March 9, 2025 • 46 mins
Dr. Rachaele Carver turns conventional wisdom about heart disease on its head with groundbreaking insights into how your mouth bacteria might be silently damaging your cardiovascular system. Drawing from her presentation on "Oral Pathogens as Causative Agents in Cardiovascular Disease," she reveals the missing link between oral health and the world's leading killer.
While most cardiologists focus on managing high blood pressure and cholesterol levels, Dr. Carver explains these are merely consequences—not causes—of the real problem: endothelial dysfunction. This damage to the delicate lining of blood vessels often begins with bacteria from periodontal disease entering the bloodstream, where they employ sophisticated mechanisms to evade your immune system and directly colonize arterial walls.
The research is stunning: oral bacteria are found in 48-100% of atherosclerotic plaque specimens. These bacteria release toxins that degrade collagen, increase inflammation, and even hijack your platelets to form protective shields around themselves. Most remarkably, some bacteria can force your platelets to clump together, creating the small blood clots that can eventually block blood supply to your heart or brain.
Dr. Carver challenges the traditional approach of simply "killing bad bugs" with antibiotics or scaling procedures. Instead, she advocates for addressing the environmental factors that allowed pathogenic bacteria to flourish in the first place—increasing oxygen in the oral environment, supporting mitochondrial function with red light therapy, and addressing critical nutrient deficiencies in minerals and fat-soluble vitamins.
Ready to transform your understanding of oral-systemic health? Listen now to discover simple, effective strategies to protect your heart by optimizing your oral microbiome—and learn why rising cholesterol might actually be your body's desperate attempt to repair damage caused by hidden infections, not the villain it's made out to be.
To learn more about holistic dentistry, check out Dr. Carver's website:
http://carverfamilydentistry.com
To contact Dr. Carver directly, email her at drcarver@carverfamilydentistry.com
Want to talk with someone at Dr. Carver's office? Call her practice: 413-663-7372
Reverse Gum Disease In 6 Weeks! With Dr. Rachaele Carver Online Course!
Learn more about here: https://reversegumdiseaseinsixweeks.info/optinpage
Disclaimer: This podcast is for educational purposes only. Information discussed is not intended for diagnosis, curing, or prevention of any disease and is not intended to replace advice given by a licensed healthcare practitioner. Before using any products mentioned or attempting methods discussed, please speak with a licensed healthcare provider. This podcast disclaims responsibility from any possible adverse reactions associated with products or methods discussed. Opinions from guests are their own, and this podcast does not condone or endorse opinions made by guests. We do not provide guarantees about the guests' qualifications or credibility. This podcast and its guests may have direct or indirect financial interests associated with products mentioned.
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Transcript
Episode Transcript
Available transcripts are automatically generated. Complete accuracy is not guaranteed.
Dr. Carver (00:00):
Hello everybody, welcome back to another episode
of the Root of the Matterpodcast.
I am your host, dr RachelCarver, a board-certified
biologic and naturopathicdentist, as well as a certified
health coach.
I was asked by Cellcor to givea live talk about how oral
health is related tocardiovascular health, which
(00:22):
kind of tied in perfectly on mynew idea of doing a solo podcast
at least once a month.
Ryan, my fabulous marketingdirector, editor for this
podcast, his topic was hearthealth for Fit Me, right.
So it worked out perfectly andI was able to really dive into
some of the new research, whichwas really exciting.
So today we're going to talk alittle bit about that.
(00:45):
I'm going to go through mypresentation that I gave the
other day.
The title of that was OralPathogens as Causative Agents in
Cardiovascular Disease, and ifyou've been listening to this
podcast for a while, you knowhow I believe bacteria or any
kind of microbial infection andor toxins are really at the root
(01:05):
of most chronic diseases, right?
So it's not a surprise that anyoral bacteria could be a
causative factor in manysystemic diseases, right?
So if the bacteria gets intothe bloodstream which we know,
even just by regular chewing andjust regular brushing of your
food.
Bacteria gets into the gumtissue where there's lots of
(01:27):
blood vessels, right, or you'reswallowing that bacteria.
But if you don't have robuststomach acid to kill some of
those microbes, then thosemicrobes can get into the gut
and go wherever they might go.
So let's talk a little bit more.
How specifically it's related toheart disease, because heart
disease is still the number onekiller globally.
(01:47):
So endocarditis back when I wasgoing through school in the
early 2000s, we were basicallytold there were two ways that
oral health affected the rest ofthe body.
One was through this infectionof heart valves called
endocarditis, and it was faintlyrelated to diabetes, right,
blood sugar control.
(02:08):
Now we know, of course, 20 someodd years later, the links
between oral bacteria, dementiaand other brain issues is really
the research is really prolificthese days.
We know it's related to birthconsequences, right, prenatal
issues in the mom.
We know there's related tokidney disease, certain kinds of
(02:30):
cancers, etc.
Etc, etc.
So interestingly, thisendocarditis, which is an
infection of heart valves, wasfirst described in 1806.
That was even before we evenknew what bacteria was.
Then, about a century later, ascientist first isolated strep
viridans and so that was thebacteria that's most commonly
(02:52):
known to infect the heart valve,and then by about mid-1940s, we
understood that bacteria couldbe found in the blood after
tooth extractions.
And then it was 1951 where weproposed giving antibiotics to
patients with damaged valvesundergoing dental surgery.
So if you had an artificialvalve or a damaged heart valve,
(03:15):
it's always recommended.
Typically, we give amoxicillinunless the patient is allergic
to penicillin, and that waspretty well understood ones.
And that was pretty wellunderstood.
Well, what about gum disease?
Periodontitis, or gum disease,is another prominent factor that
can lead to heart disease.
(03:35):
We know that, as I mentioned,heart disease, number one cause
of death worldwide, accounts for33% of all deaths.
We also know that infection andinflammation of the gums, or
periodontitis, is consistentlyone of the top 10 most prevalent
conditions globally.
I found a couple of differentarticles, and one stated that
(03:57):
periodontitis is the sixth mostcommon disease in the world,
affecting over 11% of thepopulation.
Another article stated theglobal prevalence was 45%, and
so it depends how somebody isgoing to categorize what
periodontitis is.
It can be anywhere frominflamed gums like gingivitis,
all the way to severeperiodontitis where we're losing
(04:19):
teeth and have a lot of loss ofbone structure there.
There are many studies that areshowing.
Patients with severeperiodontitis have increased
levels of systemic inflammatorymarkers like interleukin-6,
c-reactive protein, and theyshow signs of the cholesterol
levels being off and endothelialdysfunction.
(04:41):
What is endothelial dysfunction?
The endothelium is the tissuethat lines inside of our blood
vessels, so when it gets damagedand there's a lot of
inflammation, we have thisendothelial dysfunction.
There was also a recent studyshowing that people who have
lost their teeth have a 66%higher chance of dying from
(05:03):
heart-related issues.
As I just mentioned, there are acouple different stages of gum
disease.
We have normal, healthy teeth.
That means when we brush and wefloss there's no bleeding,
there's very little amounts ofbreath, right, we do not have
decay, we don't have any boneloss.
Then we get into gingivitis andagain that's usually when the
(05:25):
gums are a little bit puffy.
They may bleed easily withbrushing and flossing.
Sometimes people who are mouthbreathers right their gums tend
to be especially right in thefront teeth, their gums tend to
be a little more sore and theymay bleed easily.
So then we have to make thatdistinction.
Is it the mouth breathing or isthere infection and
inflammation happening?
Has there been a shift in theoral microbiome that is causing
(05:49):
the gums to be inflamed and thenwe can have mild, moderate or
severe periodontitis and webasically categorize that based
on what are your popconingdepths, how much tartar calculus
is there and how much bone lossthat you have.
Obviously, with the moreseverity of the disease, the
more severity of systemicconsequences as well.
(06:09):
So, as many of you, if you'velistened to this podcast, my big
belief that disease comes fromwithin, right.
I'm a big proponent of theTureen theory, right.
I do not believe that pathogens, we swallow bad bugs all of a
sudden and get dumb disease.
Nor do I think that justbecause your mother, father,
brother, uncle, whatever, haddumb disease, that you are
(06:31):
destined to have it as well.
And there's a hypothesis calledthe ecological plaque
hypothesis, and even a recentarticle I was just reading
recently was stating that it iswell-established that disease is
not caused by exogenouspathogens but rather changes in
the microbial communitystructure.
(06:52):
So the environment within themouth changes for some reason.
Is it some kind of toxin?
And usually it's some kind oftoxin, toxin in the body.
I personally made somethinghappening in the gut that is
then causing deficiency, in myopinion, which I learned from
Cellcor, right, that toxicitycreates deficiency.
(07:14):
Think of it this way, right.
If we have a toxin, whetherit's a bug or a heavy metal,
this is affecting our body andour body is very smart, right.
The immune system can recognizethis foreign substance and
wants to get rid of it.
So we have to upregulate theimmune system right.
So we have to use, get ourmitochondria.
We have to upregulateeverything.
(07:34):
We need more nutrients, we haveto have more cells, and so if
we are not taking in more ofthose nutrients, then we become
very deficient and then thatdeficiency leads to a whole
other problem.
So that's what we want toreally think about.
There is, how do we get thatenvironment back to health?
(07:54):
Interestingly, in dental, inthe oral microbiome, we actually
want to have less diversity Inthe gut, the lower down gut,
right.
We're always talking about themore diverse species of bacteria
and fungus we have, thehealthier we are, and in the met
it's actually the opposite.
So when we start having gumdisease let's say you're doing a
(08:16):
saliva test, right, we mightshow increase in the number of
species, and in the oralmicrobiome that's actually a
negative thing.
So again, we have to thinkabout this is multifactorial.
There's not necessarily onething.
There may be that toxin thattips the scale.
But then it has to have thatperfect storm, right?
(08:36):
Just eating sugar isn't going tocause cavity, right?
We have to have a whole set ofenvironmental things for it to
manifest.
So we have to understand whatare the host defenses?
