everybody's going to have to start lowering their prices.
That's what I think. I have to believe that that's
how that's how it works. They don't have a monopoly.
There's other companies come into play soon because now everybody's like, oh,

(21:45):
these things work, let's develop our own.

Speaker 1 (21:47):
Yeah. Well, if somebody were to come, okay, somebody has
has weight to lose, they're ready to go on a
GLP one. They can't afford that, can they trust? I mean,
I'm sure that they're There's a lot of scammers out
there too that are selling products that are GLP one
but compounded in a way that maybe it's not even

(22:09):
the real thing. I mean, so that's a huge issue
as well.

Speaker 2 (22:12):
Yeah, I don't trust any of those people. I think
they're all a bunch of scammers. And like, here's the thing.
They're to say that all compounded versions are terrible would
be a lie as well. I've I know of a few.
There are different types of compounding pharmacies too. You can
get mom and pop compounding pharmacy. There's five O three A,

(22:33):
and then there's these five oh three bs that can
do mass marketing. And some of these five O three
b's are even trying to develop their own generic version
for future submission to the FDA. So like I would
trust those The problem is they were only available during
the shortages that made them It made it legal for
them to copy the patentent drug. So now the shortages

(22:56):
are over, it's not now the five O three A
pharmacies who aren't supposed to be doing mass production of
the medicines. Uh, they're able to make uh customized versions
of them. So if they change the dosing. So instead
of let's say we'll take some maglutide, for example, with
we go V, you have to go from point to

(23:19):
five milligrams and the point five milligrams and you go
to one milligram, then you get to one point seven
and two point four. So let's say that you don't
tolerate the jump from a point five milligrams to the
one milligram, So they're going to do a point seventy
five milligram custom dose for you. And that's technically allowed,
And honestly, I think it's smart. If Novo Nordisk were smart,

(23:40):
they would make their they would make it to where
which you can do this in other countries on their
little pen. In fact, I got my little sample pen
here because I'm a big pharmashill allow they allow you
to adjust the in between doses and you can actually
do it the no zepic pen. This is a sample
we govy pen, but in other countries you can other countries.

Speaker 1 (24:06):
Yeah, yeah, it's everywhere now, right.

Speaker 2 (24:09):
It's everywhere, everywhere you look. So that's the thing. So
these compounding pharmacies, I don't. I don't trust like I
always take. Would I send my family? Would I give
it to my family member? No? Would I give it
to myself? I wouldn't do it?

Speaker 1 (24:23):
Yeah, I know, how does the consumer know?

Speaker 2 (24:26):
You wouldn't? You wouldn't. You'd have to go out and
test every batch yourself, or you're just gonna You're gonna
trust that that place you're getting from is fine, and
you see, you'll see a lot of influence. I'm sure
there's people listening to this that are definitely taken compounded
and they're like that doctor and Adolski's definitely getting paid
by big Farmer to say this. I I'm just telling
you what I would personally do. If you want to
take your risk. There are people getting it on the
gray market. I'm sure there's people listening to this who

(24:49):
get it from research peptide companies online where I always joke.
So I'm in some of these doctor obesity doctor groups
on Facebook, and I always talk about bathtub terms churs epetite,
So I make a joke about bathtub chursepetite dot com
and where you can get your churs appetite, because like

(25:12):
because you can go out there and it's you get
research grade peptides. Now, some people have tested and they're
they're extremely pure at some of these places. But I'm
not going to take that chance. If you inject something
that not that it isn't pure and isn't done correctly,
you can die. So I'm not messing around. But people
do that out there, so I would urge not to

(25:34):
do that. I do understand the cost is an issue
because then what they're they're going to say is like,
look at you, you rich doctor. Of course you can
afford it. And I get it, Like I understand, I
get it, So I understand why people are desperate and
wanting to get a more affordable option. So I don't
I don't blame them. I would just say, like, just
know there's a risk.

Speaker 1 (25:56):
Well, and maybe some of those those risks that people
are like when's this sh you going to drop? Like
of all these other issues and diseases are something maybe
that comes from the compound that you have no idea
if it's.