How strong and robust is yourimmune system?
What is your nutritional status?
As I just said, toxicity leadsto deficiency.
Are we getting enough of thefat soluble vitamins?
Do we have enough B vitamins?
(08:58):
Do we have enough fat solublevitamins?
The minerals very important.
How many toxins are we exposedto on a daily basis?
How many products are weputting in our mouths, on our
skin, in our hair, right?
Not to mention cleaningproducts.
We're just exposed to so manyenvironmental toxins at all
times.
(09:19):
And what's the pH?
Right?
We can't really have disease inour mouths if we haven't
neutral pH for the majority ofthe time.
Our mouth is always going to assoon as we eat.
If you were to take a pH strip,you would notice that it dips
down and becomes acidic.
That's normal In a healthymouth.
When you have a healthy amountof bacteria, within an hour or
two you should go back to aneutral pH.
(09:39):
But if you're consistentlystaying acidic again, that's one
type of environment that'sgoing to cause some of those bad
bugs to overgrow.
Interesting as the gum tissuestarts breaking down, we get
these collagen peptides andamino acids which actually feed
the what we call pathobionts.
(10:02):
And this is an interestingdistinction between the word
pathogen, because things likePorphylonis gingivalis, this is
present in health and that's theother thing.
To really understand, these fadbugs are present, they're all
around.
It's when the balance we getout of balance and there's too
many of those, that thesepathobionts become pathogens.
(10:24):
Right.
And the way that they're ableto overgrow is as we're breaking
down our own gum tissue, right.
That's feeding those nowpathogens, which increases their
numbers even more.
And this is why, on my SILHAtest, one of the markers on that
screening tool is protein.
If you see increased protein inyour saliva, that's a sign of
(10:48):
body's breaking things down.
It's a sign of you're insympathetic system.
Your body is in fight or flight, trying to get ahead of some
disease or pathogen or toxin.
And interestingly also is thatthe severity of periodontitis
will increase with differentspecies.
So P gingivalis is one of themost common bugs, that's most
(11:10):
commonly studied bugs, and weknow that it's implicated in
every chronic disease that weknow of.
But it gets even morepathogenic when also the
presence of TF orethnonucleotides.
So that's when we're doingthese saliva tests and we're
seeing these together, we knowthat it's going to be more
problematic.
We know that we haveperiodontitis which is right at
(11:36):
the gum line, but we also haveapical periodontitis, which is
what most people think of as anabscess.
So this is a lesion,inflammation, infection, tip way
, way down in the bone.
Sometimes it's hard to see on aregular 2D image.
This is why cone beam or 3Dx-rays are becoming more and
more common and more standard ofcare, because what we can see
(11:57):
in a three-dimensional is somuch more clear.
And many times these kinds ofinfections are silent.
We have absolutely no symptoms.
So your immune system could becompletely jacked up and you
have no idea why you have thischronic fatigue or fibromyalgia
or long COVID and you can'tfigure out why you can't get
better.
There may be an underlyinginfection that is causing that
(12:21):
immune system to go out of whackand not be able to heal
yourself and create energy.
We've talked about that before.
Right, the mitochondria can bein defense mode or they can be
in energy production mode, butthey can't be in both.
So, people with chronic fatigue, you've got something that is
bringing your mitochondria downright.
So is it the heavy?
Is it an infection?
(12:42):
What kind of chemical could itbe?
We've got to dig a littledeeper and try to get at the
root of it, versus just tryingto pep you up with caffeine or
apply you with other supplements.
We've got to get rid of thatroot cause.
So what are the typical riskfactors for cardiovascular
(13:02):
disease?
If you go to the cardiologist,they're going to freak out right
.
If you have high blood pressure, if your cholesterol levels are
out of whack, boom, we got toput you on stout, got to put you
on blood pressure medication.
The definition ofcardiovascular disease is it's
an inflammatory disease thatpresents as thickening of the
intima within the vessel.
So that basically meansnarrowing of all of the
(13:25):
vasculature, and that happens.
We get plaques that are laiddown right, and so now
everything becomes more narrow.
The more narrow yourcirculatory system is, the
harder right we have to pressureis going to be in order to get
that blood to all the areas ofthe body right.
So is it the blood pressurethat caused the cardiovascular
(13:47):
disease, or did something causethat blood pressure to go up?
So what I believe in all theresearch that I've done, is that
the initiating factor isactually what I mentioned
earlier endothelial dysfunction.
So it's not an increase inblood pressure, it's not an
increase in LDL causing thecardiovascular disease.
(14:08):
I see it, those areconsequences of this endothelial
dysfunction, and so we'vetalked about it a little bit
before.
What is the role in the body ofLDL?
Most of us think of it as thebad cholesterol.
We can't let that get too high.
We've got to suppress that.
But LDL, actually its functionis to be a band-aid, right?
(14:32):
So it accumulates in areas ofdamage, right?
These blood vessels are justlike the gut, are like one cell
line and cell layer, so they canbe very easily damaged, right?
So when this LDL they'reaccumulating these areas of
damage, then they becomeoxidized and it's the oxidized
fats that are really problematic.
(14:55):
That's why just looking at anLDL is really not sufficient to
tell you what your risk is ofcardioid, but we have to look at
the oxidized particles, thevery low density proteins there,
right?
So when they become oxidized,we get an increase in adhesion
molecules and then macrophages,which are cells of the immune
(15:16):
system.
They are activated and they aresupposed to scavenge and take
care of these LDL cells and wecreate what we call folin cells
and that's defined as firststage of atherosclerosis right,
or the plaque building up andthe hardening of our arteries.
These macromodules also producethese pro-inflammatory
(15:36):
cytokines.
And then the T cells, anotherimmune cell, is going to
stimulate proliferation of thesmooth muscle within the vessels
right.
So the smooth muscle cells aregoing to proliferate, they're
going to get thicker, there'sgoing to be more of them and
again that's going to cause anarrowing.
So if cholesterol, increasedcholesterol and increased blood
(15:57):
pressure are a consequence, whatis causing the dysfunction of
the endothelium in the firstplace?
So we know that strep, mutanssanguinus, aa, p gingival, t,
denticle all of these arebacteria strains that have been
found in aortic aneurysmspecimens and in diseased heart
(16:22):
valves.
They are frequently detected inatherosclerotic plaques and
studies have found differentoral species in anywhere from
48% to 100% of specimens.
Other infections herpes simplex, chlamydia, pneumonia that was
going around a lot in our areain its fall P gingivalis, hep C,
(16:44):
h pylori very common rightCauses a decrease in stomach
acid, influenza, a,cytomegalovirus, hiv those are
all implicated as risk factorsbecause all of those infections
can cause endothelialdysfunction.
In periodontitis we talk aboutthe red complex bacteria, the
(17:04):
worst of the worst.
So that includes the Pgingivalis, trypanema, denticola
, tannerella, forsythia,fusobacterium nucleatum, which
has recently been identified incolon cancers, prevotella
intermedia.
But again I want to stress thatthese that are only looking at
(17:30):
these red complex and maybe theorange complex, we're not really
getting a whole view of theoral cavity.
Although they are measuringlevels, it's important to
understand that they are goingto be present, so we cannot be
concerned if we have them.
We want to know how much of thegood commensal bacteria do we
(17:51):
have?
So all of these bacteria sharein common that they are grand
negative anaerobic, which meansthey thrive in a less oxygenated
environment.
So this is why, if you've everheard me talk or do my webinars,
I'm a big proponent of oxygendrops.
Getting more oxygen, a littlebit of hydrogen peroxide can go
a long way, because oxygen it'show the good commensal bacteria
(18:14):
they thrive on.
So the more oxygen we have, thefewer of these red complex
bacteria are going to be able tosurvive.
As I mentioned before too, whenwe find them together, they
create pathogenic synergism, andthe scary thing about these is
why.
Well, just previously, all weever want to do with these we're
(18:34):
going along.
Pasteur's theory right is justkill everything, so let's just
throw antibiotics at it.
But if you're more holisticwith oh I don't want to do
antibiotics, let me just throwessential oils at it, which are
also killing.
And again, we have to becareful because just if we kill
these bacteria but we don't fixthe environment that allowed
(18:54):
them to overgrow in the firstplace, we're just on that
hamster wheel, spinning andspinning and we're never
actually correcting the problem.
So we're managing the diseasewithout reversing it.
The interesting thing for me Iknow everything is so
interesting to me but thebacteria actually suppress our
innate immune system.
So not only are they creatingall sorts of inflammation and
(19:19):
endothelial dysfunction, theyare preventing our immune system
from being able to eradicatethem, they also cause blood
clotting right.
So we know the thicker ourblood is, the more easily that
it clumps, the more easily itcan adhere right to those
plaques that are forming theartery or they're more narrow,
the vessels are, harder it isfor that thick, clotty blood to
(19:42):
get through.
So that becomes a real problem.
They also produce enzymescalled proteases, which is what
helps some of the blood clotsand other things adhere and
cause invasion of other healthycells.
So let's talk a little bitabout these different bacteria.
P gingivalis it will upregulatefatty acid binding, so more and
(20:04):
more LDL will be attracted tothose sites which can then be
potentially oxidized, whichleads to more of the
proliferation of cells and theformation of these plaques.
P gingivalis can prevent theHDL right, that's the good
cholesterol from binding andtransporting right.
Normally HDL comes along and itchews up those LDL and goes on
(20:27):
its way.
But if the LDL now can't dothat anymore, right, that's when
we get that increase that theLDL isn't being taken away.
We just keep making more andmore because there's more and
more damage occurring.
P gingivals can oxidize HDL andthen now, instead of being that
protective transporter, it'sbecoming pro-aphrogenic.
(20:48):
P genovas also can ship theballot of our immune cells, th17
and T-regulatory cells toencourage more plaque formation
and it causes the aggregationand the clotting of the
platelets.
F nucleonum has been found inmany about 34% according to this
one study, 34% in carotidspecimen.
(21:11):
It increases cardiovascularrisk by increasing systemic
inflammation by directcolonization of these arterial
walls.
Okay, so it's directlyattaching to the arterial walls,
causing endothelial dysfunctionand, as I said too, it acts as
a bridge.
So when F-nucleotide is therewith P gingivalis, they work
(21:34):
together to create more adhesionmolecules and form more complex
microbial communities Often.
There was one study just donein 2022 in mice and they said
the oral administration ofF-nucleotide caused increases in
triglyceride and cholesterol,the T-fersyphia, t-dentacoloc,
(21:56):
both showing an atheroscleroticreasons average of 53%.
They cause an increase inC-reactive protein.
That is a marker often run byhopefully run by most internists
to look at any type ofinflammation.
It's a non-specific marker forchronic inflammation.
It also causes increase in LDL.
(22:16):
Right, again, the more damageto the endothelium, the more LDL
we're going to produce.
So when I see somebody whoseLDL is creeping up or getting
higher, I'm thinking what'scausing the damage?
Why is the liver beingrecruited to create more
Band-Aid?
Why do we need all theseBand-Aids?
Also, these two species alsowill lower nitric oxide.
(22:37):
There's one study done in mice.
Nitric oxide is one of themolecules responsible for
dilating our blood vessels.
Right we're talking about.
All these things are causingnarrowing of our blood vessels
and so it makes it harder to getthe blood and get oxygen where
it needs to go.
So nitric oxide important,right?
It's also correlated.
These two are correlated withhemorrhage plaques in the
(23:00):
vessels.
There was a study done byJenkinson and his colleagues and
they found that onestreptococcus bacteria was able
to get into the bloodstream.
And again, this can simply befrom chewing food, right,
brushing our teeth.
They found a protein theycalled PAD-A.
It sits on the outer surface ofthis bacteria and they say that
(23:23):
it hijacked blood platelets andforced them to clump together,
making these blood clots.
And when these clump togetherlike this, they surround the
bacteria and create almost ashell.
So when the bacteria is likeinside this clump of platelets,
they avoid detection by the hostimmune system.
(23:44):
This also allows them to hidefrom antibiotics.
So we know there's a hugedilemma with antibiotic
resistance happening and we'regetting all these superbots
happening.
Bacteria are very smart andthey can evolve and find ways to
evade detection and destruction.
So, very hard you have theseinfections of just throwing.
(24:05):
Again, when we talk about let'sjust kill everything with all
these antibiotics If theantibiotic can't get to the
bacteria, we're not really doinggood work right, where instead
we could be potentially causingmore problems by killing off
good commensal bacteria that areso important for absorbing our
nutrients and helping our immunesystem.
So, again, as well as helpingout the bacteria, this platelet
(24:28):
clumping, called small bloodclot, and they can grow in the
heart valves, causing thisendocarditis and further
inflammation of the bloodvessels, which can actually
block blood supply right to theheart and brain.
So they're working on somethingto try to counteract this
PADD-A where, yes, that's great,but wouldn't it be wonderful if
(24:49):
we just cleaned up the mouthright and the mouth is so
accessible?
That's what I love about my job.
It's a lot easier to try tocreate balance in the oral
cavity than trying to heal leakygut or, you know, ibs, right?
So we have the opportunity tohave direct impact on the mouth
and it's going to have directimpact on heart health, brain
(25:10):
health, liver health, you nameit.
How exactly is this dysfunctionhappening?
We've mentioned a few things,right?
So these infections get intothe bloodstream, they cause
clotting, they cause lyticsystems to activate all of this
fibrous, these specific cells,these foam cells.
They activate all of the innateimmune systems or start
(25:33):
producing more cytokines thatlead to more inflammation and
that causes just acceleration ofthese plaques throughout the
body.
Many of these plaques also showsigns of releasing endotoxins.
So it's not just the bacteria,but it's the byproducts of this
bacteria that can further causemore damage, and there are
(25:55):
several different kinds ofbyproducts we'll talk about
quickly.
So gingipines is one of thesetoxins given off by these
bacteria and these are cysteineproteases.
They attach to the host tissue,so they're going to attach to
that cell wall in the bloodvessel and they cause fibrous
(26:16):
maturation.
It gets a little thicker inthere.
Hemoglobin also causeshemoglobin binding and so that
hemoglobin binding causes thebacteria to be able to collect
amino acid and that's what theyfeed on.
These ginger peels are whatdirectly cause the degradation
of the collagen and theextracellular matrix proteins.
(26:36):
Right, so that's when we startto see the bone loss happen.
Right, and this is why I thinkof periodontitis as in the same
category as autoimmunity.
Right, body is eating away atyourself.
But I listened to this greatpodcast the other day.
It was Dave Asprey and GaryBrekka.
Gary was talking about what isautoimmunity and most of us
(27:00):
think of the body's gone haywire.
Right, it's doing something itshouldn't do.
But what he's saying?
No, the body is doing exactlywhat it's supposed to do because
by attacking the toxin.
I totally share this opinionthat, again, everything is
toxins and infections.
Why do we have he's talkingabout thyroid issue why is the
body attacking the thyroidtissue.
(27:22):
That thyroid tissue has beeninfected by a toxin, so the body
is producing antibodies rightto go after the toxin.
The toxin is in that thyroidtissue.
So what happens is the thyroidtissue ends up being attacked as
the body is trying to attackthe toxin.
So same exact thing in gumdisease.
Right, all of the endotoxinslike gingipanes and LPS are
(27:43):
being released.
The body wants to get rid ofthose toxins.
Part of if that toxin is in thebone that's surrounding the
teeth, then the bone is going toget eaten away in the body's
attempt to get rid of the toxin.
So, again, if we get rid of thetoxin, we get rid of the
infection, then the body isgoing to stop having this
autoimmune reaction and stopdestructing our own tissue.
(28:06):
What else do these genoplastiesdo?
They will inactivateimmunoglobulins and cytokines
that compromise the immunesystem.
We said that it suppresses theinnate immune system, causes
dysregulation of the coagulationcascade.
So this is why in gingivitisand periodontal disease we see
increased bleeding, so we canhave blood clotting, but we also
can see increased bleeding.
(28:27):
It also increases the vascularpermeability right, we all know
about leaky gut.
Anywhere you have a barrier,right In your mouth, in your
blood-brain barrier vessels ingeneral right Leaky vessels,
right.
So we're going to increase thatvascular permeability, which
will attract more white bloodcells to the infection.
It also activates MMPs thatdegrade more of that alveolar
(28:50):
bone, as well as prostaglandins,which cause pain.
Lps this is a really well-knowntoxin that bacteria give off.
It is, according to someresearch, one of the major
factors leading to fatty liverdisease.
So again, really important tounderstand the consequence of
having these low-grade chronicinfections.
(29:10):
Lps is actually a component ofthe outer membrane of
gram-negative bacteria.
Normally it's recognized by theinnate immune system at these
CD14 receptors and that willinitiate the cytokine cascade
and platelet aggregation againtrying to encapsulate these
bacteria and move them out ofthe way.
Lps will increase calcification.
(29:34):
So for you women out, there yougo, you have your mammogram and
they're like oh yes, you havedense breasts, lots of
calcification.
This is just normal, it's verycommon, but it doesn't mean
that's normal.
So if we have a lot ofcalcification, we're producing a
lot of tartar on our teeth allthe time, right?
Why do we have this increasedcalcification?
Could it be caused from someagain low-grade infection going?
(29:58):
Lps causes a progression of theplaque lipid deposition, so it
increases more LDL by damagingthat endothelial barrier,
increases oxidative stress.
Most diseases have increasedoxidative like.
The whole idea of aging is allabout oxidative stress, right?
It also LPS directly increasesthe C reactive protein.
(30:18):
There are also bioprones calledleukotoxins.
So from the bacteria AA itactually causes pores in the
lipid bilayer, right?
So every cell has this lipidbilayer, every cell has this
lipid bilayer that protects,actually, the brain of the cell.
It's this lipid bilayer, and soAA can directly cause holes and
can kill white blood cells.
(30:39):
By creating these holes itcauses apoptosis, which is
programmed cell death, increasedbreakdown of the macrophages
right?
Those are supposed to be thecleanup crew and get rid of all
the bad stuff.
It can, those are supposed tobe the cleanup crew, get rid of
all the bad stuff.
It can break down naturalkiller cells and the T cells,
(31:00):
which are protective, right?
So again, suppressing thatinnate immune system by directly
killing our immune cells.
It can also the glucotoxinsbind to the white blood cells
and therefore wherever thatwhite blood cell goes in the
bloodstream, so does theinfection, can go somewhere else
in the body and cause problemsin that part of the body.
There are also heat shockproteins, so during an infection
we have bacterial heat shockproteins and they're very highly
(31:21):
immunogenic.
So they're very highlystimulatory to our immune system
.
And the problem is we havehuman heat shock proteins and
the body may get confused andnot be able to differentiate
between them.
So those heat shock proteinscan further cause endothelial
function.
They also enhance adhesionmolecules, which causes more
thinning of the blood vessels,more patient.
(31:45):
The cytokines everybody heard alot about cytokines during the
pandemic and how that cytokinestorm really caused death.
Right when the inflammation gotway too ramped up there was
just such an increase ininfection that the body couldn't
keep up.
So having cytokines isimportant, but having too many
is not really a good thing.
We get increased swelling, moregranulation tissue when we have
(32:08):
too many cytokines.
So what is the traditionaltherapy?
Traditionally for a gone disease, periodontitis, we do scaling
and root cleaning, right.
We numb you up and we scrapethe crap out of your teeth
trying to get all that off andthen send you on your way and
most studies say that's great,but actually we can regrow
plaque and tartar within 10 days.
(32:30):
So that doesn't reduce theamount of bacteria in the mouth.
Reduce the amount of bacteriain the mouth, but did we do
anything to address theenvironment.
Again, all the idea of justkilling, just get rid of the
bacteria and then everythingwill be wonderful.
So then down the line westarted.
(32:55):
What if we add antibiotics withthe scaling root planing?