Speaker 2 (26:08):
There, that's there, and that's being there. They're rumblings around
like what like some of the reporting being done at
the FDA about like what I've seen some weird stuff,
but you never know that could also you could also
have some big Pharma propaganda trying to make it look
like some of these places are bad. I get it.
There's corruption everywhere. So I'm not going to say that

(26:28):
Big Pharma is not corrupt.

Speaker 3 (26:31):
So you recommend people doing just the name brund is
what you're saying, Like just from.

Speaker 2 (26:36):
The would that would be my advice, the safest that
would be my advice. I don't get paid, not yet
by big Pharma. I am going to be on an
advisory board here soon. I I never took Big pharm
of money. But I asked my patients and followers, I'm like,
what would you think. They're like, well, your haters will
just say, see, you're a big Pharma shill, but we
want you to go and try to voice your opinion

(26:58):
to try to try to get them the lower So
I said, okay, what if I what if I like
took that money and donated it. So I was like,
all right, I'm running some studies. So basically what I'm
going to do is take that money and if people
are going through one of my studies and runs out
of insurance coverage for the medicine. I will then get
them the medicine with that money. So that's what I'm

(27:21):
going to do.

Speaker 1 (27:22):
That's cool. I love that, all right, So I want
to shift the conversation. There's nine hundred tabs open in
my brain of where I want to go with this.
But I really liked the post that you just did
with the physician about MHT and how that plays into

(27:44):
GLP ones and if taking MHT helps with I don't know,
more significant weight loss because this is this is all
the depot. The conversation in our world is do you
take MHT do you not? And then there's that whole
pendulum of women that have gone through this, But now

(28:05):
we're adding a GLP one? What do you think?

Speaker 2 (28:07):
Yeah? So, I mean the issue is the Women's Health
Initiative that came out. It really pushed like people not
to prescribe it at all. That's that was wrong. And
now but we're seeing menopause influencers and I just there
shouldn't be like that. Shouldn't there shouldn't be a thing.
We should just be like evidence based doctors promoting indicated

(28:29):
therapies and going against false information. So women who are
struggling and I will never go through menopause. That's the
elephant in the room. They're like, you don't understand. You're like, well,
I do understand because I have a ton of patients.
I have hundreds of women on the stuff, and they've
been ignored by their other doctors because they were too

(28:50):
scared to do it. It's like, no, it's clearly an
indication to use menopausal hormonal therapy to alleviate all the
symptoms that occur during menopause. But I do have a
lot of women that never had symptoms and they feel
great that went through menopause whatever. Lucky them, you know.
So then what you see now is that like, in
order to promote it even more, even without menopausals, phasomotor

(29:13):
symptoms or anything else like that, they're feeling great. It's
kind of this big push to like take menopausal hormonal
therapy to help you lose weight. And it's like, m
I don't it's never been studied like that. There was
a retrospective, what we call retrospective, so they looked back
at the patient charts and it looked like last year,

(29:34):
those who took somemaglutide by itself. People say semi glue tide.
It's actually semaglutide. Nobody pronounces it correctly.

Speaker 1 (29:42):
Now that you're saying it that way, real Campbell says
that that way, I'm like I was saying, get wrong guarantee.

Speaker 2 (29:49):
People are going to say, why should we listen to
he's not even pronouncing it right. I'd be like, well,
screw you. Uh, I pronounced it correctly. I talked to
Nova Nordak. This is how this is how it's pronounced.

Speaker 1 (29:59):
I think way better though, blue tide.

Speaker 2 (30:02):
Some magnetide anyway. So they look back at the charts,
people are like, why this guy is a real asshole
some magnetide. They looked at who took some magnetide by itself,
and then they looked at oh, look look at the
people that took menopausal hormonal therapy with some magnetide. It
looked like the people that took both lost a lot

(30:23):
more weight. Now, the thing is, when you look at
a retrospective design, they're looking back at just kind of
what happened, as opposed to what we'd like to do
as a prospect of a design and randomized placebo trial
the reason retrospective designs are bad. You can get all
these different what's called biases put in, like why did

(30:44):
that person get prescribed the menopausal hormonal therapy though, and
what what makes them different compared to the people that
didn't get prescribed it. There's all sorts of different things.
And so that's why you got to do a placebo
blinded trial, because then people on a placebo may do
just as well. In the study, though it was tiny,