So we can give systemicantibiotics, which we now know
is probably not a great idea, orwe can give targeted
antibiotics.
Let's just put that in that 10millimeter pocket, right, so we
have certain kinds ofantibiotics that we can localize
and put it right in.
But again, is anything reallylocalized?
Because, right, it can get intoyour mean bloodstream, but it
is in a smaller dose and thereare a lot of studies that really
(33:18):
show no inhibition.
Again, maybe initially for afew weeks, but then really we go
right back to square one.
So in my mind, like, okay.
Again I think about theenvironment.
Yes, we have to reduce theamount of bacteria.
So, yes, let's get the tartarout of there, let's get these
roots nice and cleaning right sowe can have the gum tissue
(33:40):
reattached in these areas.
What else can we do?
What if we use laser, right?
Laser light energy, as you'veheard me talk about in previous
podcasts, is amazinglystimulatory for mitochondria.
We need that increased energyin order to produce more oxygen,
make more ATP to help ourimmune cells fight whatever the
(34:02):
inflammation is going on.
So laser totally non-invasiveand wonderful, I find that we
really do need to use it incombination with other things.
So we have scalar and plateletplus ozone.
Again, we need to take downthat and we need to put more
oxygen in that environmentdirectly.
And then enzymes I really likeproteolytic enzymes because,
(34:24):
again, remember we talked abouthow amino acids are used by the
pathogens in order to grow andincrease their numbers.
So if we degrade down all ofthose amino acids, we're going
to take away the food source,right.
So again, think about theenvironment.
I love oxygen drops.
I want my patients all to be onoxygen drop and then we can use
(34:46):
combinations from all thesetoxicity.
We need to increase our minerals.
We need to increase the fatsoluble vitamins, right D, b, a
and K.
They all have to work together.
The fat soluble vitamins andminerals work together to
(35:08):
produce healthy teeth and gums.
You can't just take vitamin D,you can't just take magnesium.
The combination together iswhat creates health.
And again, with prebiotics,probiotics we're providing the
food for the good stuff toovergrow.
And I've done my own littlepilot studies and I've seen
incredible results with thistype of therapy Without changing
(35:33):
diet, which we know.
Diet is so crucial.
Right, a lot of the toxins weis from our diet.
We eat so much processed food.
It's just hard to eat healthywhole foods these days, just the
way society is.
But again, with even justtargeting these things in the
mouth, it's going to make a hugesystemic impact.
So the goal is always toincrease the good bacteria and
(35:57):
reduce the diversity in the oralmicrobe.
So we need to reduce the toxinand we need to improve our
nutrition.
Now there are some people withcertain polymorphisms in their
genetics, right.
There's polymorphisms or SNPs,we call them right in certain
cytokines and that can lead tosome people have greater
inflammation.
(36:17):
There's some about MTHFR.
Many of you have heard of that.
It's probably one of the beststudies out there.
If we don't methylate properly,we can't detox properly.
Chronic anemia that is also canbe a sign of chronic disease.
So a lot of people freak out ohmy gosh, we need to supplement
you with iron because you'rebecoming so anemic.
(36:39):
But again, why are they anemic?
Could it be a chronic infectionstealing all of that iron?
Again, let's dig a littledeeper on these things and
figure out the real reason.
So let's sum everything up here.
We need to think aboutnutrients versus toxins, right?
Toxicity leads to deficiency,so our best goal to fight any
(37:02):
chronic disease is making surewe're getting those nutrients
and minimizing the toxins to thebest of our ability.
And in today's society, Ibelieve that supplements are
necessary.
And again, basics Everybody'sdifferent, right?
Our lifestyles are a littledifferent, Our genetics are a
little different, our geneticsare a little different, but it's
very basic.
Having those fat solublevitamins, having the minerals
(37:23):
that is key cornerstone of goodhealth.
So you just got to have thebasic building blocks for
everything to work well.
Then we have to also thinkabout our nervous system, right?
Those of us who tend to havemore chronic issues are those of
us who spend way too much timein that sympathetic nervous
system.
We're overthinking stuff allthe time.
(37:46):
There's just so much bombardingus and we don't realize it.
Led lights, too much of that.
We're inside all the time, thenutrient-poor food, so that we
have a lot working against us.
But that's okay, because we'reaware now and so we know we can
take these little steps to bringus that much healthier.
All right.
(38:08):
So the biggest takeaways for meis that, remember, the cause of
heart disease is not increasedblood pressure and increased
cholesterol, right, the cause isthe endothelial dysfunction.
The cause is from.
It could be these bacteria,these viruses, these toxins like
the LPS and the gingipanes,right, and it is the shift in
(38:31):
the environment that leads to anincrease in these pathobionts
that become pathogens, right, soit's the lack of nutrients, the
lack of oxygen, the lack of theminerals, the lack of vitamins
and excess sugars right, wedidn't even talk about sugar at
all.
We know sugar can lead to decay, right, but sugar's also
directly caused an increase inthe reactive oxidative species.
(38:54):
Anytime we have those increasedROS, we're going to have
chronic disease and inflammation.
So, an interesting thing is,blood glucose will also
stimulate HMG-CoA reductase.
So this is the enzyme that'stargeted for statin drugs.
If you have high cholesterol,often your doctor throws you on
(39:16):
a statin, and that is what'sgoing to stop the cholesterol
biosynthesis, right, stop theproduction of that LDL.
But what did we just mentiontoday?
Right, ldl is the band beingproduced.
We want to understand whythere's an increase of Band-Aids
(39:38):
being produced, and bloodglucose is a big one, right?
So if your cholesterol is goingup, the first thing I want you
to do is look for the hiddeninfection.
Test your HbA1c, right.
Because glucose this is goingto affect your liver.
Liver is where cholesterol ismade.
Liver also is protective of thepancreas.
(39:59):
So diabetes, and that reallystarts with a liver problem and
a metabolism problem.
So we've got to think about howall these things work together.
And blood glucose in and ofitself causes direct damage to
the endothelium.
Think about diabetics losinglimbs, losing their eyesight.
This is because that excessblood is directly causing
(40:21):
endothelial dysfunction.
I had a patient the other daywas talked about having swelling
in his legs.
He had recently been put oncholesterol medication.
A blood pressure medicationsaid his cholesterol was 500.
And so then the doctor thoughtoh, you have a heart problem, no
, you have a kidney problem.
He was diagnosed with a kidneydisease.
My first question was have they?
(40:42):
Even I can't seem to loseweight and I said have they
checked your blood sugar?
Because what he's experiencedis this leaky vessel syndrome.
Remember, we talked about allof the bacteria and these toxins
cause vascular permeability,the leaking out of fluids, right
.
So when we get edema orswelling, that is a leaky vessel
right, that is endothelialdysfunction.
(41:05):
So let's take it all the wayback and think what is the
infection, what is the toxinthat could be causing, instead
of simply taking a certainprescription to minimize the
symptom?
Because we may minimize thesymptom, but if we don't correct
the damage, we're just pushingthe buck down the line a little
bit without really resolving theissue.
So a really important keyfactor too, with increased blood
(41:29):
pressure and increasedcholesterol.
Please take a look at it.
Is your C-reactive proteinelevated and what is your blood
flow?
Look at the insulin.
Look at your blood glucose.
Look at your.
Is your C-reactive proteinelevated and what is your blood
flow?
Look at the insulin.
Look at your blood glucose.
Look at your HbA1c, which is ameasure of your blood sugar over
the last three months.
So it's a good thing to see.
Okay, and we know that type 2diabetes is reversible.
(41:50):
It may not be simple, but withcertain lifestyle changes and
tweaking our nutrition, weabsolutely can reverse all of
these things.
It's just a matter of thinkinga little differently and taking
the time to dig a little bitdeeper, right?
So we want to talk about theaction steps here, right?
(42:11):
So we might want to test fororal microblom.
I like Br Health Check for 700different species so we can see
the balance.
Right, as I just mentioned,check the C-reactive protein,
all of the blood levels and whatabout your mineral levels and
fat soluble vitamins.
Let's check those levels.
Let's talk about diet andstress.
How about sleeping?
(42:32):
Briefly mentioning, mouthbreathing causes a huge shift in
the oral microbiome, so shouldwe have a sleep study?
No, we can do them at home.
They're super easy and portable, right?
So let's talk about how we'rebreathing during the day.
Right?
Are we mouth breathing duringthe day?
Let's talk about some additivefunctions at the dentist, right?
(42:53):
Between ozone you can buyaffordable ozone machines now to
have in your home.
Even lasers we sell a littlered light hand-held, looks like
a flashlight about $225, right,that's not bad if it's something
you're using every single dayand you're really enhancing your
mitochondria.
And at home, let's think abouttoothpaste that don't have a lot
of chemicals in them.
Let's think about somethinglike Rewitin prebiotic minerals
(43:17):
don't have a lot of chemicals.
Let's think about somethinglike rewiten prebiotic minerals,
vitamins it's enhancing thatenvironment.
Liquid minerals A lot of us havedigestive issues.
If we take a liquid mineral,we're going to absorb that
through the mucosa faster.
It doesn't have to go throughthe digestive process.
Let's think about enzymes,right?
I've used a lot of enzymes tohelp people with arthritis,
right, which is basicallyinflammation in the joint.
You need to break down allthose excess proteins that are
(43:39):
causing the body to cause moreinflammation and breakdown of
the joints.
Oxygen and binders expand ofthe binders.
We talk about cell 4 bindersall the time.
The fulvic and humic acids notonly bind up all of these toxins
, but they also supplyelectrolytes and amino acids at
the same time.
(43:59):
So sometimes binders can taketoo much of our fluids and leave
us at more deficiency, whereasthe cell core binders are very
different or created muchdifferently than, say, like
activated charcoal.
So these are things that Ialways think about when I'm
looking at my patients and Iwant to really reverse all of
these kind of inflammationchanges that are going on.
(44:23):
So we've talked about a lot ofstuff today and I hope that all
of you can take a nugget or twoout of this and really start
thinking about.