(31:05):
it was a small, tiny, tiny, little there were only
sixteen people who took the menopausal hormonal therapy with the samagnetide.
And so the doctor we had on our podcast, she
did an analysis of the surmount trial and that's the
one looking at chru zeppetide versus a placebo. And what
they did was they looked at the various reproductive phases

(31:27):
throughout throughout the women's life, and it looked like no
matter what, there wasn't a major difference in how well
or how much weight people lost percentage wise throughout the
different reproductive phases. So premenopausal, perimenopausal, postmenopausal, and they tried
to look at differences. It's hard to look at it

(31:47):
like who was on the menopausal hormonal therapy versus who not,
who wasn't birth control and all these different things, but
it looked like no matter which way you spliced and
diced it, there wasn't much of a difference. The thought though,
is that maybe estrogen does have this leptin sensitizing maybe effect.
So it's because there was maybe a signal in some

(32:09):
other studies that postmenopausal women do worse than pre menopausal
and we all know, well, I don't know, maybe people
don't know, actually women do better than men on these drugs.
It's I always make a joke like you know how
the meme is where man I I cut out, I
only eat basically carrots and lettuce and some chicken all day.

(32:29):
My husband cut out looking at French fries and he
lost ten pounds and I always I only lost one.
You know, have you seen that joke? I mean, everybody
talk about how their husband can just do whatever and
lose twenty pounds. Of the women's just it's finally the
first time it's like, wow, women are they in every
aspect there beating men with weight loss with these drugs.

(32:50):
And the newer drug were tatritide. Everybody calls it red
a true tide attritize right if it's I and unless
they change the way it pronounced, because if it follows
the same trures epetide, it's some aglutide, liraglutide anyway, that
one too. It looks like similar women do better. So

(33:12):
the thought is maybe there's something about the estrogen that
is sensitizing. No, I'm not going to sit here and
say that, Nope, there's no effect. Stop being silly. It's
very possible. There's a fact. I always just urge caution
in saying like, look, I wouldn't prescribe menopausal hormonal therapy
for the very for the only indication of weight loss.

(33:36):
Only what I would say, though, if a woman is
coming in and clearly has the perimenopausal and menopausal symptoms,
unless there's a weird contry, there's some sort of contraindication
that is going on, you prescribe them the hormonal the
menopausal hormonal therapy regardless. So like, here's what I wouldn't do, though,

(33:59):
if if a woman comes in and she's postmenopausal, she
stopped having a period a year ago, she's fifty one
and wants to go on trus epetitis semagnetide. I wouldn't say,
all right, here's your turrus epetite and semaglutide. You don't
have any symptoms of menopause, the vasomotor symptoms, nothing, zero,
they're feeling great. I wouldn't go, here's also your menopausal

(34:22):
hormono therapy to augment it, because there's we basically have
very small, retrospective, non randomized, placebo controlled data on it.
If they plateaued. Let's say, okay, let's say they're fifty one,
they've been on some magnetide trus epetite and they got
you know, ten to fifteen percent weight loss. We want

(34:43):
them to get twenty. For whatever reason, I wouldn't also
put them on menopausal hormonotherapy unless they had those symptoms,
because I don't believe that the data is not strong enough.
It's possible, though, if they do a randomized trial and
the way that I was set up, I'd be I'm
going to be really interested. Someone's probably I've told somebody's
trying to do this trial. I'm going to be interested

(35:04):
in the design because what I wouldn't set it up
with you imagine you have two groups of postmenopausal women.
Who are both having vasomotor symptoms. Nice, whit's ear to
build all the different things, all the terrible things that
are going on. You put them both on whatever, pick
your poison, turzepetide, and one of them has a turuzepetide

(35:27):
plus a placebo, and the other one has a terzepetide
plus menopausal hormonotherapy. I wouldn't run it like that that
other person that you're confounded, because the people that get
the placebo, they're not going to have their vasomotor symptoms improved.
So the only way I would do this trial is
if you'd have to recruit postmenopausal therapy women or postmenopausal

(35:49):
women who don't have any symptoms of menopause, no, no vasomotors, nothing.
I think that, and then you got to then you
can do a placebo blinded turs epi tide plus menopausal
hormonal therapy versus trzepetide anaplacy boat. That's the way that
I would run it. I've thought about this a lot
over the past few months because I actually think it

(36:10):
would be a really cool study because maybe there's an effect.