My goal in doing this podcastis to make us all our own best
doctors, to make us think alittle bit more critically about
the cause of certain things andsimple things that we can do
(44:44):
after to give us a better,healthiest life possible.
I hope you enjoyed this solopodcast.
Please feel free to reach outwith questions and I love
comments.
I love ideas for new podcastsor people you want to hear from,
so keep the comments coming Ifyou are getting some value out
of the podcast.
(45:05):
Again, I thank you.
Please share this with others.
You can give us a review thatreally helps more people see, so
that we can create as muchawareness and help as many
people as possible.
So thanks again, everybody.
I hope you have a wonderful dayand I'll see you on the next
episode.
Hello, I'm Dr Rachel Carver, aboard-certified naturopathic,
(45:26):
biologic dentist and a certifiedhealth coach.
Did you know that over 80% ofthe US population has some form
of gum disease?
Many of us don't even know thatwe have this source of chronic
infection and inflammation inour mouth that's been linked to
serious consequences like heartdisease, diabetes, stroke,
dementia, colon cancer, kidneydisease, even pregnancy
(45:47):
complications.
Would you like to learn how toreverse and prevent these
chronic debilitating conditionswithout spending a lot of time
and money at the dentist?
Join me for my six-week coursewhere I will teach you the root
cause of disease.
You'll learn how to be your ownbest doctor.
Are you ready to get started?
Let's go.
Hello everybody, welcome back to another episode
of the Root of the Matterpodcast.
I am your host, dr RachelCarver, a board-certified
biologic and naturopathicdentist, as well as a certified
health coach.
I was asked by Cellcor to givea live talk about how oral
health is related tocardiovascular health, which
(00:22):
kind of tied in perfectly on mynew idea of doing a solo podcast
at least once a month.
Ryan, my fabulous marketingdirector, editor for this
podcast, his topic was hearthealth for Fit Me, right.
So it worked out perfectly andI was able to really dive into
some of the new research, whichwas really exciting.
So today we're going to talk alittle bit about that.
(00:45):
I'm going to go through mypresentation that I gave the
other day.
The title of that was OralPathogens as Causative Agents in
Cardiovascular Disease, and ifyou've been listening to this
podcast for a while, you knowhow I believe bacteria or any
kind of microbial infection andor toxins are really at the root
(01:05):
of most chronic diseases, right?
So it's not a surprise that anyoral bacteria could be a
causative factor in manysystemic diseases, right?
So if the bacteria gets intothe bloodstream which we know,
even just by regular chewing andjust regular brushing of your
food.
Bacteria gets into the gumtissue where there's lots of
(01:27):
blood vessels, right, or you'reswallowing that bacteria.
But if you don't have robuststomach acid to kill some of
those microbes, then thosemicrobes can get into the gut
and go wherever they might go.
So let's talk a little bit more.
How specifically it's related toheart disease, because heart
disease is still the number onekiller globally.
(01:47):
So endocarditis back when I wasgoing through school in the
early 2000s, we were basicallytold there were two ways that
oral health affected the rest ofthe body.
One was through this infectionof heart valves called
endocarditis, and it was faintlyrelated to diabetes, right,
blood sugar control.
(02:08):
Now we know, of course, 20 someodd years later, the links
between oral bacteria, dementiaand other brain issues is really
the research is really prolificthese days.
We know it's related to birthconsequences, right, prenatal
issues in the mom.
We know there's related tokidney disease, certain kinds of
(02:30):
cancers, etc.
Etc, etc.
So interestingly, thisendocarditis, which is an
infection of heart valves, wasfirst described in 1806.
That was even before we evenknew what bacteria was.
Then, about a century later, ascientist first isolated strep
viridans and so that was thebacteria that's most commonly
(02:52):
known to infect the heart valve,and then by about mid-1940s, we
understood that bacteria couldbe found in the blood after
tooth extractions.
And then it was 1951 where weproposed giving antibiotics to
patients with damaged valvesundergoing dental surgery.
So if you had an artificialvalve or a damaged heart valve,
(03:15):
it's always recommended.
Typically, we give amoxicillinunless the patient is allergic
to penicillin, and that waspretty well understood ones.
And that was pretty wellunderstood.
Well, what about gum disease?
Periodontitis, or gum disease,is another prominent factor that
can lead to heart disease.
(03:35):
We know that, as I mentioned,heart disease, number one cause
of death worldwide, accounts for33% of all deaths.
We also know that infection andinflammation of the gums, or
periodontitis, is consistentlyone of the top 10 most prevalent
conditions globally.
I found a couple of differentarticles, and one stated that
(03:57):
periodontitis is the sixth mostcommon disease in the world,
affecting over 11% of thepopulation.
Another article stated theglobal prevalence was 45%, and
so it depends how somebody isgoing to categorize what
periodontitis is.
It can be anywhere frominflamed gums like gingivitis,
all the way to severeperiodontitis where we're losing
(04:19):
teeth and have a lot of loss ofbone structure there.
There are many studies that areshowing.
Patients with severeperiodontitis have increased
levels of systemic inflammatorymarkers like interleukin-6,
c-reactive protein, and theyshow signs of the cholesterol
levels being off and endothelialdysfunction.
(04:41):
What is endothelial dysfunction?
The endothelium is the tissuethat lines inside of our blood
vessels, so when it gets damagedand there's a lot of
inflammation, we have thisendothelial dysfunction.
There was also a recent studyshowing that people who have
lost their teeth have a 66%higher chance of dying from
(05:03):
heart-related issues.
As I just mentioned, there are acouple different stages of gum
disease.
We have normal, healthy teeth.
That means when we brush and wefloss there's no bleeding,
there's very little amounts ofbreath, right, we do not have
decay, we don't have any boneloss.
Then we get into gingivitis andagain that's usually when the
(05:25):
gums are a little bit puffy.
They may bleed easily withbrushing and flossing.
Sometimes people who are mouthbreathers right their gums tend
to be especially right in thefront teeth, their gums tend to
be a little more sore and theymay bleed easily.
So then we have to make thatdistinction.
Is it the mouth breathing or isthere infection and
inflammation happening?
Has there been a shift in theoral microbiome that is causing
(05:49):
the gums to be inflamed and thenwe can have mild, moderate or
severe periodontitis and webasically categorize that based
on what are your popconingdepths, how much tartar calculus
is there and how much bone lossthat you have.
Obviously, with the moreseverity of the disease, the
more severity of systemicconsequences as well.
(06:09):
So, as many of you, if you'velistened to this podcast, my big
belief that disease comes fromwithin, right.
I'm a big proponent of theTureen theory, right.
I do not believe that pathogens, we swallow bad bugs all of a
sudden and get dumb disease.
Nor do I think that justbecause your mother, father,
brother, uncle, whatever, haddumb disease, that you are
(06:31):
destined to have it as well.
And there's a hypothesis calledthe ecological plaque
hypothesis, and even a recentarticle I was just reading
recently was stating that it iswell-established that disease is
not caused by exogenouspathogens but rather changes in
the microbial communitystructure.
(06:52):
So the environment within themouth changes for some reason.
Is it some kind of toxin?
And usually it's some kind oftoxin, toxin in the body.
I personally made somethinghappening in the gut that is
then causing deficiency, in myopinion, which I learned from
Cellcor, right, that toxicitycreates deficiency.
(07:14):
Think of it this way, right.
If we have a toxin, whetherit's a bug or a heavy metal,
this is affecting our body andour body is very smart, right.
The immune system can recognizethis foreign substance and
wants to get rid of it.
So we have to upregulate theimmune system right.
So we have to use, get ourmitochondria.
We have to upregulateeverything.
(07:34):
We need more nutrients, we haveto have more cells, and so if
we are not taking in more ofthose nutrients, then we become
very deficient and then thatdeficiency leads to a whole
other problem.
So that's what we want toreally think about.
There is, how do we get thatenvironment back to health?
(07:54):
Interestingly, in dental, inthe oral microbiome, we actually
want to have less diversity Inthe gut, the lower down gut,
right.
We're always talking about themore diverse species of bacteria
and fungus we have, thehealthier we are, and in the met
it's actually the opposite.
So when we start having gumdisease let's say you're doing a
(08:16):
saliva test, right, we mightshow increase in the number of
species, and in the oralmicrobiome that's actually a
negative thing.
So again, we have to thinkabout this is multifactorial.
There's not necessarily onething.
There may be that toxin thattips the scale.
But then it has to have thatperfect storm, right?
(08:36):
Just eating sugar isn't going tocause cavity, right?
We have to have a whole set ofenvironmental things for it to
manifest.
So we have to understand whatare the host defenses?
How strong and robust is yourimmune system?
What is your nutritional status?
As I just said, toxicity leadsto deficiency.
Are we getting enough of thefat soluble vitamins?
Do we have enough B vitamins?
(08:58):
Do we have enough fat solublevitamins?
The minerals very important.
How many toxins are we exposedto on a daily basis?
How many products are weputting in our mouths, on our
skin, in our hair, right?
Not to mention cleaningproducts.
We're just exposed to so manyenvironmental toxins at all
times.
(09:19):
And what's the pH?
Right?
We can't really have disease inour mouths if we haven't
neutral pH for the majority ofthe time.
Our mouth is always going to assoon as we eat.
If you were to take a pH strip,you would notice that it dips
down and becomes acidic.
That's normal In a healthymouth.
When you have a healthy amountof bacteria, within an hour or
two you should go back to aneutral pH.
(09:39):
But if you're consistentlystaying acidic again, that's one
type of environment that'sgoing to cause some of those bad
bugs to overgrow.
Interesting as the gum tissuestarts breaking down, we get
these collagen peptides andamino acids which actually feed
the what we call pathobionts.
(10:02):
And this is an interestingdistinction between the word
pathogen, because things likePorphylonis gingivalis, this is
present in health and that's theother thing.
To really understand, these fadbugs are present, they're all
around.
It's when the balance we getout of balance and there's too
many of those, that thesepathobionts become pathogens.
(10:24):
Right.
And the way that they're ableto overgrow is as we're breaking
down our own gum tissue, right.
That's feeding those nowpathogens, which increases their
numbers even more.
And this is why, on my SILHAtest, one of the markers on that
screening tool is protein.
If you see increased protein inyour saliva, that's a sign of
(10:48):
body's breaking things down.
It's a sign of you're insympathetic system.
Your body is in fight or flight, trying to get ahead of some
disease or pathogen or toxin.
And interestingly also is thatthe severity of periodontitis
will increase with differentspecies.
So P gingivalis is one of themost common bugs, that's most
(11:10):
commonly studied bugs, and weknow that it's implicated in
every chronic disease that weknow of.
But it gets even morepathogenic when also the
presence of TF orethnonucleotides.
So that's when we're doingthese saliva tests and we're
seeing these together, we knowthat it's going to be more
problematic.
We know that we haveperiodontitis which is right at
(11:36):
the gum line, but we also haveapical periodontitis, which is
what most people think of as anabscess.
So this is a lesion,inflammation, infection, tip way
, way down in the bone.
Sometimes it's hard to see on aregular 2D image.
This is why cone beam or 3Dx-rays are becoming more and
more common and more standard ofcare, because what we can see
(11:57):
in a three-dimensional is somuch more clear.
And many times these kinds ofinfections are silent.
We have absolutely no symptoms.
So your immune system could becompletely jacked up and you
have no idea why you have thischronic fatigue or fibromyalgia
or long COVID and you can'tfigure out why you can't get
better.
There may be an underlyinginfection that is causing that
(12:21):
immune system to go out of whackand not be able to heal
yourself and create energy.
We've talked about that before.
Right, the mitochondria can bein defense mode or they can be
in energy production mode, butthey can't be in both.
So, people with chronic fatigue, you've got something that is
bringing your mitochondria downright.
So is it the heavy?
Is it an infection?
(12:42):
What kind of chemical could itbe?
We've got to dig a littledeeper and try to get at the
root of it, versus just tryingto pep you up with caffeine or
apply you with other supplements.
We've got to get rid of thatroot cause.
So what are the typical riskfactors for cardiovascular
(13:02):
disease?
If you go to the cardiologist,they're going to freak out right
.
If you have high blood pressure, if your cholesterol levels are
out of whack, boom, we got toput you on stout, got to put you
on blood pressure medication.
The definition ofcardiovascular disease is it's
an inflammatory disease thatpresents as thickening of the
intima within the vessel.
So that basically meansnarrowing of all of the
(13:25):
vasculature, and that happens.
We get plaques that are laiddown right, and so now
everything becomes more narrow.
The more narrow yourcirculatory system is, the
harder right we have to pressureis going to be in order to get
that blood to all the areas ofthe body right.
So is it the blood pressurethat caused the cardiovascular
(13:47):
disease, or did something causethat blood pressure to go up?
So what I believe in all theresearch that I've done, is that
the initiating factor isactually what I mentioned
earlier endothelial dysfunction.
So it's not an increase inblood pressure, it's not an
increase in LDL causing thecardiovascular disease.
(14:08):
I see it, those areconsequences of this endothelial
dysfunction, and so we'vetalked about it a little bit
before.
What is the role in the body ofLDL?
Most of us think of it as thebad cholesterol.
We can't let that get too high.
We've got to suppress that.
But LDL, actually its functionis to be a band-aid, right?
(14:32):
So it accumulates in areas ofdamage, right?
These blood vessels are justlike the gut, are like one cell
line and cell layer, so they canbe very easily damaged, right?
So when this LDL they'reaccumulating these areas of
damage, then they becomeoxidized and it's the oxidized
fats that are really problematic.
(14:55):
That's why just looking at anLDL is really not sufficient to
tell you what your risk is ofcardioid, but we have to look at
the oxidized particles, thevery low density proteins there,
right?
So when they become oxidized,we get an increase in adhesion
molecules and then macrophages,which are cells of the immune
(15:16):
system.
They are activated and they aresupposed to scavenge and take
care of these LDL cells and wecreate what we call folin cells
and that's defined as firststage of atherosclerosis right,
or the plaque building up andthe hardening of our arteries.
These macromodules also producethese pro-inflammatory
(15:36):
cytokines.
And then the T cells, anotherimmune cell, is going to
stimulate proliferation of thesmooth muscle within the vessels
right.
So the smooth muscle cells aregoing to proliferate, they're
going to get thicker, there'sgoing to be more of them and
again that's going to cause anarrowing.
So if cholesterol, increasedcholesterol and increased blood
(15:57):
pressure are a consequence, whatis causing the dysfunction of
the endothelium in the firstplace?
So we know that strep, mutanssanguinus, aa, p gingival, t,
denticle all of these arebacteria strains that have been
found in aortic aneurysmspecimens and in diseased heart
(16:22):
valves.
They are frequently detected inatherosclerotic plaques and
studies have found differentoral species in anywhere from
48% to 100% of specimens.
Other infections herpes simplex, chlamydia, pneumonia that was
going around a lot in our areain its fall P gingivalis, hep C,
(16:44):
h pylori very common rightCauses a decrease in stomach
acid, influenza, a,cytomegalovirus, hiv those are
all implicated as risk factorsbecause all of those infections
can cause endothelialdysfunction.
In periodontitis we talk aboutthe red complex bacteria, the
(17:04):
worst of the worst.
So that includes the Pgingivalis, trypanema, denticola
, tannerella, forsythia,fusobacterium nucleatum, which
has recently been identified incolon cancers, prevotella
intermedia.
But again I want to stress thatthese that are only looking at
(17:30):
these red complex and maybe theorange complex, we're not really
getting a whole view of theoral cavity.
Although they are measuringlevels, it's important to
understand that they are goingto be present, so we cannot be
concerned if we have them.
We want to know how much of thegood commensal bacteria do we
(17:51):
have?
So all of these bacteria sharein common that they are grand
negative anaerobic, which meansthey thrive in a less oxygenated
environment.
So this is why, if you've everheard me talk or do my webinars,
I'm a big proponent of oxygendrops.
Getting more oxygen, a littlebit of hydrogen peroxide can go
a long way, because oxygen it'show the good commensal bacteria
(18:14):
they thrive on.
So the more oxygen we have, thefewer of these red complex
bacteria are going to be able tosurvive.
As I mentioned before too, whenwe find them together, they
create pathogenic synergism, andthe scary thing about these is
why.
Well, just previously, all weever want to do with these we're
(18:34):
going along.
Pasteur's theory right is justkill everything, so let's just
throw antibiotics at it.
But if you're more holisticwith oh I don't want to do
antibiotics, let me just throwessential oils at it, which are
also killing.
And again, we have to becareful because just if we kill
these bacteria but we don't fixthe environment that allowed
(18:54):
them to overgrow in the firstplace, we're just on that
hamster wheel, spinning andspinning and we're never
actually correcting the problem.
So we're managing the diseasewithout reversing it.
The interesting thing for me Iknow everything is so
interesting to me but thebacteria actually suppress our
innate immune system.
So not only are they creatingall sorts of inflammation and
(19:19):
endothelial dysfunction, theyare preventing our immune system
from being able to eradicatethem, they also cause blood
clotting right.
So we know the thicker ourblood is, the more easily that
it clumps, the more easily itcan adhere right to those
plaques that are forming theartery or they're more narrow,
the vessels are, harder it isfor that thick, clotty blood to
(19:42):
get through.
So that becomes a real problem.
They also produce enzymescalled proteases, which is what
helps some of the blood clotsand other things adhere and
cause invasion of other healthycells.
So let's talk a little bitabout these different bacteria.
P gingivalis it will upregulatefatty acid binding, so more and
(20:04):
more LDL will be attracted tothose sites which can then be
potentially oxidized, whichleads to more of the
proliferation of cells and theformation of these plaques.
P gingivalis can prevent theHDL right, that's the good
cholesterol from binding andtransporting right.
Normally HDL comes along and itchews up those LDL and goes on
(20:27):
its way.
But if the LDL now can't dothat anymore, right, that's when
we get that increase that theLDL isn't being taken away.
We just keep making more andmore because there's more and
more damage occurring.
P gingivals can oxidize HDL andthen now, instead of being that
protective transporter, it'sbecoming pro-aphrogenic.
(20:48):
P genovas also can ship theballot of our immune cells, th17
and T-regulatory cells toencourage more plaque formation
and it causes the aggregationand the clotting of the
platelets.
F nucleonum has been found inmany about 34% according to this
one study, 34% in carotidspecimen.
(21:11):
It increases cardiovascularrisk by increasing systemic
inflammation by directcolonization of these arterial
walls.
Okay, so it's directlyattaching to the arterial walls,
causing endothelial dysfunctionand, as I said too, it acts as
a bridge.
So when F-nucleotide is therewith P gingivalis, they work
(21:34):
together to create more adhesionmolecules and form more complex
microbial communities Often.
There was one study just donein 2022 in mice and they said
the oral administration ofF-nucleotide caused increases in
triglyceride and cholesterol,the T-fersyphia, t-dentacoloc,
(21:56):
both showing an atheroscleroticreasons average of 53%.
They cause an increase inC-reactive protein.
That is a marker often run byhopefully run by most internists
to look at any type ofinflammation.
It's a non-specific marker forchronic inflammation.
It also causes increase in LDL.
(22:16):
Right, again, the more damageto the endothelium, the more LDL
we're going to produce.
So when I see somebody whoseLDL is creeping up or getting
higher, I'm thinking what'scausing the damage?
Why is the liver beingrecruited to create more
Band-Aid?
Why do we need all theseBand-Aids?
Also, these two species alsowill lower nitric oxide.
(22:37):
There's one study done in mice.
Nitric oxide is one of themolecules responsible for
dilating our blood vessels.
Right we're talking about.
All these things are causingnarrowing of our blood vessels
and so it makes it harder to getthe blood and get oxygen where
it needs to go.
So nitric oxide important,right?