Speaker 1 (36:14):
I think it's probably a couple of years out until
we have a ton of research about them.

Speaker 2 (36:19):
Yeah, yeah, because then because the other thing that what
people will say is that menopausal hormonal therapy and there's
some people saying this it's it can prevent heart attacks
and heart disease. Well, there's a suggestion that that could happen.
We just can't say that definitively the way that the
studies have been so I think, you know, if they
want to start making that claim, they get it, you're

(36:39):
gonna have to have somebody pay for it. Because is
big Pharma gonna pony up the money to pay for
that huge trial when you can get these drugs generically,
I don't know. I don't think that that's the issue.
Someone's got to pay for it too. I do think
it should be done though, because it would be really
cool to see, well maybe maybe we go back and
the pendulum does swing back and everybody should go on

(37:01):
metapausal hormonotherapy. But like, we just can't make those claims
right now.

Speaker 1 (37:06):
I think that it's beautiful though, that it gives women
because I'm in a different space. Obviously I'm not a physician,
but women will come to me and they're just they're
at their width end. Like there's all of these changes happening,
and they're just like help somebody, help me, just help me.
And if you're in the camp that you don't want

(37:26):
to take MHT or you're in the camp that you do,
it's so refreshing for the women to be like, oh,
I don't have to do this if I don't want
to go on this, but I do want to take
a GLP one great that works for me if I
If I do want to take it, and you know
whether it changes anything or not, which we don't. It
sounds like we don't know at this point. Then they

(37:50):
there's options, there's not just like one way, which is
usually where the confusion comes when we're streamlining one way
only to anything.

Speaker 2 (38:00):
Yeah, a good physician should be able to have these
risk benefit discussions with the patient. The system's kind of
broken though. That's why I left the system. I do
like a it's I don't call it concierge. It's a
direct care model. It's just one hundred and fifty dollars
a month. I try to you know, with a dietitian
and a strength coach and all the different things. But

(38:22):
like a regular model, you get like five to ten
minutes with your doctor. You see them every once a
year you try to get in. It's every awaiting you know,
weight period of three to six months type of thing.
So it's really terrible system because women are like I
get a ton of women that are like, man, I swear,
I'm not going crazy. The doctor wanted to put me

(38:42):
on antidepressants, but it's because they didn't have enough time
to explain their symptoms. And it was metopause. It was
literally metopause. It's like, no, So that's that's the system's broken.
I don't I don't know how to fix it. Outher
then I just had to leave. So I was like,
all right, whatever.

Speaker 1 (38:59):
But how long have you? And it's called Vineyard, right.

Speaker 2 (39:02):
Yeah, so this one. So I left the system in
twenty sixteen. It was with a few startups Ahead Sequence,
which was in bought by weight Watchers. That's more of
like a again, you just it's more of just access
to the medicine versus now I'm like, no, I want
a full spectrum cardio metabolic health program where you can

(39:23):
spend time with patients. They get your own dietitian. We
have a strength training program. Everything that the person would
ever need is in this program. Now like, if you
have your good PCP, you don't really need them for
much and you see him once or twice a year
and it's just like a refills. You may not find
as much value out of my program. But if it's
like not, I want to. I know when I message in,

(39:45):
I want to get a response within a day, and
I know it's personalized and if I need a next
day appointment or jumping on the call with a doctor,
that would be like my program. So I started that
in January.

Speaker 1 (39:57):
Just this year.

Speaker 2 (39:59):
Yeah, it's going really well. People really like it. Again,
if if you don't value having direct access to a
doctor and a dietitian and all that stuff and you
don't really need much, some people don't find as much value.
But if you're like, no, I want to have someone
there when I'm having side effects, I want my zofrans
sent in that day. I want to I want like,
oh I'm having hair loss. We can do some you know,

(40:22):
your various testing, and we can put you on monoxid
to all these different things to basically personalize your care.

Speaker 1 (40:30):
Wow. Okay, so let's go back to the obesity, because
is this like this sounds like the population that you've
spent a large amount of your time working with is
there like the obesity gene FTO, and I think there's one,
what's the other one m M something MC four.

Speaker 2 (40:49):
There's a bunch of them, So FTO is a small
like is related. There's polygenic obesity where you have multiple
genes kind of pushing you in one way. And then
there's that's what most people have. Then there's monogenic where
you have a mutation in your leptin or your MC
four POMC, or you have a few other there's a

(41:10):
couple other syndromes that are genetic. Those are the things
where it's like the GLP one might actually help a
little bit, but you may need some other other therapies
like what what would be other? Yeah, there's one called
set melanotide in sivery. It's only approved for these what's
called monogenic obesity issues. So, and they're upstream in the brain,

(41:34):
these these upper order neurons that are related to satiety
and energy metabolism. So when if you have like a
severe mutation or deletion in one of those, like you,
that's where they're young, Like you see these two hundred
pounds young, very young kids and they're just voracious appetite.
It's likely they have that that's where the parents were getting,

(41:58):
you know, accuses of child abuse. And it's only been
in the past, you know, a couple of decades where
it's like and still even people don't understand it very well.
But they're like, oh, yeah, that was genetics. Sorry, so
hopefully you're not in jail, you know. Yeah, so those
are monogenetic, but like you see, most people have just

(42:19):
small little variations in their genes that kind of push
them in another direction. And people this is the argument
people like because if you look at the studies, actually
you can prevent obesity, and people that have not the
monogenic but polygenic forms of obesity, you can prevent the
weight gain in the first place. And you're like, it's

(42:42):
not a destiny. If you have a monogenic you're you're
pretty much destined. It's it's almost impossible without treatment to
prevent and treat that obesity. But if you have the polygenic,
you know, it's not destiny.

Speaker 1 (42:57):
Okay, So then this is the million dollar quiestquestion that
people are going.

Speaker 2 (43:01):
To ask really and probably.

Speaker 1 (43:04):
For so for for people women, not women, but just
people in general out there that are that have the
there they are in the camp. You're eating too much
and you're not moving enough, and this is how you've
become obese.

Speaker 2 (43:20):
Yeah, okay, so this is this is why I say,
I'm like, sure, it's an energy balanced thing. Even the
people with monogenetic obesity, it's an energy balanced thing. So
it's it's what's causing the energy imbalanced though. So the
people with let's say we'll go back to the monogenic
just because it's severe, it's like, okay, they have a
gene that is giving them a voracious appetite, like they

(43:44):
have to lock the cabinets sometimes when they're younger, they
just I mean, I see it. Brothers and sisters, fraternal twins,
young kids. You'll see one who's thinner, who will stop
at a birthday party, who will stop at like a
half of a cupcake, and then the other twin who
looks it's like a little bit heavier, eats two cupcakes,

(44:07):
maybe three, same same upbringing, same everything else. So clearly
there's something going on. So you have certain genes that
might push you in a direction in terms of appetite,
and that is causing them to eat more. In general,
the genetics of obesity are they're in the central nervous system.

(44:27):
They're related to appetite. People like to moralize appetites that
makes them not feel good. They want it to be
their metabolism being lower. But in general it's appetite related.
But the other thing is, you can have slower metabolism.
Some people have genetics that may and of course nurture
comes into this as well. But let's say that nurture

(44:48):
doesn't you may have genetics that make you not maybe
enjoy physical activity as much. So there's you know, there's
little variations there, but a lot of it's appetite related. Now,
the other thing that comes into play, why do some
people store fat in certain areas versus other people? Unfortunately,
some people store fat more around their abdomen versus some

(45:09):
people store fat and their button thighs. Actually, storing fat
in your button thighs is actually really healthy versus storing
their abdomen. And people have heard that the apple versus
the pair shape. But that's genetically that's genetically determined. So eating,
you know, eating too much, moving too little? Yeah, sure,

(45:33):
but what's the what caused that in the first place?
And then you know someone that stores their fat in
a certain way, like it's going to be hard to
modify that, and that's we all have different kind of
body shapes and so. And then there's other people. There's
some people that walk around in life that don't try
at all. They're not trying to. We all see them.