It's also correlated.
These two are correlated withhemorrhage plaques in the
(23:00):
vessels.
There was a study done byJenkinson and his colleagues and
they found that onestreptococcus bacteria was able
to get into the bloodstream.
And again, this can simply befrom chewing food, right,
brushing our teeth.
They found a protein theycalled PAD-A.
It sits on the outer surface ofthis bacteria and they say that
(23:23):
it hijacked blood platelets andforced them to clump together,
making these blood clots.
And when these clump togetherlike this, they surround the
bacteria and create almost ashell.
So when the bacteria is likeinside this clump of platelets,
they avoid detection by the hostimmune system.
(23:44):
This also allows them to hidefrom antibiotics.
So we know there's a hugedilemma with antibiotic
resistance happening and we'regetting all these superbots
happening.
Bacteria are very smart andthey can evolve and find ways to
evade detection and destruction.
So, very hard you have theseinfections of just throwing.
(24:05):
Again, when we talk about let'sjust kill everything with all
these antibiotics If theantibiotic can't get to the
bacteria, we're not really doinggood work right, where instead
we could be potentially causingmore problems by killing off
good commensal bacteria that areso important for absorbing our
nutrients and helping our immunesystem.
So, again, as well as helpingout the bacteria, this platelet
(24:28):
clumping, called small bloodclot, and they can grow in the
heart valves, causing thisendocarditis and further
inflammation of the bloodvessels, which can actually
block blood supply right to theheart and brain.
So they're working on somethingto try to counteract this
PADD-A where, yes, that's great,but wouldn't it be wonderful if
(24:49):
we just cleaned up the mouthright and the mouth is so
accessible?
That's what I love about my job.
It's a lot easier to try tocreate balance in the oral
cavity than trying to heal leakygut or, you know, ibs, right?
So we have the opportunity tohave direct impact on the mouth
and it's going to have directimpact on heart health, brain
(25:10):
health, liver health, you nameit.
How exactly is this dysfunctionhappening?
We've mentioned a few things,right?
So these infections get intothe bloodstream, they cause
clotting, they cause lyticsystems to activate all of this
fibrous, these specific cells,these foam cells.
They activate all of the innateimmune systems or start
(25:33):
producing more cytokines thatlead to more inflammation and
that causes just acceleration ofthese plaques throughout the
body.
Many of these plaques also showsigns of releasing endotoxins.
So it's not just the bacteria,but it's the byproducts of this
bacteria that can further causemore damage, and there are
(25:55):
several different kinds ofbyproducts we'll talk about
quickly.
So gingipines is one of thesetoxins given off by these
bacteria and these are cysteineproteases.
They attach to the host tissue,so they're going to attach to
that cell wall in the bloodvessel and they cause fibrous
(26:16):
maturation.
It gets a little thicker inthere.
Hemoglobin also causeshemoglobin binding and so that
hemoglobin binding causes thebacteria to be able to collect
amino acid and that's what theyfeed on.
These ginger peels are whatdirectly cause the degradation
of the collagen and theextracellular matrix proteins.
(26:36):
Right, so that's when we startto see the bone loss happen.
Right, and this is why I thinkof periodontitis as in the same
category as autoimmunity.
Right, body is eating away atyourself.
But I listened to this greatpodcast the other day.
It was Dave Asprey and GaryBrekka.
Gary was talking about what isautoimmunity and most of us
(27:00):
think of the body's gone haywire.
Right, it's doing something itshouldn't do.
But what he's saying?
No, the body is doing exactlywhat it's supposed to do because
by attacking the toxin.
I totally share this opinionthat, again, everything is
toxins and infections.
Why do we have he's talkingabout thyroid issue why is the
body attacking the thyroidtissue.
(27:22):
That thyroid tissue has beeninfected by a toxin, so the body
is producing antibodies rightto go after the toxin.
The toxin is in that thyroidtissue.
So what happens is the thyroidtissue ends up being attacked as
the body is trying to attackthe toxin.
So same exact thing in gumdisease.
Right, all of the endotoxinslike gingipanes and LPS are
(27:43):
being released.
The body wants to get rid ofthose toxins.
Part of if that toxin is in thebone that's surrounding the
teeth, then the bone is going toget eaten away in the body's
attempt to get rid of the toxin.
So, again, if we get rid of thetoxin, we get rid of the
infection, then the body isgoing to stop having this
autoimmune reaction and stopdestructing our own tissue.
(28:06):
What else do these genoplastiesdo?
They will inactivateimmunoglobulins and cytokines
that compromise the immunesystem.
We said that it suppresses theinnate immune system, causes
dysregulation of the coagulationcascade.
So this is why in gingivitisand periodontal disease we see
increased bleeding, so we canhave blood clotting, but we also
can see increased bleeding.
(28:27):
It also increases the vascularpermeability right, we all know
about leaky gut.
Anywhere you have a barrier,right In your mouth, in your
blood-brain barrier vessels ingeneral right Leaky vessels,
right.
So we're going to increase thatvascular permeability, which
will attract more white bloodcells to the infection.
It also activates MMPs thatdegrade more of that alveolar
(28:50):
bone, as well as prostaglandins,which cause pain.
Lps this is a really well-knowntoxin that bacteria give off.
It is, according to someresearch, one of the major
factors leading to fatty liverdisease.
So again, really important tounderstand the consequence of
having these low-grade chronicinfections.
(29:10):
Lps is actually a component ofthe outer membrane of
gram-negative bacteria.
Normally it's recognized by theinnate immune system at these
CD14 receptors and that willinitiate the cytokine cascade
and platelet aggregation againtrying to encapsulate these
bacteria and move them out ofthe way.
Lps will increase calcification.
(29:34):
So for you women out, there yougo, you have your mammogram and
they're like oh yes, you havedense breasts, lots of
calcification.
This is just normal, it's verycommon, but it doesn't mean
that's normal.
So if we have a lot ofcalcification, we're producing a
lot of tartar on our teeth allthe time, right?
Why do we have this increasedcalcification?
Could it be caused from someagain low-grade infection going?
(29:58):
Lps causes a progression of theplaque lipid deposition, so it
increases more LDL by damagingthat endothelial barrier,
increases oxidative stress.
Most diseases have increasedoxidative like.
The whole idea of aging is allabout oxidative stress, right?
It also LPS directly increasesthe C reactive protein.
(30:18):
There are also bioprones calledleukotoxins.
So from the bacteria AA itactually causes pores in the
lipid bilayer, right?
So every cell has this lipidbilayer, every cell has this
lipid bilayer that protects,actually, the brain of the cell.
It's this lipid bilayer, and soAA can directly cause holes and
can kill white blood cells.
(30:39):
By creating these holes itcauses apoptosis, which is
programmed cell death, increasedbreakdown of the macrophages
right?
Those are supposed to be thecleanup crew and get rid of all
the bad stuff.
It can, those are supposed tobe the cleanup crew, get rid of
all the bad stuff.
It can break down naturalkiller cells and the T cells,
(31:00):
which are protective, right?
So again, suppressing thatinnate immune system by directly
killing our immune cells.
It can also the glucotoxinsbind to the white blood cells
and therefore wherever thatwhite blood cell goes in the
bloodstream, so does theinfection, can go somewhere else
in the body and cause problemsin that part of the body.
There are also heat shockproteins, so during an infection
we have bacterial heat shockproteins and they're very highly
(31:21):
immunogenic.
So they're very highlystimulatory to our immune system
.
And the problem is we havehuman heat shock proteins and
the body may get confused andnot be able to differentiate
between them.
So those heat shock proteinscan further cause endothelial
function.
They also enhance adhesionmolecules, which causes more
thinning of the blood vessels,more patient.
(31:45):
The cytokines everybody heard alot about cytokines during the
pandemic and how that cytokinestorm really caused death.
Right when the inflammation gotway too ramped up there was
just such an increase ininfection that the body couldn't
keep up.
So having cytokines isimportant, but having too many
is not really a good thing.
We get increased swelling, moregranulation tissue when we have
(32:08):
too many cytokines.
So what is the traditionaltherapy?
Traditionally for a gone disease, periodontitis, we do scaling
and root cleaning, right.
We numb you up and we scrapethe crap out of your teeth
trying to get all that off andthen send you on your way and
most studies say that's great,but actually we can regrow
plaque and tartar within 10 days.
(32:30):
So that doesn't reduce theamount of bacteria in the mouth.
Reduce the amount of bacteriain the mouth, but did we do
anything to address theenvironment.
Again, all the idea of justkilling, just get rid of the
bacteria and then everythingwill be wonderful.
So then down the line westarted.
(32:55):
What if we add antibiotics withthe scaling root planing?
So we can give systemicantibiotics, which we now know
is probably not a great idea, orwe can give targeted
antibiotics.
Let's just put that in that 10millimeter pocket, right, so we
have certain kinds ofantibiotics that we can localize
and put it right in.
But again, is anything reallylocalized?
Because, right, it can get intoyour mean bloodstream, but it
is in a smaller dose and thereare a lot of studies that really
(33:18):
show no inhibition.
Again, maybe initially for afew weeks, but then really we go
right back to square one.
So in my mind, like, okay.
Again I think about theenvironment.
Yes, we have to reduce theamount of bacteria.
So, yes, let's get the tartarout of there, let's get these
roots nice and cleaning right sowe can have the gum tissue
(33:40):
reattached in these areas.
What else can we do?
What if we use laser, right?
Laser light energy, as you'veheard me talk about in previous
podcasts, is amazinglystimulatory for mitochondria.
We need that increased energyin order to produce more oxygen,
make more ATP to help ourimmune cells fight whatever the
(34:02):
inflammation is going on.
So laser totally non-invasiveand wonderful, I find that we
really do need to use it incombination with other things.
So we have scalar and plateletplus ozone.
Again, we need to take downthat and we need to put more
oxygen in that environmentdirectly.
And then enzymes I really likeproteolytic enzymes because,
(34:24):
again, remember we talked abouthow amino acids are used by the
pathogens in order to grow andincrease their numbers.