(45:56):
They like shit, they don't care about their exercise. They
don't care at all, and yet they look great and
they don't care. And so it's like, well that's not
really fair. And people are like, well they have fast metabolisms.
Well no, if you watch them, they eat some crappy food,
but that's like all they eat all day, and it's

(46:18):
a smaller amount than usual. There can be changes in
the differences in the metabolism. Actually, there's some interesting genetics
of how much they'll absorb. They may actually poop out
more calories, just all sorts of stuff. Yeah, where it's like, oh, man,
what the heck. And other people are just like they
look at French frise and they gain five pounds in
their app theman it's like thinks a lot.

Speaker 1 (46:40):
I had a friend like that, seriously, the second in
high school. She was done eating anything. She was like,
well gotta go, And I was like, how many times
had they been going but super thin and just like
never dieted, never had to worry about what she ate.
So is that where appetite dis regulation? Is that what

(47:01):
you're meaning? Yeah, what do you mean by that? And
can you explain that because most listeners won't know.

Speaker 2 (47:05):
Yeah, So think think about someone that gets done with
their meal, normal sized meal and feels good, doesn't want
anything afterwards. Another person they're eating enough for what their
activity and body size is, and they want more, and
they want more and they want they want high calorie dessert,

(47:27):
whatever types of foods afterwards, when it's like no, this
person like they should they should be done. The appetite
disregulation it's and it's we have our environment. These foods
are designed in a way that make us want to
eat more, and just trying to figure out exactly what

(47:48):
about them makes us eat more, but it doesn't matter.
We eat more of them, you know, think about I
always talk about like the the think about tortilla chips
that elson they're adding a hint of line. They're doing
all sorts of things that you get, you got the savor,
you got sweet, you got crunchy, salty, fat, and just
in perfect combinations to make it, you know, want more.
I love that stuff too versus, so we got the

(48:09):
environment working against us. Once we gain that weight, though,
there can be inflammatory changes, and they've kind of seen
this with like functional MRIs and PET scans of the
brain to kind of see how it fires and dopamine
signals and all these different things. It seems to be
those with obesity have more of the develop what looks

(48:30):
like dysfunctioned or dysregulation. And we can see it when
people like they lose one hundred pounds or whatever, and
their appetite just ramps up where it's like, Okay, they
lost one hundred pounds. Now they're at a weight that
should be healthy for their height, let's say, and their
appetite makes them want to eat so much more to

(48:51):
where they regain a lot of that weight back, if
not more. So that's that's kind of the appetite dysregulation.
That's why these medicines work so well. They come and
hit the receptors and basic shut off that high appetite level.
So yeah, that's the gist.

Speaker 4 (49:09):
Well, what did you do before you before the GLP ones,
because you were a physical practicing physician before that, did
you have people come in that really weren't eating a
ton and still just could not lose weight, that were
at an obese weight and they were not moving the
needle with diet and exercise.

Speaker 2 (49:31):
Yeah, all the time. We have older drugs. There's fentermine,
if you've heard of that one. It's a nor edge. Yeah,
so fenturmine was the component of fen fen that didn't
cause heart valve issues. It was the fen fluramine component
that they're like, ah, this thing. It was a serratinergic
drug and it hit a receptor on the valves. It
doesn't really matter. But fentermine by itself seemed to be safe.

(49:54):
But it's an amphetamine, like it's got simp what it
called sympathomimetic properties, kind of like an that helped a
lot of people, but like you know, the upper component
gave a lot of side effects. We had a drug
called contrave. They combined buproprin, which is a dopamine nouropinephrine
reuptake inhibitor. It helps with depression and smoking cessation. They

(50:16):
combine it with now treksone, which is used for an
opioid blocker used for alcohol cessation, and they work synergistically
in the brain. To help with appetite. It doesn't work
that well, but for some did okay. So you know,
we had some of these drugs that were okay, and
then we had bioatric surgery. So it was it was frustrating.
People come in and like, I'm trying really hard, I

(50:38):
can't lose the weight. Now, the thing is, everybody will
say that they're eating twelve hundred calories. But if they're
three hundred pounds and they see they're eating twelve hundred calories,
it's not possible. It's literally impossible. But you see, I
hear it all the time, and I can't just sit
there and say you're a liar, because that's not nice. Yeah, yeah,
it's just and it's not helpful. So what I usually say, yeah,

(51:00):
I bet you're trying really hard, because I guarantee they
are training hard. They have a very what's called a
high perceived level of effort, meaning they feel like they're
eating twelve hundred calories. It's not possible that they're eating
actually twelve hundred calories. They're eating a lot more than that.
But so that's why these drugs, they basically make it
to where that perceived level of effort just lowers so

(51:22):
much to where then all of a sudden they are
able to eat that. You know, however many calories. It's
not necessarily twelve hundred, but it could be fifteen hundred two. Thought,
whatever it is, it is that the reason I love
these drugs so much is just finally the patients feel
like they have hope, They feel good, they can finally
do the things that they always wanted to do and

(51:43):
just couldn't and felt like they were gas lit by
doctors all the time.

Speaker 1 (51:46):
Well, I mean, and again just my own little world here.
When I have somebody come into a course that's just
you know whatever, and I figure out through the numbers
that they're obese, I don't want it to be one
extra step of them not getting results, Like there's this
huge psychological component as well, coming in and having one

(52:12):
more failure from not being able to do it. But
then these you know, now, in the last couple of
courses that I have, there's been a handful or more
of women that are on a GLP one and they're like,
for the first time, I don't have food noise running
through my head, and now I'm motivated to go to
the gym and motivated to eat better. Like it kind

(52:35):
of comes full circle. Do you see that?

Speaker 2 (52:37):
Yeah, that's exactly that they finally feel like they can
do all those things.

Speaker 3 (52:41):
That's why I feel like it's not cheating to do it.
I think it's a tool in your tool products and
if anything, it motivates people to work harder and eat better.

Speaker 2 (52:51):
Yeah, it takes all that. It helps the mental space
that they had that was committed towards just worrying about
food and everything to nows and they can focus and
get things done.

Speaker 1 (53:04):
So you would put if you had a family member
that was struggling significantly with weight, you would have no
problem putting that.

Speaker 2 (53:13):
Yeah, I have some of them. I try to put
them on if they don't listen to me, but I try.

Speaker 1 (53:18):
If you give it to him for a Christmas present.

Speaker 2 (53:20):
Shoot them up when they're sleeping. No, I'm kidding obviously,
but of course I would only do consent. But no
I did. That's a but yeah, yeah, yeah, no, I.

Speaker 1 (53:30):
Are they all the same?

Speaker 2 (53:32):
Are all of them? The medicine? No, some magnetide is
a GLP one receptor agonist, only to Resteputite is a
GLP one which is gone like peptide. Slash g IP
GluN uh or glucose uh in glucose dependent insulinotropic polypeptide.

(53:53):
It's a mouthful and every time I always have to
think about it. But they hit two different receptors. There's
gonna be more that hit different receptors. It's going to
be quite so.

Speaker 3 (54:04):
Do you feel like people need to try different ones
to see what works best for them?

Speaker 2 (54:10):
Generally, put people to churs eppetite first because it's better tolerated,
works it's stronger, but once in a while they don't
tolerate that one. I'll try that we go VI or
some magnetide, but a lot of people that are on
some magnetide didn't tolerate it, and they actually do better
on the terz epetide, So that's my go to in
the future. It's going to be interesting because I think
we're going to see with the different receptors we're gonna

(54:34):
it's it will it'll be more of a of a
game of figuring out what people tolerate best.

Speaker 1 (54:39):
Interesting if we're wrapping this up and really there's sounds
like there's a few a small percentage of people that
it might not be a good option for and probably
for the uh, the side effects that they're experiencing. So
there's no contraindications for someone with like a liver, I

(55:01):
don't know anything like that.

Speaker 2 (55:02):
No, the only contriunications can't be pregnant. I mean maybe
in the future just put everybody including the baby on it,
but not right now. We don't want to do that.
Contriunications medullary thyroid cancer. And the reason is is because
they saw it in rats. Rats have different receptors on
their thyroid C cells. Humans don't have them or don't

(55:22):
readily have them, but they saw the signal in rats,
so we don't put it on any with a specific
It's a rare type of thyroid cancer, medullary thyroid cancer,
or something called multiple endocrine neoplasia type two. And the
reason is that because it also arises medullary thyroid cancer.
Those are like the big contridications reasons you can't take it.