So if we degrade down all ofthose amino acids, we're going
to take away the food source,right.
So again, think about theenvironment.
I love oxygen drops.
I want my patients all to be onoxygen drop and then we can use
(34:46):
combinations from all thesetoxicity.
We need to increase our minerals.
We need to increase the fatsoluble vitamins, right D, b, a
and K.
They all have to work together.
The fat soluble vitamins andminerals work together to
(35:08):
produce healthy teeth and gums.
You can't just take vitamin D,you can't just take magnesium.
The combination together iswhat creates health.
And again, with prebiotics,probiotics we're providing the
food for the good stuff toovergrow.
And I've done my own littlepilot studies and I've seen
incredible results with thistype of therapy Without changing
(35:33):
diet, which we know.
Diet is so crucial.
Right, a lot of the toxins weis from our diet.
We eat so much processed food.
It's just hard to eat healthywhole foods these days, just the
way society is.
But again, with even justtargeting these things in the
mouth, it's going to make a hugesystemic impact.
So the goal is always toincrease the good bacteria and
(35:57):
reduce the diversity in the oralmicrobe.
So we need to reduce the toxinand we need to improve our
nutrition.
Now there are some people withcertain polymorphisms in their
genetics, right.
There's polymorphisms or SNPs,we call them right in certain
cytokines and that can lead tosome people have greater
inflammation.
(36:17):
There's some about MTHFR.
Many of you have heard of that.
It's probably one of the beststudies out there.
If we don't methylate properly,we can't detox properly.
Chronic anemia that is also canbe a sign of chronic disease.
So a lot of people freak out ohmy gosh, we need to supplement
you with iron because you'rebecoming so anemic.
(36:39):
But again, why are they anemic?
Could it be a chronic infectionstealing all of that iron?
Again, let's dig a littledeeper on these things and
figure out the real reason.
So let's sum everything up here.
We need to think aboutnutrients versus toxins, right?
Toxicity leads to deficiency,so our best goal to fight any
(37:02):
chronic disease is making surewe're getting those nutrients
and minimizing the toxins to thebest of our ability.
And in today's society, Ibelieve that supplements are
necessary.
And again, basics Everybody'sdifferent, right?
Our lifestyles are a littledifferent, Our genetics are a
little different, our geneticsare a little different, but it's
very basic.
Having those fat solublevitamins, having the minerals
(37:23):
that is key cornerstone of goodhealth.
So you just got to have thebasic building blocks for
everything to work well.
Then we have to also thinkabout our nervous system, right?
Those of us who tend to havemore chronic issues are those of
us who spend way too much timein that sympathetic nervous
system.
We're overthinking stuff allthe time.
(37:46):
There's just so much bombardingus and we don't realize it.
Led lights, too much of that.
We're inside all the time, thenutrient-poor food, so that we
have a lot working against us.
But that's okay, because we'reaware now and so we know we can
take these little steps to bringus that much healthier.
All right.
(38:08):
So the biggest takeaways for meis that, remember, the cause of
heart disease is not increasedblood pressure and increased
cholesterol, right, the cause isthe endothelial dysfunction.
The cause is from.
It could be these bacteria,these viruses, these toxins like
the LPS and the gingipanes,right, and it is the shift in
(38:31):
the environment that leads to anincrease in these pathobionts
that become pathogens, right, soit's the lack of nutrients, the
lack of oxygen, the lack of theminerals, the lack of vitamins
and excess sugars right, wedidn't even talk about sugar at
all.
We know sugar can lead to decay, right, but sugar's also
directly caused an increase inthe reactive oxidative species.
(38:54):
Anytime we have those increasedROS, we're going to have
chronic disease and inflammation.
So, an interesting thing is,blood glucose will also
stimulate HMG-CoA reductase.
So this is the enzyme that'stargeted for statin drugs.
If you have high cholesterol,often your doctor throws you on
(39:16):
a statin, and that is what'sgoing to stop the cholesterol
biosynthesis, right, stop theproduction of that LDL.
But what did we just mentiontoday?
Right, ldl is the band beingproduced.
We want to understand whythere's an increase of Band-Aids
(39:38):
being produced, and bloodglucose is a big one, right?
So if your cholesterol is goingup, the first thing I want you
to do is look for the hiddeninfection.
Test your HbA1c, right.
Because glucose this is goingto affect your liver.
Liver is where cholesterol ismade.
Liver also is protective of thepancreas.
(39:59):
So diabetes, and that reallystarts with a liver problem and
a metabolism problem.
So we've got to think about howall these things work together.
And blood glucose in and ofitself causes direct damage to
the endothelium.
Think about diabetics losinglimbs, losing their eyesight.
This is because that excessblood is directly causing
(40:21):
endothelial dysfunction.
I had a patient the other daywas talked about having swelling
in his legs.
He had recently been put oncholesterol medication.
A blood pressure medicationsaid his cholesterol was 500.
And so then the doctor thoughtoh, you have a heart problem, no
, you have a kidney problem.
He was diagnosed with a kidneydisease.
My first question was have they?
(40:42):
Even I can't seem to loseweight and I said have they
checked your blood sugar?
Because what he's experiencedis this leaky vessel syndrome.
Remember, we talked about allof the bacteria and these toxins
cause vascular permeability,the leaking out of fluids, right
.
So when we get edema orswelling, that is a leaky vessel
right, that is endothelialdysfunction.
(41:05):
So let's take it all the wayback and think what is the
infection, what is the toxinthat could be causing, instead
of simply taking a certainprescription to minimize the
symptom?
Because we may minimize thesymptom, but if we don't correct
the damage, we're just pushingthe buck down the line a little
bit without really resolving theissue.
So a really important keyfactor too, with increased blood
(41:29):
pressure and increasedcholesterol.
Please take a look at it.
Is your C-reactive proteinelevated and what is your blood
flow?
Look at the insulin.
Look at your blood glucose.
Look at your.
Is your C-reactive proteinelevated and what is your blood
flow?
Look at the insulin.
Look at your blood glucose.
Look at your HbA1c, which is ameasure of your blood sugar over
the last three months.
So it's a good thing to see.
Okay, and we know that type 2diabetes is reversible.
(41:50):
It may not be simple, but withcertain lifestyle changes and
tweaking our nutrition, weabsolutely can reverse all of
these things.
It's just a matter of thinkinga little differently and taking
the time to dig a little bitdeeper, right?
So we want to talk about theaction steps here, right?
(42:11):
So we might want to test fororal microblom.
I like Br Health Check for 700different species so we can see
the balance.
Right, as I just mentioned,check the C-reactive protein,
all of the blood levels and whatabout your mineral levels and
fat soluble vitamins.
Let's check those levels.
Let's talk about diet andstress.
How about sleeping?
(42:32):
Briefly mentioning, mouthbreathing causes a huge shift in
the oral microbiome, so shouldwe have a sleep study?
No, we can do them at home.
They're super easy and portable, right?
So let's talk about how we'rebreathing during the day.
Right?
Are we mouth breathing duringthe day?
Let's talk about some additivefunctions at the dentist, right?
(42:53):
Between ozone you can buyaffordable ozone machines now to
have in your home.
Even lasers we sell a littlered light hand-held, looks like
a flashlight about $225, right,that's not bad if it's something
you're using every single dayand you're really enhancing your
mitochondria.
And at home, let's think abouttoothpaste that don't have a lot
of chemicals in them.
Let's think about somethinglike Rewitin prebiotic minerals
(43:17):
don't have a lot of chemicals.
Let's think about somethinglike rewiten prebiotic minerals,
vitamins it's enhancing thatenvironment.
Liquid minerals A lot of us havedigestive issues.
If we take a liquid mineral,we're going to absorb that
through the mucosa faster.
It doesn't have to go throughthe digestive process.
Let's think about enzymes,right?
I've used a lot of enzymes tohelp people with arthritis,
right, which is basicallyinflammation in the joint.
You need to break down allthose excess proteins that are
(43:39):
causing the body to cause moreinflammation and breakdown of
the joints.
Oxygen and binders expand ofthe binders.
We talk about cell 4 bindersall the time.
The fulvic and humic acids notonly bind up all of these toxins
, but they also supplyelectrolytes and amino acids at
the same time.
(43:59):
So sometimes binders can taketoo much of our fluids and leave
us at more deficiency, whereasthe cell core binders are very
different or created muchdifferently than, say, like
activated charcoal.
So these are things that Ialways think about when I'm
looking at my patients and Iwant to really reverse all of
these kind of inflammationchanges that are going on.
(44:23):
So we've talked about a lot ofstuff today and I hope that all
of you can take a nugget or twoout of this and really start
thinking about.
My goal in doing this podcastis to make us all our own best
doctors, to make us think alittle bit more critically about
the cause of certain things andsimple things that we can do
(44:44):
after to give us a better,healthiest life possible.
I hope you enjoyed this solopodcast.
Please feel free to reach outwith questions and I love
comments.
I love ideas for new podcastsor people you want to hear from,
so keep the comments coming Ifyou are getting some value out
of the podcast.
(45:05):
Again, I thank you.
Please share this with others.
You can give us a review thatreally helps more people see, so
that we can create as muchawareness and help as many
people as possible.
So thanks again, everybody.
I hope you have a wonderful dayand I'll see you on the next
episode.
Hello, I'm Dr Rachel Carver, aboard-certified naturopathic,
(45:26):
biologic dentist and a certifiedhealth coach.
Did you know that over 80% ofthe US population has some form
of gum disease?
Many of us don't even know thatwe have this source of chronic
infection and inflammation inour mouth that's been linked to
serious consequences like heartdisease, diabetes, stroke,
dementia, colon cancer, kidneydisease, even pregnancy
(45:47):
complications.
Would you like to learn how toreverse and prevent these
chronic debilitating conditionswithout spending a lot of time
and money at the dentist?
Join me for my six-week coursewhere I will teach you the root
cause of disease.
You'll learn how to be your ownbest doctor.
Are you ready to get started?
Let's go.
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