(55:46):
People always like, well I had pancreatitis before, Well, if
it was just from your gallbladder, you can get gullstone
pancreatitis or alcohol or triglyceride induced pancreatitis, not a contraindication.
Just some of those things are caution So also breastfeeding,
it's not known. A little bit might get in the milk,
but if they're if the baby's getting it, orally, it's

(56:08):
probably getting broken down. So actually people think that it's
probably safe, but on the label of the medicine it's
like caution. I tend to be more risk averse, so
I don't do it. But it's I think in the
future we're probably going to be like, it's probably fine,
but I don't personally just yet. I don't want to
get sued.

Speaker 1 (56:29):
Nobody wants to get sued. Okay, So if someone is
coming to you because concierge care, there's a lot of
physicians now starting to do concierge care for good reason. Yeah,
a good reason for everything you say. But I mean
that can range up to eight hundred to one thousand
dollars a telehealth visit.

Speaker 2 (56:50):
Yeah some people. Some people charge thirty five hundred and
a couple of you know, fifteen hundred dollars or whatever
for follow ups for no So I want I want
mine to be subscription because I want long lasting relationships
with patients, like instead of just like, all right, I'm
gonna make a lot of money. Now it's I'm thinking
about this as like doctors that care about medicine go

(57:11):
into it to develop relationships with patients, and you want
to get it. Makes it my job easier. Once I
get to know somebody, I'm like, I can be myself.
I don't have to be this sterile doctor or whatever.
I can make jokes, you know, laugh and whatever. So
that's that's the way I That's why I make it
the way I do.

Speaker 1 (57:29):
Okay, so people can find you at Vineyard what's the
what's the joint?

Speaker 2 (57:33):
Yeah, join vineyard dot com. Apparently someone owned Vineyard because
there's probably a wine uh.

Speaker 1 (57:39):
I was thinking vineyard store.

Speaker 2 (57:42):
Yeah. Well yeah, there's a whole story of why I
chose I basically wanted to see there's some of these
places out there. They're so cheap and it's a race
to the bottom. Uh. So I want it to be
like a little bit higher upscale version of what you
see online. And so I was like Vineyard. It sounds
like a classy, nice name, and nobody owned us. So
I was like, all right, I'm going with that. So
that's why I chose the name. And I was like, yeah,

(58:04):
be a place where i'd want to hang out with
people at a Vineyard drinking wine, even though if you're
taking the medicine, you're not going to be one a
drinking wine. But like that's kind of the top busied.
Let's get the medicine for a week so we can
drink a little bit of wine. But yeah, so join
vineyard dot com. I only hire obesity trained doctors. You

(58:26):
get direct connection with them. There's no a lot of
these other places. They have a care coordinator, so you're
talking to a care coordinator and not the actual doctor,
and they sound like bots. So I small patient panels.
It's just it's very it's supposed to be white glove,
but it's not white glove prices where you're paying a
thousand dollars a month or whatever.

Speaker 3 (58:45):
Ye.

Speaker 1 (58:45):
Wow, fascinating. Well, thank you so much Spencer for coming on.
And I've been wanting to chat with you for a
long time because I told you I met you like
fifteen years ago at a gym when Alex Brice.

Speaker 2 (58:59):
Was yeah you don't remember, still my strength coach. So
it's good he is. Yeah, Yeah, he's my He does
my lift r X program which everybody gets for free,
and Vineyard Yeah.

Speaker 1 (59:12):
He wrote program.

Speaker 2 (59:13):
I love. He's great.

Speaker 1 (59:16):
He's great. Well, thank you for joining us, and thank
you for your time and for what you are bringing
to the space. I know our our audience is really
going to appreciate it, so thank you so much.

Speaker 2 (59:28):
Thanks for having me on.

Speaker 1 (59:30):
Thanks for listening everyone.

Speaker 3 (59:31):
If you enjoyed this episode, please consider giving us a
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